{"status":"ok","message-type":"work","message-version":"1.0.0","message":{"indexed":{"date-parts":[[2026,4,7]],"date-time":"2026-04-07T00:03:44Z","timestamp":1775520224062,"version":"3.50.1"},"reference-count":20,"publisher":"Wiley","issue":"9","license":[{"start":{"date-parts":[[2020,6,21]],"date-time":"2020-06-21T00:00:00Z","timestamp":1592697600000},"content-version":"vor","delay-in-days":0,"URL":"http:\/\/onlinelibrary.wiley.com\/termsAndConditions#vor"}],"funder":[{"DOI":"10.13039\/100000002","name":"National Institutes of Health","doi-asserted-by":"publisher","award":["UL1 TR001860"],"award-info":[{"award-number":["UL1 TR001860"]}],"id":[{"id":"10.13039\/100000002","id-type":"DOI","asserted-by":"publisher"}]}],"content-domain":{"domain":["onlinelibrary.wiley.com"],"crossmark-restriction":true},"short-container-title":["J of Clinical Ultrasound"],"published-print":{"date-parts":[[2020,11]]},"abstract":"<jats:title>Abstract<\/jats:title><jats:sec><jats:title>Objective<\/jats:title><jats:p>The aim of the study was to review the different etiologies and outcomes of patients with hepatic artery velocities greater than 200\u2009cm\/s.<\/jats:p><\/jats:sec><jats:sec><jats:title>Methods<\/jats:title><jats:p>This retrospective study included 88 hospitalized patients in whom angle\u2010corrected proper hepatic artery flow velocities greater than 200\u2009cm\/s were obtained during an abdominal ultrasonographic examination. Peak systolic hepatic artery flow velocities, hepatic artery resistance index, and portal vein flow velocities were evaluated. The patients were then allocated to one of four groups based on their primary underlying diagnosis: structural liver disease, nonstructural liver disease, generalized infection, or miscellaneous.<\/jats:p><\/jats:sec><jats:sec><jats:title>Results<\/jats:title><jats:p>The median hepatic artery velocity was similar for all groups, ranging from 226 to 238\u2009cm\/s<jats:italic>.<\/jats:italic> The <jats:italic>maximum<\/jats:italic> portal venous velocities were not significantly different between groups. No lab values were statistically different between the groups, except total bilirubin that was greater in the nonstructural liver disease group (8 mg\/dL). Overall, 9\/88 (10.2%) of patients with elevated hepatic artery velocity died within 30\u2009days of their ultrasonographic examination.<\/jats:p><\/jats:sec><jats:sec><jats:title>Conclusion<\/jats:title><jats:p>Elevated hepatic artery velocity greater than 200\u2009cm\/s in hospitalized patients is not specific to primary hepatobiliary disease but may indicate acute hepatic dysfunction from other causes such as infection or sepsis.<\/jats:p><\/jats:sec>","DOI":"10.1002\/jcu.22885","type":"journal-article","created":{"date-parts":[[2020,6,22]],"date-time":"2020-06-22T05:27:16Z","timestamp":1592803636000},"page":"532-537","update-policy":"https:\/\/doi.org\/10.1002\/crossmark_policy","source":"Crossref","is-referenced-by-count":4,"title":["Markedly elevated hepatic arterial velocity\u2014HAV greater than 200\u2009cm\/s\u2014is not specific to hepatobiliary disease"],"prefix":"10.1002","volume":"48","author":[{"given":"Michael 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