{"status":"ok","message-type":"work","message-version":"1.0.0","message":{"indexed":{"date-parts":[[2026,5,16]],"date-time":"2026-05-16T03:02:18Z","timestamp":1778900538972,"version":"3.51.4"},"reference-count":25,"publisher":"Wiley","issue":"4","license":[{"start":{"date-parts":[[2003,1,30]],"date-time":"2003-01-30T00:00:00Z","timestamp":1043884800000},"content-version":"vor","delay-in-days":0,"URL":"http:\/\/onlinelibrary.wiley.com\/termsAndConditions#vor"}],"content-domain":{"domain":[],"crossmark-restriction":false},"short-container-title":["Genes Chromosomes &amp;amp; Cancer"],"published-print":{"date-parts":[[2003,4]]},"abstract":"<jats:title>Abstract<\/jats:title><jats:p>Recently, it was shown that t(10;16)(q22;p13) fuses the <jats:italic>MORF<\/jats:italic> and <jats:italic>CREBBP<\/jats:italic> genes in a case of childhood acute myeloid leukemia (AML) M5a, with a complex karyotype containing other rearrangements. Here, we report a new case with the <jats:italic>MORF<\/jats:italic>\u2010<jats:italic>CREBBP<\/jats:italic> fusion in an 84\u2010year\u2010old patient diagnosed with AML M5b, in which the t(10;16)(q22;p13) was the only cytogenetic aberration. This supports that this is a recurrent pathogenic translocation in AML. \u00a9 2003 Wiley\u2010Liss, Inc.<\/jats:p>","DOI":"10.1002\/gcc.10174","type":"journal-article","created":{"date-parts":[[2003,3,18]],"date-time":"2003-03-18T22:54:05Z","timestamp":1048028045000},"page":"402-405","source":"Crossref","is-referenced-by-count":25,"title":["t(10;16)(q22;p13) and <i>MORF<\/i>\u2010<i>CREBBP<\/i> fusion is a recurrent event in acute myeloid leukemia"],"prefix":"10.1002","volume":"36","author":[{"given":"Jos\u00e9 L.","family":"Vizmanos","sequence":"first","affiliation":[],"role":[{"role":"author","vocabulary":"crossref"}]},{"given":"Mar\u00eda 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