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This channel is highly expressed in numerous cancers and has been implicated in the regulation of proliferation and migration. The molecular details of the signaling pathways linked to regulation exerted by K\n                    <jats:sub>Ca<\/jats:sub>\n                    3.1 in cancer cells are, however, not fully elucidated yet. Therefore, we determined the interactome of K\n                    <jats:sub>Ca<\/jats:sub>\n                    3.1 using proximity labeling in intact KPC pancreatic cancer cells that mirror the aggressive metastatic behavior of human pancreatic cancer. The results highlight several novel interactors, including those residing in intracellular membranes. The K\n                    <jats:sub>Ca<\/jats:sub>\n                    3.1 channel proxisome and related pathways are discussed in light of our current knowledge about K\n                    <jats:sub>Ca<\/jats:sub>\n                    3.1 and pancreatic cancer, available in public databases.\n                  <\/jats:p>","DOI":"10.1002\/jcp.70092","type":"journal-article","created":{"date-parts":[[2025,9,15]],"date-time":"2025-09-15T13:20:28Z","timestamp":1757942428000},"update-policy":"https:\/\/doi.org\/10.1002\/crossmark_policy","source":"Crossref","is-referenced-by-count":3,"title":["Intermediate Conductance Calcium\u2010Dependent Potassium Channel (K\n                    <sub>Ca<\/sub>\n                    3.1) Interacting Proteins Using Turboid\u2010Based Proximity Labeling Technology: Insights Into Interactome and Related Signaling Pathways in Pancreatic 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