{"status":"ok","message-type":"work","message-version":"1.0.0","message":{"indexed":{"date-parts":[[2023,10,24]],"date-time":"2023-10-24T10:14:11Z","timestamp":1698142451936},"reference-count":20,"publisher":"Wiley","issue":"7","license":[{"start":{"date-parts":[[2004,10,13]],"date-time":"2004-10-13T00:00:00Z","timestamp":1097625600000},"content-version":"vor","delay-in-days":5218,"URL":"http:\/\/onlinelibrary.wiley.com\/termsAndConditions#vor"}],"content-domain":{"domain":[],"crossmark-restriction":false},"short-container-title":["Muscle and Nerve"],"published-print":{"date-parts":[[1990,7]]},"abstract":"<jats:title>Abstract<\/jats:title><jats:p>Local cooling of the muscle resulted in significant increases in M wave surface areas in patients with ALS, myasthenia gravis, the Lambert\u2010Eaton myasthenic syndrome, and also in controls. The most striking increases were seen in patients with early ALS who had minimal lower motor neuron involvement and\/or little defect on neuromuscular transmission and patients with the myasthenic syndrome. Patients with myasthenia gravis had intermediate increases between these groups and the controls; there was a large increases in M wave surface area in myasthenia gravis compared with controls but this could be accounted for by improvement in neuromuscular transmission. The large increase in M wave surface area in early ALS could be the result of temporal summation of individual muscle fibers in motor units with increased fiber densities. The increase in both ALS and the myasthenic syncrome could result also from the loss of a trophic factor causing changes at the muscle membrane.<\/jats:p>","DOI":"10.1002\/mus.880130709","type":"journal-article","created":{"date-parts":[[2005,1,1]],"date-time":"2005-01-01T09:40:50Z","timestamp":1104572450000},"page":"613-617","source":"Crossref","is-referenced-by-count":10,"title":["M wave changes with temperature in amyotrophic lateral sclerosis and disorders of neuromuscular transmission"],"prefix":"10.1002","volume":"13","author":[{"given":"Eric H.","family":"Denys","sequence":"first","affiliation":[],"role":[{"role":"author","vocabulary":"crossref"}]}],"member":"311","published-online":{"date-parts":[[2004,10,13]]},"reference":[{"key":"e_1_2_1_2_2","doi-asserted-by":"publisher","DOI":"10.1016\/0014-4886(72)90103-3"},{"key":"e_1_2_1_3_2","doi-asserted-by":"publisher","DOI":"10.1212\/WNL.31.2.202"},{"key":"e_1_2_1_4_2","doi-asserted-by":"publisher","DOI":"10.1136\/jnnp.44.5.407"},{"key":"e_1_2_1_5_2","doi-asserted-by":"publisher","DOI":"10.1016\/S0140-6736(74)91632-8"},{"key":"e_1_2_1_6_2","doi-asserted-by":"publisher","DOI":"10.1136\/jnnp.34.5.551"},{"key":"e_1_2_1_7_2","first-page":"598","article-title":"The effect of temperature on the compound action potential in neuromuscular disease and normal controls","volume":"4","author":"Denys EH","year":"1977","journal-title":"Electroenceph Clin Neurophys"},{"key":"e_1_2_1_8_2","first-page":"248","volume-title":"Plasmapheresis and the Immunobiology of Myasthenia Gravis","author":"Denys EH","year":"1979"},{"key":"e_1_2_1_9_2","doi-asserted-by":"publisher","DOI":"10.1001\/archneur.1979.00500400056008"},{"key":"e_1_2_1_10_2","first-page":"104","volume-title":"Handbook of Clinical Neurology","author":"Desmedt JE","year":"1970"},{"key":"e_1_2_1_11_2","doi-asserted-by":"crossref","first-page":"109","DOI":"10.1113\/jphysiol.1952.sp004735","article-title":"Spontaneous subthreshold activity at motor nerve endings","volume":"117","author":"Fatt P","year":"1952","journal-title":"J Physiol (Lond.)"},{"key":"e_1_2_1_12_2","doi-asserted-by":"publisher","DOI":"10.1016\/0006-8993(74)90436-3"},{"key":"e_1_2_1_13_2","doi-asserted-by":"publisher","DOI":"10.1001\/archneur.1976.00500030031006"},{"key":"e_1_2_1_14_2","doi-asserted-by":"publisher","DOI":"10.1113\/jphysiol.1949.sp004388"},{"key":"e_1_2_1_15_2","first-page":"24","article-title":"Sensory nerve action potential amplitudes: alterations with temperature","volume":"45","author":"Hulley WL","year":"1978","journal-title":"Electroenceph Clin Neurophysiol"},{"key":"e_1_2_1_16_2","first-page":"135","volume-title":"Motor Neuron Disease","author":"Lambert EH","year":"1969"},{"key":"e_1_2_1_17_2","doi-asserted-by":"crossref","unstructured":"OhSJ DwyerDS BradleyRJ: Overlap myasthenic syndrome: combined myasthenia gravis and Eaton\u2010Lambert syndrome.Neurology(NY)1987;1411\u20131414.","DOI":"10.1212\/WNL.37.8.1411"},{"key":"e_1_2_1_18_2","doi-asserted-by":"publisher","DOI":"10.1007\/BF00312813"},{"key":"e_1_2_1_19_2","first-page":"105","volume-title":"Research Progress in Motor Neurone Disease","author":"Stalberg E","year":"1984"},{"key":"e_1_2_1_20_2","doi-asserted-by":"publisher","DOI":"10.1113\/jphysiol.1960.sp006463"},{"key":"e_1_2_1_21_2","doi-asserted-by":"publisher","DOI":"10.1002\/mus.880090811"}],"container-title":["Muscle &amp; Nerve"],"original-title":[],"language":"en","link":[{"URL":"https:\/\/api.wiley.com\/onlinelibrary\/tdm\/v1\/articles\/10.1002%2Fmus.880130709","content-type":"unspecified","content-version":"vor","intended-application":"text-mining"},{"URL":"https:\/\/onlinelibrary.wiley.com\/doi\/pdf\/10.1002\/mus.880130709","content-type":"unspecified","content-version":"vor","intended-application":"similarity-checking"}],"deposited":{"date-parts":[[2023,10,23]],"date-time":"2023-10-23T02:07:37Z","timestamp":1698026857000},"score":1,"resource":{"primary":{"URL":"https:\/\/onlinelibrary.wiley.com\/doi\/10.1002\/mus.880130709"}},"subtitle":[],"short-title":[],"issued":{"date-parts":[[1990,7]]},"references-count":20,"journal-issue":{"issue":"7","published-print":{"date-parts":[[1990,7]]}},"alternative-id":["10.1002\/mus.880130709"],"URL":"https:\/\/doi.org\/10.1002\/mus.880130709","archive":["Portico"],"relation":{},"ISSN":["0148-639X","1097-4598"],"issn-type":[{"value":"0148-639X","type":"print"},{"value":"1097-4598","type":"electronic"}],"subject":[],"published":{"date-parts":[[1990,7]]}}}