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However, it has not been established whether the precursors that reside in the SVZ retain the capacity to produce the full range of striatal neurons that has been destroyed. By using a neonatal rat model of hypoxic\/ischemic brain damage, we show here that virtually all of the newly produced striatal neurons are calretinin (CR)\u2010immunoreactive (+), but not DARPP\u201032<jats:sup>+<\/jats:sup>, calbindin\u2010D\u201028K<jats:sup>+<\/jats:sup>, parvalbumin<jats:sup>+<\/jats:sup>, somatostatin<jats:sup>+<\/jats:sup>, or choline acetyltransferase<jats:sup>+<\/jats:sup>. Retroviral fate\u2010mapping studies confirm that these newly born CR<jats:sup>+<\/jats:sup> neurons are indeed descendants of the SVZ. Our studies indicate that, although the postnatal SVZ has the capacity to produce a range of neurons, only a subset of this repertoire is manifested in the brain after injury. J. Comp. Neurol. 511:19\u201333, 2008. \u00a9 2008 Wiley\u2010Liss, Inc.<\/jats:p>","DOI":"10.1002\/cne.21819","type":"journal-article","created":{"date-parts":[[2008,8,21]],"date-time":"2008-08-21T13:39:23Z","timestamp":1219325963000},"page":"19-33","source":"Crossref","is-referenced-by-count":73,"title":["Neonatal hypoxic\/ischemic brain injury induces production of calretinin\u2010expressing interneurons in the striatum"],"prefix":"10.1002","volume":"511","author":[{"given":"Zhengang","family":"Yang","sequence":"first","affiliation":[],"role":[{"vocabulary":"crossref","role":"author"}]},{"given":"Yan","family":"You","sequence":"additional","affiliation":[],"role":[{"vocabulary":"crossref","role":"author"}]},{"given":"Steven 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