{"status":"ok","message-type":"work","message-version":"1.0.0","message":{"indexed":{"date-parts":[[2026,6,26]],"date-time":"2026-06-26T23:46:22Z","timestamp":1782517582023,"version":"3.54.5"},"reference-count":45,"publisher":"Wiley","issue":"7","license":[{"start":{"date-parts":[[2026,5,11]],"date-time":"2026-05-11T00:00:00Z","timestamp":1778457600000},"content-version":"vor","delay-in-days":0,"URL":"http:\/\/creativecommons.org\/licenses\/by\/4.0\/"},{"start":{"date-parts":[[2026,5,11]],"date-time":"2026-05-11T00:00:00Z","timestamp":1778457600000},"content-version":"tdm","delay-in-days":0,"URL":"http:\/\/doi.wiley.com\/10.1002\/tdm_license_1.1"}],"funder":[{"DOI":"10.13039\/501100000781","name":"European Research Council","doi-asserted-by":"publisher","award":["101087395"],"award-info":[{"award-number":["101087395"]}],"id":[{"id":"10.13039\/501100000781","id-type":"DOI","asserted-by":"publisher"}]},{"DOI":"10.13039\/100010661","name":"Horizon 2020 Framework Programme","doi-asserted-by":"publisher","award":["965381"],"award-info":[{"award-number":["965381"]}],"id":[{"id":"10.13039\/100010661","id-type":"DOI","asserted-by":"publisher"}]},{"DOI":"10.13039\/501100000265","name":"Medical Research Council","doi-asserted-by":"publisher","award":["MR\/V012878\/1"],"award-info":[{"award-number":["MR\/V012878\/1"]}],"id":[{"id":"10.13039\/501100000265","id-type":"DOI","asserted-by":"publisher"}]},{"DOI":"10.13039\/100000002","name":"National Institutes of Health","doi-asserted-by":"publisher","award":["AG046938"],"award-info":[{"award-number":["AG046938"]}],"id":[{"id":"10.13039\/100000002","id-type":"DOI","asserted-by":"publisher"}]}],"content-domain":{"domain":["onlinelibrary.wiley.com"],"crossmark-restriction":true},"short-container-title":["Obesity"],"published-print":{"date-parts":[[2026,7]]},"abstract":"<jats:title>ABSTRACT<\/jats:title>\n                  <jats:sec>\n                    <jats:title>Objective<\/jats:title>\n                    <jats:p>We examined whether genetic risk for higher BMI, parental feeding practices (restriction, pressure to eat, and monitoring), and their interactions were associated with overweight trajectories during adolescence.<\/jats:p>\n                  <\/jats:sec>\n                  <jats:sec>\n                    <jats:title>Methods<\/jats:title>\n                    <jats:p>Data came from 5568 participants aged 10\u201318 in the Twins' Early Development Study who had at least one measurement of weight. Parental feeding practices were reported at age 10 using the Child Feeding Questionnaire. A polygenic score for BMI was used as a proxy for genetic risk. We identified overweight trajectories using latent class growth analysis, and associations with genetic risk, feeding practices, and their interactions were tested using multinomial logistic regression with trajectory as the outcome.<\/jats:p>\n                  <\/jats:sec>\n                  <jats:sec>\n                    <jats:title>Results<\/jats:title>\n                    <jats:p>Three trajectories were identified: non\u2010overweight, persistent overweight, and adolescent\u2010onset overweight. Higher genetic risk and greater parental restriction and monitoring were associated with persistent overweight, whereas pressure to eat was associated with non\u2010overweight. Associations remained after adjustment for general parenting and additional risk factors. We found no evidence for gene\u2013environment interaction.<\/jats:p>\n                  <\/jats:sec>\n                  <jats:sec>\n                    <jats:title>Conclusions<\/jats:title>\n                    <jats:p>Genetic risk and parental feeding practices were independently associated with adolescent weight development. While this suggests feeding practices may be relevant targets for prevention, the findings likely represent complex bidirectional parent\u2013child dynamics.<\/jats:p>\n                  <\/jats:sec>","DOI":"10.1002\/oby.70217","type":"journal-article","created":{"date-parts":[[2026,5,11]],"date-time":"2026-05-11T12:41:34Z","timestamp":1778503294000},"page":"1468-1476","update-policy":"https:\/\/doi.org\/10.1002\/crossmark_policy","source":"Crossref","is-referenced-by-count":0,"title":["Interaction Between Genetic Risk and Parental Feeding Practices in the Prediction of Overweight Across Adolescence"],"prefix":"10.1002","volume":"34","author":[{"given":"Marthe","family":"de Roo","sequence":"first","affiliation":[{"name":"Faculty of Behavioral and Social Sciences, Department of Pedagogy and Educational Sciences University of Groningen  Groningen the Netherlands"}],"role":[{"vocabulary":"crossref","role":"author"}]},{"ORCID":"https:\/\/orcid.org\/0000-0001-6936-9285","authenticated-orcid":false,"given":"Tina","family":"Kretschmer","sequence":"additional","affiliation":[{"name":"Department of Psychology Friedrich\u2010Alexander University Erlangen\u2010Nuremberg  Erlangen Germany"}],"role":[{"vocabulary":"crossref","role":"author"}]},{"ORCID":"https:\/\/orcid.org\/0000-0002-0066-2827","authenticated-orcid":false,"given":"Clare","family":"Llewellyn","sequence":"additional","affiliation":[{"name":"Department of Behavioural Science and Health University College London  London UK"}],"role":[{"vocabulary":"crossref","role":"author"}]},{"ORCID":"https:\/\/orcid.org\/0000-0002-8145-5482","authenticated-orcid":false,"given":"Bonamy R.","family":"Oliver","sequence":"additional","affiliation":[{"name":"Department of Psychology and Human Development UCL Institute of Education, University College London  London UK"}],"role":[{"vocabulary":"crossref","role":"author"}]},{"given":"Catharina A.","family":"Hartman","sequence":"additional","affiliation":[{"name":"Interdisciplinary Center Psychopathology and Emotion Regulation (ICPE) University of Groningen, University Medical Center Groningen  Groningen the 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