{"status":"ok","message-type":"work","message-version":"1.0.0","message":{"indexed":{"date-parts":[[2026,4,1]],"date-time":"2026-04-01T20:15:48Z","timestamp":1775074548148,"version":"3.50.1"},"reference-count":41,"publisher":"Wiley","issue":"1","license":[{"start":{"date-parts":[[2018,1,15]],"date-time":"2018-01-15T00:00:00Z","timestamp":1515974400000},"content-version":"vor","delay-in-days":14,"URL":"http:\/\/creativecommons.org\/licenses\/by\/4.0\/"}],"funder":[{"DOI":"10.13039\/501100000265","name":"Medical Research Council","doi-asserted-by":"publisher","award":["MR\/J002380\/1"],"award-info":[{"award-number":["MR\/J002380\/1"]}],"id":[{"id":"10.13039\/501100000265","id-type":"DOI","asserted-by":"publisher"}]},{"DOI":"10.13039\/501100000278","name":"Department for International Development","doi-asserted-by":"publisher","id":[{"id":"10.13039\/501100000278","id-type":"DOI","asserted-by":"publisher"}]},{"DOI":"10.13039\/100000002","name":"National Institutes of Health","doi-asserted-by":"publisher","award":["K08 AI104352"],"award-info":[{"award-number":["K08 AI104352"]}],"id":[{"id":"10.13039\/100000002","id-type":"DOI","asserted-by":"publisher"}]},{"DOI":"10.13039\/100000002","name":"National Institutes of Health","doi-asserted-by":"publisher","award":["NIH P30 AI 54999"],"award-info":[{"award-number":["NIH P30 AI 54999"]}],"id":[{"id":"10.13039\/100000002","id-type":"DOI","asserted-by":"publisher"}]},{"DOI":"10.13039\/100000002","name":"National Institutes of Health","doi-asserted-by":"publisher","award":["NIH P30 AI110527"],"award-info":[{"award-number":["NIH P30 AI110527"]}],"id":[{"id":"10.13039\/100000002","id-type":"DOI","asserted-by":"publisher"}]},{"DOI":"10.13039\/100000002","name":"National Institutes of Health","doi-asserted-by":"publisher","award":["NIH K 24 AI065298"],"award-info":[{"award-number":["NIH K 24 AI065298"]}],"id":[{"id":"10.13039\/100000002","id-type":"DOI","asserted-by":"publisher"}]},{"DOI":"10.13039\/501100003323","name":"Agence Nationale de Recherches sur le Sida et les H\u00e9patites Virales","doi-asserted-by":"publisher","id":[{"id":"10.13039\/501100003323","id-type":"DOI","asserted-by":"publisher"}]},{"DOI":"10.13039\/501100004954","name":"Augustinus Fonden","doi-asserted-by":"publisher","id":[{"id":"10.13039\/501100004954","id-type":"DOI","asserted-by":"publisher"}]},{"DOI":"10.13039\/501100004963","name":"Seventh Framework Programme","doi-asserted-by":"publisher","award":["FP7\/2007\u20102013"],"award-info":[{"award-number":["FP7\/2007\u20102013"]}],"id":[{"id":"10.13039\/501100004963","id-type":"DOI","asserted-by":"publisher"}]},{"DOI":"10.13039\/501100000272","name":"National Institute for Health Research","doi-asserted-by":"publisher","award":["NF\u2010SI\u20100611\u201010168"],"award-info":[{"award-number":["NF\u2010SI\u20100611\u201010168"]}],"id":[{"id":"10.13039\/501100000272","id-type":"DOI","asserted-by":"publisher"}]}],"content-domain":{"domain":["onlinelibrary.wiley.com"],"crossmark-restriction":true},"short-container-title":["J Intern AIDS Soc"],"published-print":{"date-parts":[[2018,1]]},"abstract":"Abstract<\/jats:title>\n \n Introduction<\/jats:title>\n \n HIV<\/jats:styled-content>\n \u20101 infection leads to chronic inflammation and to an increased risk of non\u2010\n AIDS<\/jats:styled-content>\n mortality. Our objective was to determine whether\n AIDS<\/jats:styled-content>\n \u2010defining events (\n ADE<\/jats:styled-content>\n s) were associated with increased overall and cause\u2010specific non\u2010\n AIDS<\/jats:styled-content>\n related mortality after antiretroviral therapy (\n ART<\/jats:styled-content>\n ) initiation.