{"status":"ok","message-type":"work","message-version":"1.0.0","message":{"indexed":{"date-parts":[[2023,11,20]],"date-time":"2023-11-20T00:07:41Z","timestamp":1700438861537},"reference-count":10,"publisher":"Wiley","issue":"1","license":[{"start":{"date-parts":[[2006,12,5]],"date-time":"2006-12-05T00:00:00Z","timestamp":1165276800000},"content-version":"vor","delay-in-days":1799,"URL":"http:\/\/onlinelibrary.wiley.com\/termsAndConditions#vor"}],"content-domain":{"domain":[],"crossmark-restriction":false},"short-container-title":["Clinical Cardiology"],"published-print":{"date-parts":[[2002,1]]},"abstract":"<jats:title>Abstract<\/jats:title><jats:p><jats:italic>Background<\/jats:italic>: Electrocardiographic (ECG) Q waves in V leads (V<jats:sub>2<\/jats:sub> or V<jats:sub>3<\/jats:sub>) pose a diagnostic challenge in the presence of left anterior fascicular block (LAFB). Benign Q waves in the absence of coronary artery disease (CAD) primarily due to LAFB have been described. This study evaluates Q waves in the presence of LAFB.<\/jats:p><jats:p><jats:italic>Hypothesis<\/jats:italic>: Anterior Q waves in the presence of LAFB may not be indicative of myocardial infarction (MI).<\/jats:p><jats:p><jats:italic>Methods<\/jats:italic>: From 1990 to 1997, ECGs of 236 male patients with LAFB were analyzes for presence of Q waves in the V leads. Records were reviewed for evidence of CAD. Of 236 patients with LAFB, 61 (26%) had Q waves in the V leads. In this group, 31 patients were available for further analysis.<\/jats:p><jats:p><jats:italic>Results<\/jats:italic>: Of the 31 patients with Q waves in the V leads who were available for further study, LAFB was present in 22 patients (71%) and LAFB plus right bundle\u2010branch block (RBBB) were present in 9 (29%). Of 20 patients with Q waves due to MI, 13 (65%) had LAFB and 7 (35%) had LAFB plus RBBB. Of 11 patients with benign Q waves, 9 (82%) and 2 (18%) had LAFB and LAFB plus RBBB, respectively. Benign Q waves were noted in 5.3% (11\/206) patients with LAFB. The mean duration of Q waves and Q\u2010wave location limited to V<jats:sub>2<\/jats:sub> and\/or V<jats:sub>3<\/jats:sub> are 0.029 s and 64% versus 0.053 s and 15% in benign versus pathologic Q waves, respectively.<\/jats:p><jats:p><jats:italic>Conclusion<\/jats:italic>: Patients with LAFB in the absence of MI may have Q waves in the V leads that are approximately 0.02 s in duration and restricted to one or two leads. This anomaly may represent a variation of conduction in the initial 0.02 s QRS vector due to LAFB.<\/jats:p>","DOI":"10.1002\/clc.4950250106","type":"journal-article","created":{"date-parts":[[2007,6,27]],"date-time":"2007-06-27T13:48:32Z","timestamp":1182952112000},"page":"19-22","source":"Crossref","is-referenced-by-count":1,"title":["Significance of anterior Q waves in left anterior fascicular block\u2014A clinical and noninvasive assessment"],"prefix":"10.1002","volume":"25","author":[{"given":"Udipi R.","family":"Shettigar","sequence":"first","affiliation":[],"role":[{"role":"author","vocabulary":"crossref"}]},{"given":"Anu","family":"Pannuri","sequence":"additional","affiliation":[],"role":[{"role":"author","vocabulary":"crossref"}]},{"given":"George H.","family":"Barbier","sequence":"additional","affiliation":[],"role":[{"role":"author","vocabulary":"crossref"}]},{"given":"Doreen O.","family":"Appunn","sequence":"additional","affiliation":[],"role":[{"role":"author","vocabulary":"crossref"}]}],"member":"311","published-online":{"date-parts":[[2006,12,5]]},"reference":[{"key":"e_1_2_1_2_2","volume-title":"Los hemibloqueos","author":"Rosenbaum MB","year":"1968"},{"key":"e_1_2_1_3_2","first-page":"141","article-title":"The hemiblocks: Diagnostic criteria and clinical significance","volume":"39","author":"Rosenbaum MB","year":"1970","journal-title":"Mod Concepts Cardiovasc Dis"},{"key":"e_1_2_1_4_2","first-page":"363","article-title":"Left anterior hemiblock simulating anteroseptal myocardial infarction","volume":"92","author":"Farnham DJ","year":"1976","journal-title":"Am Heart J"},{"key":"e_1_2_1_5_2","doi-asserted-by":"publisher","DOI":"10.1016\/0002-8703(71)90365-6"},{"key":"e_1_2_1_6_2","doi-asserted-by":"publisher","DOI":"10.1016\/0002-8703(74)90006-4"},{"key":"e_1_2_1_7_2","doi-asserted-by":"publisher","DOI":"10.7326\/0003-4819-60-3-430"},{"key":"e_1_2_1_8_2","doi-asserted-by":"publisher","DOI":"10.1016\/0002-9149(88)91058-2"},{"key":"e_1_2_1_9_2","doi-asserted-by":"publisher","DOI":"10.1378\/chest.65.2.123"},{"key":"e_1_2_1_10_2","doi-asserted-by":"publisher","DOI":"10.1016\/0002-8703(69)90496-7"},{"key":"e_1_2_1_11_2","doi-asserted-by":"publisher","DOI":"10.1378\/chest.62.1.91"}],"container-title":["Clinical Cardiology"],"original-title":[],"language":"en","link":[{"URL":"https:\/\/api.wiley.com\/onlinelibrary\/tdm\/v1\/articles\/10.1002%2Fclc.4950250106","content-type":"unspecified","content-version":"vor","intended-application":"text-mining"},{"URL":"https:\/\/onlinelibrary.wiley.com\/doi\/pdf\/10.1002\/clc.4950250106","content-type":"unspecified","content-version":"vor","intended-application":"similarity-checking"}],"deposited":{"date-parts":[[2023,11,19]],"date-time":"2023-11-19T11:06:16Z","timestamp":1700391976000},"score":1,"resource":{"primary":{"URL":"https:\/\/onlinelibrary.wiley.com\/doi\/10.1002\/clc.4950250106"}},"subtitle":[],"short-title":[],"issued":{"date-parts":[[2002,1]]},"references-count":10,"journal-issue":{"issue":"1","published-print":{"date-parts":[[2002,1]]}},"alternative-id":["10.1002\/clc.4950250106"],"URL":"https:\/\/doi.org\/10.1002\/clc.4950250106","archive":["Portico"],"relation":{},"ISSN":["0160-9289","1932-8737"],"issn-type":[{"value":"0160-9289","type":"print"},{"value":"1932-8737","type":"electronic"}],"subject":[],"published":{"date-parts":[[2002,1]]}}}