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There are over 27 different subtypes of peripheral T\u2010cell lymphoma (PTCL), and we are now beginning to understand the differences between the various subtypes beyond histologic variations.<\/jats:p><\/jats:sec><jats:sec><jats:title>Molecular Pathogenesis of Various Subtypes of <jats:styled-content style=\"fixed-case\">PTCL<\/jats:styled-content><\/jats:title><jats:p>Gene expression profiling (GEP) can help in diagnosis and prognostication of various subtypes including PTCL\u2010nos and anaplastic large cell lymphoma (ALCL). In addition, mutational analysis is now being incorporated in clinical trials of novel agents to evaluate various biomarkers of response to allow better therapeutic choices for patients.<\/jats:p><\/jats:sec><jats:sec><jats:title>Targeted Therapies<\/jats:title><jats:p>There are many targeted agents currently in various stages of clinical trials for PTCL that take advantage of the differential expression of specific proteins or receptors in PTCL tumors. This includes the CD30 directed antibody drug conjugate brentuximab vedotin. Other notable targets are CD25, CCR4, inhibition of PI3kinase \u2013 m TOR and JAK\/STAT pathways. The ALK inhibitors are promising for ALK expressing tumors.<\/jats:p><\/jats:sec><jats:sec><jats:title>Immunotherapies<\/jats:title><jats:p>Allogeneic stem cell transplant continues to be the curative therapy for most aggressive subtypes of PTCL. The use of checkpoint inhibitors in the treatment of PTCL is still controversial. The most promising results have been seen in cases of extranodal natural killer cell\/T\u2010cell (ENK\/T) lymphomas and cutaneous T\u2010cell lymphomas (CTCL). 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