\n <\/jats:p>\n <\/jats:sec>\n \n Methods<\/jats:title>\n \n We included\n HIV<\/jats:styled-content>\n treatment\u2010na\u00efve adults from the Antiretroviral Therapy Cohort Collaboration (\n ART<\/jats:styled-content>\n \u2010\n CC<\/jats:styled-content>\n ) who initiated\n ART<\/jats:styled-content>\n from 1996 to 2014. Causes of death were assigned using the Coding Causes of Death in\n HIV<\/jats:styled-content>\n (CoDe) protocol. The adjusted hazard ratio (\n aHR<\/jats:styled-content>\n ) for overall and cause\u2010specific non\u2010\n AIDS<\/jats:styled-content>\n mortality among those with an\n ADE<\/jats:styled-content>\n (all\n ADE<\/jats:styled-content>\n s, tuberculosis (\n TB<\/jats:styled-content>\n ),\n Pneumocystis jiroveci<\/jats:italic>\n pneumonia (\n PJP<\/jats:styled-content>\n ), and non\u2010Hodgkin's lymphoma (\n NHL<\/jats:styled-content>\n )) compared to those without an\n ADE<\/jats:styled-content>\n was estimated using a marginal structural model.\n <\/jats:p>\n <\/jats:sec>\n \n Results<\/jats:title>\n \n The adjusted hazard of overall non\u2010\n AIDS<\/jats:styled-content>\n mortality was higher among those with any\n ADE<\/jats:styled-content>\n compared to those without any\n ADE<\/jats:styled-content>\n (\n aHR<\/jats:styled-content>\n 2.21, 95% confidence interval (\n CI<\/jats:styled-content>\n ) 2.00 to 2.43). The adjusted hazard of each of the cause\u2010specific non\u2010\n AIDS<\/jats:styled-content>\n related deaths were higher among those with any\n ADE<\/jats:styled-content>\n compared to those without, except metabolic deaths (malignancy\n aHR<\/jats:styled-content>\n 2.59 (95%\n CI<\/jats:styled-content>\n 2.13 to 3.14), accident\/suicide\/overdose\n aHR<\/jats:styled-content>\n 1.37 (95%\n CI<\/jats:styled-content>\n 1.05 to 1.79), cardiovascular\n aHR<\/jats:styled-content>\n 1.95 (95%\n CI<\/jats:styled-content>\n 1.54 to 2.48), infection\n aHR<\/jats:styled-content>\n (95%\n CI<\/jats:styled-content>\n 1.68 to 2.81), hepatic\n aHR<\/jats:styled-content>\n 2.09 (95%\n CI<\/jats:styled-content>\n 1.61 to 2.72), respiratory\n aHR<\/jats:styled-content>\n 4.28 (95%\n CI<\/jats:styled-content>\n 2.67 to 6.88), renal\n aHR<\/jats:styled-content>\n 5.81 (95%\n CI<\/jats:styled-content>\n 2.69 to 12.56) and central nervous\n aHR<\/jats:styled-content>\n 1.53 (95%\n CI<\/jats:styled-content>\n 1.18 to 5.44)). The risk of overall and cause\u2010specific non\u2010\n AIDS<\/jats:styled-content>\n mortality differed depending on the specific\n ADE<\/jats:styled-content>\n of interest (TB, PJP, NHL).\n <\/jats:p>\n <\/jats:sec>\n \n Conclusions<\/jats:title>\n \n In this large multi\u2010centre cohort collaboration with standardized assignment of causes of death, non\u2010\n AIDS<\/jats:styled-content>\n mortality was twice as high among patients with an\n ADE<\/jats:styled-content>\n compared to without an\n ADE<\/jats:styled-content>\n . However, non\u2010\n AIDS<\/jats:styled-content>\n related mortality after an\n ADE<\/jats:styled-content>\n depended on the\n ADE<\/jats:styled-content>\n of interest. Although there may be unmeasured confounders, these findings suggest that a common pathway may be independently driving both\n ADE<\/jats:styled-content>\n s and\n NADE<\/jats:styled-content>\n mortality. While prevention of\n ADE<\/jats:styled-content>\n s may reduce subsequent death due to\n NADE<\/jats:styled-content>\n s following\n ART<\/jats:styled-content>\n initiation, modification of risk factors for\n NADE<\/jats:styled-content>\n mortality remains important after\n ADE<\/jats:styled-content>\n survival.\n <\/jats:p>\n <\/jats:sec>","DOI":"10.1002\/jia2.25031","type":"journal-article","created":{"date-parts":[[2018,1,16]],"date-time":"2018-01-16T07:06:28Z","timestamp":1516086388000},"update-policy":"https:\/\/doi.org\/10.1002\/crossmark_policy","source":"Crossref","is-referenced-by-count":49,"title":["Increased non\u2010\n AIDS<\/scp>\n mortality among persons with\n AIDS<\/scp>\n \u2010defining events after antiretroviral therapy initiation"],"prefix":"10.1002","volume":"21","author":[{"ORCID":"https:\/\/orcid.org\/0000-0001-8832-0866","authenticated-orcid":false,"given":"April C","family":"Pettit","sequence":"first","affiliation":[{"name":"Division of Infectious Diseases 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