{"status":"ok","message-type":"work","message-version":"1.0.0","message":{"indexed":{"date-parts":[[2026,8,17]],"date-time":"2026-08-17T23:31:27Z","timestamp":1787009487734,"version":"build-2736575974"},"reference-count":28,"publisher":"Wiley","issue":"2","license":[{"start":{"date-parts":[[2005,11,17]],"date-time":"2005-11-17T00:00:00Z","timestamp":1132185600000},"content-version":"vor","delay-in-days":2818,"URL":"http:\/\/onlinelibrary.wiley.com\/termsAndConditions#vor"}],"content-domain":{"domain":[],"crossmark-restriction":false},"short-container-title":["Magnetic Resonance Imaging"],"published-print":{"date-parts":[[1998,3]]},"abstract":"<jats:title>Abstract<\/jats:title><jats:p>We describe MR findings in patients with Minamata disease who have been followed for a long time. All patients examined were affected after daily eating of a large quantity of methylmercury\u2010contaminated seafood, from 1955 to 1958, and showed typical neurological findings. On MR images, the visual cortex, the cerebellar vermis and hemispheres, and the postcentral cortex are significantly atrophic in Minamata disease. The visual cortex is slightly hypointense on T1\u2010weighted images and hyperintense on T2\u2010weighted images, probably representing the pathologic changes of status spongiosus. MRI can demonstrate the lesions located in the calcarine area, cerebellum, and postcentral gyri, which are probably related to three of the characteristic manifestations of this disease: the constriction of the visual fields, ataxia, and sensory disturbance, respectively.<\/jats:p>","DOI":"10.1002\/jmri.1880080210","type":"journal-article","created":{"date-parts":[[2007,3,16]],"date-time":"2007-03-16T12:35:54Z","timestamp":1174048554000},"page":"308-316","source":"Crossref","is-referenced-by-count":53,"title":["Invited. MR findings of Minamata disease \u2014 Organic mercury poisoning"],"prefix":"10.1002","volume":"8","author":[{"given":"Yukunori","family":"Korogi","sequence":"first","affiliation":[],"role":[{"vocabulary":"crossref","role":"author"}]},{"given":"Mutsumasa","family":"Takahashi","sequence":"additional","affiliation":[],"role":[{"vocabulary":"crossref","role":"author"}]},{"given":"Toru","family":"Okajima","sequence":"additional","affiliation":[],"role":[{"vocabulary":"crossref","role":"author"}]},{"given":"Komyo","family":"Eto","sequence":"additional","affiliation":[],"role":[{"vocabulary":"crossref","role":"author"}]}],"member":"311","published-online":{"date-parts":[[2005,11,17]]},"reference":[{"key":"e_1_2_1_2_2","first-page":"536","article-title":"Minamata disease","volume":"2","author":"Tokuomi H","year":"1961","journal-title":"World Neurol"},{"key":"e_1_2_1_3_2","doi-asserted-by":"publisher","DOI":"10.1212\/WNL.32.12.1369"},{"key":"e_1_2_1_4_2","doi-asserted-by":"publisher","DOI":"10.1007\/BF00341942"},{"key":"e_1_2_1_5_2","first-page":"1575","article-title":"MR findings in seven patients with organic mercury poisoning (Minamata Disease)","volume":"15","author":"Korogi Y","year":"1994","journal-title":"Am J Neuroradiol"},{"key":"e_1_2_1_6_2","doi-asserted-by":"publisher","DOI":"10.1007\/BF00685743"},{"key":"e_1_2_1_7_2","first-page":"207","volume-title":"Recent advances in Minamata disease studies. 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