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The possibility exists that SARS\u2010CoV\u20102 could induce diabetes, but it is not yet clear whether this might be a fulminant\u2010type diabetes, autoimmune diabetes, or a new\u2010onset transient hyperglycaemia. This viewpoint discusses mechanisms by which SARS\u2010CoV\u20102 might trigger type 1 diabetes mellitus (T1DM). Specifically, we looked at the role of post\u2010translational protein modifications (PTMs) and the generation of neoepitopes as a potential mechanism in the induction of islet autoimmunity, and the pathways via which coronavirus infections might exacerbate the formation of PTMs and, in so doing, provoke the onset of T1DM.<\/jats:p>","DOI":"10.1002\/dmrr.3508","type":"journal-article","created":{"date-parts":[[2022,1,6]],"date-time":"2022-01-06T14:40:07Z","timestamp":1641480007000},"update-policy":"https:\/\/doi.org\/10.1002\/crossmark_policy","source":"Crossref","is-referenced-by-count":18,"title":["SARS\u2010CoV\u20102 induced post\u2010translational protein modifications: A trigger for developing autoimmune diabetes?"],"prefix":"10.1002","volume":"38","author":[{"ORCID":"https:\/\/orcid.org\/0000-0001-8401-719X","authenticated-orcid":false,"given":"Chaplin","family":"Catriona","sequence":"first","affiliation":[{"name":"Queen Mary University of London  London 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