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The aim of this study is to reveal the risk factors for <jats:styled-content style=\"fixed-case\">AoD<\/jats:styled-content> during pregnancy and to compare the 2 main risk factors, Marfan syndrome and pregnancy itself in the previously healthy woman. The pregnant patients developed <jats:styled-content style=\"fixed-case\">AoD<\/jats:styled-content> at 31.7\u2009\u00b1\u20097.6\u2009weeks of gestation. It occurred much earlier in the Marfan patients than in the previously healthy women (30.7\u2009\u00b1\u20098.6\u2009weeks of gestation vs 34.4\u2009\u00b1\u20094.4\u2009weeks of gestation, <jats:italic>P<\/jats:italic> = 0.0263). In the Marfan patients, <jats:styled-content style=\"fixed-case\">AoD<\/jats:styled-content> developed in 3 (3.2%), 15 (15.8%), and 43 (46.3%) patients in the 3 trimesters, respectively, compared with 31 of the previously healthy women, and only in the third trimester. The neonates of the Marfan patients had better Apgar scores at 1 and 5\u2009minutes, lower intubation rates, and fewer stays in the neonatal intensive care unit than those of the previously healthy women. Marfan syndrome and pregnancy itself in the previously healthy woman were the 2 main risk factors responsible for the occurrence of <jats:styled-content style=\"fixed-case\">AoD<\/jats:styled-content> during pregnancy. Marfan patients may develop <jats:styled-content style=\"fixed-case\">AoD<\/jats:styled-content> at an early age and an early stage of pregnancy, probably due to the preexisting weakened aortic wall. Better outcomes for the surviving neonates of Marfan patients compared with neonates of the previously healthy women might be the result of the poor condition of Marfan patients causing a higher death rate for those fetuses.<\/jats:p>","DOI":"10.1002\/clc.22165","type":"journal-article","created":{"date-parts":[[2013,7,10]],"date-time":"2013-07-10T18:34:46Z","timestamp":1373481286000},"page":"576-584","source":"Crossref","is-referenced-by-count":56,"title":["Aortic Dissection During Pregnancy: A Difficult Clinical Scenario"],"prefix":"10.1002","volume":"36","author":[{"given":"Shi\u2010Min","family":"Yuan","sequence":"first","affiliation":[],"role":[{"vocabulary":"crossref","role":"author"}]}],"member":"311","published-online":{"date-parts":[[2013,7,10]]},"reference":[{"key":"e_1_2_6_2_1","doi-asserted-by":"publisher","DOI":"10.1016\/S0003-4975(03)00169-3"},{"key":"e_1_2_6_3_1","doi-asserted-by":"publisher","DOI":"10.1016\/j.ejogrb.2009.10.029"},{"key":"e_1_2_6_4_1","first-page":"1","volume-title":"Aortic Dissection and Related Syndromes","author":"Isselbacher EM.","year":"2007"},{"key":"e_1_2_6_5_1","doi-asserted-by":"publisher","DOI":"10.1016\/S0025-6196(11)60566-1"},{"key":"e_1_2_6_6_1","doi-asserted-by":"publisher","DOI":"10.1161\/CIRCULATIONAHA.110.958975"},{"key":"e_1_2_6_7_1","doi-asserted-by":"crossref","first-page":"12","DOI":"10.1542\/peds.102.1.e12","article-title":"Further delineation of aortic dilation, dissection and rupture in patients with Turner syndrome","volume":"102","author":"Lin AE","year":"1998","journal-title":"Pediatrics."},{"key":"e_1_2_6_8_1","doi-asserted-by":"publisher","DOI":"10.1016\/j.jacc.2003.08.054"},{"key":"e_1_2_6_9_1","first-page":"1233","article-title":"Aortic medial changes associated with bicuspid aortic valve: myth or reality?","volume":"15","author":"Parai JL","year":"1999","journal-title":"Can J Cardiol."},{"key":"e_1_2_6_10_1","doi-asserted-by":"publisher","DOI":"10.1136\/hrt.2005.074781"},{"key":"e_1_2_6_11_1","unstructured":"US National Institutes of Health National Library of Medicine Genetics Home Reference. 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