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MFS can be associated with infectious, autoimmune, and neoplastic disorders. Radiological findings have suggested both central and peripheral involvement. The anti\u2010GQ1b IgG antibody titer is most commonly elevated in MFS, but may also be increased in Guillain\u2013Barr\u00e9 syndrome (GBS) and Bickerstaff's brainstem encephalitis (BBE). Molecular mimicry, particularly in relation to antecedent <jats:italic>Campylobacter jejuni<\/jats:italic> and <jats:italic>Hemophilus influenzae<\/jats:italic> infections, is likely the predominant pathogenic mechanism, but the roles of other biological factors remain to be established. Recent studies have demonstrated the presence of neuromuscular transmission defects in association with anti\u2010GQ1b IgG antibody, both in vitro and in vivo. Collective findings from clinical, radiological, immunological, and electrophysiological techniques have helped to define MFS, GBS, and BBE as major disorders within the proposed spectrum of anti\u2010GQ1b IgG antibody syndrome. Muscle Nerve, 2007<\/jats:p>","DOI":"10.1002\/mus.20835","type":"journal-article","created":{"date-parts":[[2007,7,26]],"date-time":"2007-07-26T19:46:32Z","timestamp":1185479192000},"page":"615-627","source":"Crossref","is-referenced-by-count":134,"title":["Clinical and immunological spectrum of the Miller Fisher syndrome"],"prefix":"10.1002","volume":"36","author":[{"given":"Y. 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