{"status":"ok","message-type":"work","message-version":"1.0.0","message":{"indexed":{"date-parts":[[2025,10,31]],"date-time":"2025-10-31T07:05:18Z","timestamp":1761894318062,"version":"3.32.0"},"reference-count":15,"publisher":"Wiley","issue":"6","license":[{"start":{"date-parts":[[2006,5,26]],"date-time":"2006-05-26T00:00:00Z","timestamp":1148601600000},"content-version":"vor","delay-in-days":0,"URL":"http:\/\/onlinelibrary.wiley.com\/termsAndConditions#vor"}],"content-domain":{"domain":[],"crossmark-restriction":false},"short-container-title":["Arthritis &amp; Rheumatism"],"published-print":{"date-parts":[[2006,6]]},"abstract":"<jats:title>Abstract<\/jats:title><jats:p>AA amyloidosis is the most serious potential complication of the inherited autoinflammatory syndromes and frequently results in end\u2010stage renal failure. Although this complication is well recognized in familial Mediterranean fever, tumor necrosis factor receptor\u2013associated periodic syndrome, and Muckle\u2010Wells syndrome, there is only 1 previous published report of its occurrence in hyperimmunoglobulinemia D with periodic fever syndrome (HIDS). We report 2 further cases of patients with AA amyloidosis in HIDS, both of whom developed dialysis\u2010dependent renal failure, and we describe the outcome of the first renal transplant in this setting.<\/jats:p>","DOI":"10.1002\/art.21901","type":"journal-article","created":{"date-parts":[[2006,5,26]],"date-time":"2006-05-26T16:28:16Z","timestamp":1148660896000},"page":"2010-2014","source":"Crossref","is-referenced-by-count":36,"title":["AA amyloidosis complicating hyperimmunoglobulinemia D with periodic fever syndrome: A report of two cases"],"prefix":"10.1002","volume":"54","author":[{"given":"Helen J.","family":"Lachmann","sequence":"first","affiliation":[],"role":[{"role":"author","vocabulary":"crossref"}]},{"given":"Hugh J. 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