{"status":"ok","message-type":"work","message-version":"1.0.0","message":{"indexed":{"date-parts":[[2026,10,5]],"date-time":"2026-10-05T01:27:07Z","timestamp":1791163627277,"version":"4.1.0"},"reference-count":34,"publisher":"Wiley","issue":"12","license":[{"start":{"date-parts":[[2005,11,29]],"date-time":"2005-11-29T00:00:00Z","timestamp":1133222400000},"content-version":"vor","delay-in-days":7303,"URL":"http:\/\/onlinelibrary.wiley.com\/termsAndConditions#vor"}],"content-domain":{"domain":[],"crossmark-restriction":false},"short-container-title":["Arthritis &amp; Rheumatism"],"published-print":{"date-parts":[[1985,12]]},"abstract":"<jats:title>Abstract<\/jats:title><jats:p>Two histopathologic types of inflammatory vascular disease (IVD) occur in Sj\u00f6gren's syndrome (SS): mononuclear IVD (MIVD) and neutrophilic IVD (NIVD). We describe 50 SS patients with IVD (30 with NIVD and 20 with MIVD). Thirty\u2010three (66%) of the SS patients with biopsy\u2010documented IVD had nervous system disease unattributable to other causes. Nineteen patients (58%) had involvement of both the central and peripheral nervous systems, while 9 had peripheral and 5 had central nervous system dysfunction alone. Patients with both histopathologic types of IVD were at risk for the development of nervous system abnormalities (57% of NIVD patients and 80% of MIVD patients). Indirect evidence is presented which suggests that IVD may play a role in the immunopathogenesis of nervous system disease, at least in a subset of SS patients.<\/jats:p>","DOI":"10.1002\/art.1780281205","type":"journal-article","created":{"date-parts":[[2007,2,14]],"date-time":"2007-02-14T21:05:08Z","timestamp":1171487108000},"page":"1341-1347","source":"Crossref","is-referenced-by-count":90,"title":["Peripheral inflammatory vascular disease in Sj\u00f6gren's syndrome. Association with nervous system complications"],"prefix":"10.1002","volume":"28","author":[{"given":"R.","family":"Molina","sequence":"first","affiliation":[],"role":[{"vocabulary":"crossref","role":"author"}]},{"given":"T. T.","family":"Provost","sequence":"additional","affiliation":[],"role":[{"vocabulary":"crossref","role":"author"}]},{"given":"E. L.","family":"Alexander","sequence":"additional","affiliation":[],"role":[{"vocabulary":"crossref","role":"author"}]}],"member":"311","published-online":{"date-parts":[[2005,11,29]]},"reference":[{"key":"e_1_2_1_2_2","doi-asserted-by":"publisher","DOI":"10.7326\/0003-4819-70-4-751"},{"key":"e_1_2_1_3_2","doi-asserted-by":"publisher","DOI":"10.7326\/0003-4819-98-2-155"},{"key":"e_1_2_1_4_2","unstructured":"MolinaR ProvostTT ArnettFC BiasWB HochbergMC WilsonRW AlexanderEL:Primary Sj\u00f6gren's syndrome (SS) in men\u2014clinical serologic and immunogenetic features. 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