{"status":"ok","message-type":"work","message-version":"1.0.0","message":{"indexed":{"date-parts":[[2026,3,13]],"date-time":"2026-03-13T09:29:13Z","timestamp":1773394153826,"version":"3.50.1"},"reference-count":17,"publisher":"Wiley","issue":"1","license":[{"start":{"date-parts":[[2003,11,4]],"date-time":"2003-11-04T00:00:00Z","timestamp":1067904000000},"content-version":"vor","delay-in-days":0,"URL":"http:\/\/onlinelibrary.wiley.com\/termsAndConditions#vor"}],"content-domain":{"domain":[],"crossmark-restriction":false},"short-container-title":["Muscle and Nerve"],"published-print":{"date-parts":[[2004,1]]},"abstract":"<jats:title>Abstract<\/jats:title><jats:p>A series of eight patients with isolated clinical and electrophysiological sensory deficit related to neuralgic amyotrophy (NA) is reported. NA was diagnosed by clinical and electrodiagnostic features and disease course. Imaging and laboratory investigations excluded other disorders. The results showed mild to severe involvement of eight individual sensory nerves: lateral antebrachial cutaneous nerve lesions in three instances and partial lesions of the median nerve in five cases. Our findings suggest that isolated clinical and electrodiagnostic sensory involvement in NA is not exceptional but rather is unrecognized. The pattern of these nerve lesions agrees with the most typical pattern of NA, which is a mononeuritis or mononeuritis multiplex. The present study also shows that the spectrum of NA is diverse and may vary from pure motor to pure sensory deficit, according to the nature of the involved nerve fibers. 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