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In both patients, ipsilateral foveal pursuit and full\u2010field pursuit of a surrounding optokinetic drum were impaired while voluntary and involuntary saccades (fast components) were normal. Foveal pursuit was impaired more than full\u2010field pursuit, suggesting the existence of two separate pathways by which visual signals for OKN slow phases reach the oculomotor centers in the brainstem. We conclude that the ipsilateral slow phase OKN deficit seen in out patients resulted from damage to the foveal pursuit pathway that runs from the occipitoparietal association area to the ipsilateral brainstem horizontal gaze center.<\/jats:p>","DOI":"10.1002\/ana.410070311","type":"journal-article","created":{"date-parts":[[2005,1,1]],"date-time":"2005-01-01T16:58:40Z","timestamp":1104598720000},"page":"269-276","source":"Crossref","is-referenced-by-count":114,"title":["Optokinetic nystagmus and parietal lobe lesions"],"prefix":"10.1002","volume":"7","author":[{"given":"Robert W.","family":"Baloh","sequence":"first","affiliation":[],"role":[{"role":"author","vocabulary":"crossref"}]},{"given":"Robert 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