{"status":"ok","message-type":"work","message-version":"1.0.0","message":{"indexed":{"date-parts":[[2023,11,13]],"date-time":"2023-11-13T00:27:59Z","timestamp":1699835279789},"reference-count":19,"publisher":"Wiley","issue":"3","license":[{"start":{"date-parts":[[2006,7,11]],"date-time":"2006-07-11T00:00:00Z","timestamp":1152576000000},"content-version":"vor","delay-in-days":9018,"URL":"http:\/\/onlinelibrary.wiley.com\/termsAndConditions#vor"}],"content-domain":{"domain":[],"crossmark-restriction":false},"short-container-title":["American J Hematol"],"published-print":{"date-parts":[[1981,11]]},"abstract":"<jats:title>Abstract<\/jats:title><jats:p>In some cases, bone marrow aplasia has been thought to result from immunologic abnormalities. Our patient had severe transfusion\u2010dependent aplastic anemia, which responded to treatment with prednisone on two occasions. The exacerbations of aplastic anemia were associated with lymphocytic proliferation which on one occasion had the characteristics of T\u2010cell chronic lymphocytic leukemia. Though he had had mild lymphoproliferation for a number of years, he ultimately died with progressive diffuse lymphoid infiltration of the bone marrow and other organs. The simultaneous occurrence of the T\u2010cell lymphoproliferation and aplastic anemia and their simultaneous response to therapy suggests that this may be a clinical example of T\u2010lymphocyte mediated suppression of erythropoiesis.<\/jats:p>","DOI":"10.1002\/ajh.2830110310","type":"journal-article","created":{"date-parts":[[2007,2,21]],"date-time":"2007-02-21T06:31:15Z","timestamp":1172039475000},"page":"293-298","source":"Crossref","is-referenced-by-count":1,"title":["Prednisone\u2010responsive aplastic anemia associated with T\u2010lymphocyte proliferation"],"prefix":"10.1002","volume":"11","author":[{"given":"Sara","family":"Bodner","sequence":"first","affiliation":[],"role":[{"role":"author","vocabulary":"crossref"}]},{"given":"Harvey Jay","family":"Cohen","sequence":"additional","affiliation":[],"role":[{"role":"author","vocabulary":"crossref"}]}],"member":"311","published-online":{"date-parts":[[2006,7,11]]},"reference":[{"key":"e_1_2_1_2_2","doi-asserted-by":"crossref","first-page":"1","DOI":"10.1182\/blood.V34.1.1.1","article-title":"Studies in red cell aplasia. 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