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Transfer of spleen cells from convalescent animals, after circulating anti\u2010GBM autoantibodies are no longer detectable, inhibits reinduction of the disease by HgCl<jats:sub>2<\/jats:sub> in naive recipients. This inhibition is significantly less when the T suppressor cell population is depleted by the monoclonal antibody, MRCOX8, before transfer. Our studies suggest a role for T suppressor cells in autoregulation in this animal model of autoimmune nephritis and may form a basis for the design of specific therapy for anti\u2010GBM disease in man.<\/jats:p>","DOI":"10.1002\/eji.1830140515","type":"journal-article","created":{"date-parts":[[2007,2,28]],"date-time":"2007-02-28T21:16:21Z","timestamp":1172697381000},"page":"464-470","source":"Crossref","is-referenced-by-count":58,"title":["Autoregulation of autoantibody synthesis in mercuric chloride nephritis in the Brown Norway rat I. 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