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In this study we report the genomic structure and mutation analysis of three candidate genes: sphingosine kinase 1 (<jats:italic>SPHK1<\/jats:italic>); tissue inhibitor of metalloproteinase 2 (<jats:italic>TIMP2<\/jats:italic>); and cytoglobin (<jats:italic>CYGB<\/jats:italic>). We did not find any disease\u2010associated mutations, indicating that HNA is not caused by point mutations in these genes. However, we identified several sequencing errors in the cDNA of <jats:italic>SPHK1<\/jats:italic> as well as seven novel single\u2010nucleotide polymorphisms. 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