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In addition, some\n STI<\/jats:styled-content>\n s can increase blood\n HIV<\/jats:styled-content>\n concentration and promote progression of disease. This review is designed to investigate the complex relationship between\n HIV<\/jats:styled-content>\n and classical\n STI<\/jats:styled-content>\n s.\n <\/jats:p>\n <\/jats:sec>\n \n Discussion<\/jats:title>\n \n Treatment of\n STI<\/jats:styled-content>\n s with appropriate antibiotics reduces\n HIV<\/jats:styled-content>\n in blood, semen and female genital secretions. However, community\u2010based trials could not reliably reduce the spread of\n HIV<\/jats:styled-content>\n by mass treatment of\n STI<\/jats:styled-content>\n s. Introduction of antiretroviral agents for the treatment and prevention of\n HIV<\/jats:styled-content>\n has led to renewed interest in the complex relationship between\n STI<\/jats:styled-content>\n s and\n HIV<\/jats:styled-content>\n . Antiretroviral treatment (\n ART<\/jats:styled-content>\n ) reduces the infectiousness of\n HIV<\/jats:styled-content>\n and virtually eliminates the transmission of\n HIV<\/jats:styled-content>\n in spite of concomitant or acquired\n STI<\/jats:styled-content>\n s. However, while\n ART<\/jats:styled-content>\n interrupts\n HIV<\/jats:styled-content>\n transmission, it does not stop intermittent shedding of\n HIV<\/jats:styled-content>\n in genital secretions. Such shedding of\n HIV<\/jats:styled-content>\n is increased by\n STI<\/jats:styled-content>\n s, although the viral copies are not likely replication competent or infectious. Pre\u2010exposure prophylaxis (Pr\n EP<\/jats:styled-content>\n ) of\n HIV<\/jats:styled-content>\n with the combination of tenofovir disoproxil fumarate and emtricitabine (\n TDF<\/jats:styled-content>\n \/\n FTC<\/jats:styled-content>\n ) prevents\n HIV<\/jats:styled-content>\n acquisition in spite of concomitant\n STI<\/jats:styled-content>\n s.\n <\/jats:p>\n <\/jats:sec>\n \n Conclusions<\/jats:title>\n \n STI<\/jats:styled-content>\n s remain pandemic, and the availability of\n ART<\/jats:styled-content>\n may have led to an increase in\n STI<\/jats:styled-content>\n s, as fear of\n HIV<\/jats:styled-content>\n has diminished. Classical\n STI<\/jats:styled-content>\n s present a huge worldwide health burden that cannot be separated from\n HIV<\/jats:styled-content>\n , and they deserve far more attention than they currently receive.\n <\/jats:p>\n <\/jats:sec>","DOI":"10.1002\/jia2.25355","type":"journal-article","created":{"date-parts":[[2019,8,30]],"date-time":"2019-08-30T03:01:46Z","timestamp":1567134106000},"update-policy":"https:\/\/doi.org\/10.1002\/crossmark_policy","source":"Crossref","is-referenced-by-count":139,"title":["Sexually transmitted infections and\n HIV<\/scp>\n in the era of antiretroviral treatment and prevention: the biologic basis for epidemiologic synergy"],"prefix":"10.1002","volume":"22","author":[{"given":"Myron S","family":"Cohen","sequence":"first","affiliation":[{"name":"UNC School of Medicine Institute for Global Health & Infectious Diseases Chapel Hill NC USA"}],"role":[{"vocabulary":"crossref","role":"author"}]},{"given":"Olivia 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