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There is also an increased incidence of psychiatric diagnosis, especially intellectual disability and ADHD in childhood, lifelong anxiety, and a strikingly high rate of schizophrenia spectrum disorders, which occur in around 30% of adults with 22q11DS. Using innovative computational connectomics, we studied how 22q11DS affects high\u2010level network signatures of hierarchical modularity and its intrinsic geometry in 55 children with confirmed 22q11DS and 27 Typically Developing (TD) children. Results identified 3 subgroups within our 22q11DS sample using a K\u2010means clustering approach based on several midline structural measures\u2010of\u2010interests. Each subgroup exhibited distinct patterns of connectome abnormalities. Subtype 1, containing individuals with generally healthy\u2010looking brains, exhibited no significant differences in either modularity or intrinsic geometry when compared with TD. By contrast, the more anomalous 22q11DS Subtypes 2 and 3 brains revealed significant modular differences in the right hemisphere, while Subtype 3 (the most anomalous anatomy) further exhibited significantly abnormal connectome intrinsic geometry in the form of left\u2013right temporal disintegration. Taken together, our findings supported an overall picture of (a) anterior\u2010posteriorly differential interlobar frontotemporal\/frontoparietal dysconnectivity in Subtypes 2 and 3 and (b) differential intralobar dysconnectivity in Subtype 3. Our ongoing studies are focusing on whether these subtypes and their connnectome signatures might be valid biomarkers for predicting the degree of psychosis\u2010proneness risk found in 22q11DS. <jats:italic>Hum Brain Mapp 39:232\u2013248, 2018<\/jats:italic>. \u00a9 <jats:bold>2017 Wiley Periodicals, Inc.<\/jats:bold><\/jats:p>","DOI":"10.1002\/hbm.23838","type":"journal-article","created":{"date-parts":[[2017,10,9]],"date-time":"2017-10-09T04:22:36Z","timestamp":1507522956000},"page":"232-248","source":"Crossref","is-referenced-by-count":11,"title":["Baseline connectome modular abnormalities in the childhood phase of a longitudinal study on individuals with chromosome 22q11.2 deletion syndrome"],"prefix":"10.1002","volume":"39","author":[{"ORCID":"https:\/\/orcid.org\/0000-0002-7920-4828","authenticated-orcid":false,"given":"Liang","family":"Zhan","sequence":"first","affiliation":[{"name":"Computer Engineering Program University of Wisconsin\u2010Stout  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