{"status":"ok","message-type":"work","message-version":"1.0.0","message":{"indexed":{"date-parts":[[2026,9,17]],"date-time":"2026-09-17T04:52:04Z","timestamp":1789620724156,"version":"build-2803163510"},"reference-count":24,"publisher":"Wiley","issue":"5","license":[{"start":{"date-parts":[[2003,12,17]],"date-time":"2003-12-17T00:00:00Z","timestamp":1071619200000},"content-version":"vor","delay-in-days":0,"URL":"http:\/\/onlinelibrary.wiley.com\/termsAndConditions#vor"}],"content-domain":{"domain":[],"crossmark-restriction":false},"short-container-title":["Movement Disorders"],"published-print":{"date-parts":[[2004,5]]},"abstract":"<jats:title>Abstract<\/jats:title><jats:p>We studied 516 globus pallidus neurons in dystonic patients. The firing rate was analysed. We classified the burst activity into tonic, burst, and pause patterns. Mean \u00b1 SD firing rates and tonicity score for internal globus pallidus (GPi) and external globus pallidus (GPe) were 54.6 \u00b1 28.6; 58.01 \u00b1 39.1 and 1.18 \u00b1 0.55; 0.95 \u00b1 0.43, respectively. Differences in percentage appearance of tonic, burst, or paused neurons were not statistically significant for GPi versus GPe. GPi firing features in dystonic patients were closely similar to those of GPe. This could suggest that the abnormally patterned output from GPi would not result from increased differential inhibitory\/excitatory input arising from the direct\/indirect pathway but rather be transmitted from GPe, striatum, or either centromedian nucleus. \u00a9 2003 Movement Disorder Society<\/jats:p>","DOI":"10.1002\/mds.10700","type":"journal-article","created":{"date-parts":[[2004,5,3]],"date-time":"2004-05-03T18:01:30Z","timestamp":1083607290000},"page":"548-554","source":"Crossref","is-referenced-by-count":43,"title":["Neuronal globus pallidus activity in patients with generalised 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