{"status":"ok","message-type":"work","message-version":"1.0.0","message":{"indexed":{"date-parts":[[2023,10,4]],"date-time":"2023-10-04T05:40:37Z","timestamp":1696398037458},"reference-count":26,"publisher":"Wiley","issue":"2","license":[{"start":{"date-parts":[[2013,7,3]],"date-time":"2013-07-03T00:00:00Z","timestamp":1372809600000},"content-version":"vor","delay-in-days":0,"URL":"http:\/\/onlinelibrary.wiley.com\/termsAndConditions#vor"}],"content-domain":{"domain":[],"crossmark-restriction":false},"short-container-title":["Muscle and Nerve"],"published-print":{"date-parts":[[2014,2]]},"abstract":"<jats:title>ABSTRACT<\/jats:title><jats:p><jats:italic>Introduction<\/jats:italic>: Standard electromyography (EMG) is useful in the diagnosis of myotonic dystrophy type 1 (DM1) and type 2 (DM2), but it does not differentiate between them. The aim of this study was to estimate the utility of the short exercise test (SET) and short exercise test with cooling (SETC) in differentiating between DM1 and DM2. <jats:italic>Methods<\/jats:italic>: SET and SETC were performed in 32 patients with DM1 (mean age 35.8\u2009\u00b1\u200912.7 years) and 28 patients with DM2 (mean age 44.5\u2009\u00b1\u200912.5 years). <jats:italic>Results<\/jats:italic>: We observed a significant decline in compound motor action potential (CMAP) amplitude in DM1 with both SET and SETC immediately after effort. In DM2, there was no marked change in CMAP amplitude with either SET or SETC. <jats:italic>Conclusions<\/jats:italic>: SET and SETC may serve as useful tools for clinical differentiation between DM1 and DM2, and they may be used as a guide for molecular testing. <jats:italic>Muscle Nerve<\/jats:italic> <jats:bold>49<\/jats:bold>: 277\u2013283, 2014<\/jats:p>","DOI":"10.1002\/mus.23908","type":"journal-article","created":{"date-parts":[[2013,5,29]],"date-time":"2013-05-29T06:46:08Z","timestamp":1369809968000},"page":"277-283","source":"Crossref","is-referenced-by-count":6,"title":["Value of short exercise and short exercise with cooling tests in the diagnosis of myotonic dystrophies (DM1 AND DM2)"],"prefix":"10.1002","volume":"49","author":[{"given":"Malgorzata","family":"Gawel","sequence":"first","affiliation":[{"name":"Department of Neurology Medical University of Warsaw Banacha 1A Warsaw 02\u2013097 Poland"}],"role":[{"role":"author","vocabulary":"crossref"}]},{"given":"Elzbieta","family":"Szmidt\u2010Salkowska","sequence":"additional","affiliation":[{"name":"Department of Neurology Medical University of Warsaw Banacha 1A Warsaw 02\u2013097 Poland"}],"role":[{"role":"author","vocabulary":"crossref"}]},{"given":"Anna","family":"Lusakowska","sequence":"additional","affiliation":[{"name":"Department of Neurology Medical University of Warsaw Banacha 1A Warsaw 02\u2013097 Poland"}],"role":[{"role":"author","vocabulary":"crossref"}]},{"given":"Monika","family":"Nojszewska","sequence":"additional","affiliation":[{"name":"Department of Neurology Medical University of Warsaw Banacha 1A Warsaw 02\u2013097 Poland"}],"role":[{"role":"author","vocabulary":"crossref"}]},{"given":"Anna","family":"Sulek","sequence":"additional","affiliation":[{"name":"Department of Genetics Institute of Psychiatry and Neurology Warsaw Poland"}],"role":[{"role":"author","vocabulary":"crossref"}]},{"given":"Wioletta","family":"Krysa","sequence":"additional","affiliation":[{"name":"Department of Genetics Institute of Psychiatry and Neurology Warsaw Poland"}],"role":[{"role":"author","vocabulary":"crossref"}]},{"given":"Marta","family":"Rajkiewicz","sequence":"additional","affiliation":[{"name":"Department of Genetics Institute of Psychiatry and Neurology Warsaw Poland"}],"role":[{"role":"author","vocabulary":"crossref"}]},{"given":"Andrzej","family":"Seroka","sequence":"additional","affiliation":[{"name":"Department of Neurology Medical University of Warsaw Banacha 1A Warsaw 02\u2013097 Poland"}],"role":[{"role":"author","vocabulary":"crossref"}]},{"given":"Anna M.","family":"Kaminska","sequence":"additional","affiliation":[{"name":"Department of Genetics Institute of Psychiatry and Neurology Warsaw 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conductance in normal and myotonic muscle fibres and the action of monocarboxylic aromatic acids","volume":"219","author":"Bryant SH","year":"1971","journal-title":"J Phys"},{"key":"e_1_2_7_25_1","doi-asserted-by":"publisher","DOI":"10.1002\/mus.20295"},{"key":"e_1_2_7_26_1","doi-asserted-by":"publisher","DOI":"10.1002\/mus.880110609"},{"key":"e_1_2_7_27_1","unstructured":"TangZZ YarotskyyV WeiL SobczakK NakamoriM EichingerK et al.Muscle weakness in myotonic dystrophy associated with misregulated splicing and altered gating of Ca(V)1.1 calcium channel."}],"container-title":["Muscle &amp; 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