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LV remodeling was evaluated as LV end\u2010diastolic volume change index between two scans using cine. CD68 staining was conducted to detect monocyte\/macrophage.<\/jats:p>\n                  <\/jats:sec>\n                  <jats:sec>\n                    <jats:title>Statistical Tests<\/jats:title>\n                    <jats:p>\n                      Student\u2010\n                      <jats:italic>t<\/jats:italic>\n                      test and one\u2010way ANOVA were used to compare remote T2 with normal controls. The association of remote T2 with LV remodeling was assessed using linear regression.\n                      <jats:italic>P<\/jats:italic>\n                      values of &lt;0.05 were used to denote statistical significance.\n                    <\/jats:p>\n                  <\/jats:sec>\n                  <jats:sec>\n                    <jats:title>Results<\/jats:title>\n                    <jats:p>\n                      Compared with healthy pigs, remote T2 significantly increased from 3\u2009days to 3\u00a0months post\u2010MI (31.43\u2009\u00b1\u20090.67 vs. 33.53\u2009\u00b1\u20091.15 vs. 36.43\u2009\u00b1\u20091.07\u2009msec). CD68 staining demonstrated the inflammation in remote myocardium post\u2010MI but not in healthy pigs. Significant remote myocardial alterations in T2 were also observed in human group (40.51\u2009\u00b1\u20091.79 vs. 41.94\u2009\u00b1\u20091.14 vs. 42.52\u2009\u00b1\u20091.71\u2009msec). In patients, the 3\u2010month remote T2 (\n                      <jats:italic>\u03b2<\/jats:italic>\n                      \u00a0=\u00a00.432) and remote T2 variation between two scans (\n                      <jats:italic>\u03b2<\/jats:italic>\n                      \u00a0=\u00a00.554) were both independently associated with LV remodeling.\n                    <\/jats:p>\n                  <\/jats:sec>\n                  <jats:sec>\n                    <jats:title>Conclusion<\/jats:title>\n                    <jats:p>T2 mapping could characterize the abnormalities in the remote myocardium post\u2010MI, which was potentially caused by the inflammatory response. Moreover, variations in remote T2 were associated with LV remodeling.<\/jats:p>\n                  <\/jats:sec>\n                  <jats:sec>\n                    <jats:title>Evidence Level<\/jats:title>\n                    <jats:p>1<\/jats:p>\n                  <\/jats:sec>\n                  <jats:sec>\n                    <jats:title>Technical Efficacy<\/jats:title>\n                    <jats:p>Stage 3<\/jats:p>\n                  <\/jats:sec>","DOI":"10.1002\/jmri.27827","type":"journal-article","created":{"date-parts":[[2021,7,9]],"date-time":"2021-07-09T22:23:50Z","timestamp":1625869430000},"page":"555-564","update-policy":"https:\/\/doi.org\/10.1002\/crossmark_policy","source":"Crossref","is-referenced-by-count":18,"title":["Inflammation in Remote Myocardium and Left Ventricular Remodeling After Acute Myocardial Infarction: A Pilot Study Using\n                    <scp>T2<\/scp>\n                    Mapping"],"prefix":"10.1002","volume":"55","author":[{"given":"Meng\u2010xi","family":"Yang","sequence":"first","affiliation":[{"name":"Department of Radiology, State Key Laboratory of Biotherapy, West China Hospital Sichuan University  Sichuan China"}],"role":[{"vocabulary":"crossref","role":"author"}]},{"given":"Ke","family":"Shi","sequence":"additional","affiliation":[{"name":"Department of Radiology, West China Hospital Sichuan University  Sichuan China"}],"role":[{"vocabulary":"crossref","role":"author"}]},{"given":"Hua\u2010yan","family":"Xu","sequence":"additional","affiliation":[{"name":"Department of Radiology, Key Laboratory of Obstetric &amp; Gynecologic and Pediatric Diseases and Birth Defects of Ministry of Education, West China Second University Hospital Sichuan University  Sichuan China"}],"role":[{"vocabulary":"crossref","role":"author"}]},{"given":"Yong","family":"He","sequence":"additional","affiliation":[{"name":"Department of Cardiology, West China Hospital Sichuan 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