{"status":"ok","message-type":"work","message-version":"1.0.0","message":{"indexed":{"date-parts":[[2025,10,21]],"date-time":"2025-10-21T00:44:48Z","timestamp":1761007488699,"version":"build-2065373602"},"reference-count":24,"publisher":"Wiley","issue":"3","license":[{"start":{"date-parts":[[2005,6,16]],"date-time":"2005-06-16T00:00:00Z","timestamp":1118880000000},"content-version":"vor","delay-in-days":8628,"URL":"http:\/\/onlinelibrary.wiley.com\/termsAndConditions#vor"}],"content-domain":{"domain":["pathsocjournals.onlinelibrary.wiley.com"],"crossmark-restriction":true},"short-container-title":["The Journal of Pathology"],"published-print":{"date-parts":[[1981,11]]},"abstract":"<jats:title>Abstract<\/jats:title><jats:p>Characteristic ultrastructural alterations of the glomerular basal lamina have been reported in hereditary nephritis. The basal lamina is irregularly thickened and the lamina densa shows replication with a \u201cbasket weave\u201d pattern, enclosing electron\u2010lucent lacunae which frequently contain small dense particles. However there is controversy regarding the specificity of this lesion in hereditary nephritis. To determine the specificity, 366 renal biopsies from 310 children were studied retrospectively. Twenty\u2010four out of 27 patients with hereditary nephritis showed the characteristic changes of the basal lamina and they were widespread in 17.<\/jats:p><jats:p>Two patients with recurrent haematuria but no family history of deafness or haematuria showed similar extensive changes and are regarded as new mutant cases of hereditary nephritis.<\/jats:p><jats:p>Similar changes were seen in 17 of the 281 patients with other conditions but were always localized to a few capillary loops. We conclude that a widespread \u201cbasket weave\u201d pattern appears to be confined to hereditary nephritis and is seen in the great majority of such cases.<\/jats:p>","DOI":"10.1002\/path.1711350305","type":"journal-article","created":{"date-parts":[[2005,6,21]],"date-time":"2005-06-21T14:39:07Z","timestamp":1119364747000},"page":"199-209","update-policy":"https:\/\/doi.org\/10.1002\/crossmark_policy","source":"Crossref","is-referenced-by-count":50,"title":["The glomerular basal lamina in hereditary nephritis"],"prefix":"10.1002","volume":"135","author":[{"given":"N.","family":"Yoshikawa","sequence":"first","affiliation":[],"role":[{"role":"author","vocabulary":"crossref"}]},{"given":"A. H.","family":"Cameron","sequence":"additional","affiliation":[],"role":[{"role":"author","vocabulary":"crossref"}]},{"given":"R. H. R.","family":"White","sequence":"additional","affiliation":[],"role":[{"role":"author","vocabulary":"crossref"}]}],"member":"311","published-online":{"date-parts":[[2005,6,16]]},"reference":[{"key":"e_1_2_1_2_1","doi-asserted-by":"publisher","DOI":"10.1136\/bmj.1.3454.504"},{"key":"e_1_2_1_3_1","first-page":"374","article-title":"Pathologic characteristics of hereditary nephritis","volume":"95","author":"Churg J.","year":"1973","journal-title":"Arch. Pathol."},{"key":"e_1_2_1_4_1","doi-asserted-by":"publisher","DOI":"10.1136\/jcp.26.3.198"},{"key":"e_1_2_1_5_1","doi-asserted-by":"publisher","DOI":"10.1016\/S0046-8177(79)80110-0"},{"key":"e_1_2_1_6_1","first-page":"143","article-title":"Alport's syndrome (progressive hereditary nephritis)","volume":"2","author":"Gaboardi F.","year":"1974","journal-title":"Clin. 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Invest."},{"key":"e_1_2_1_10_1","first-page":"133","article-title":"Ultrastructural lesions of the glomerular basement membrane in hereditary chronic nephritis","volume":"3","author":"Hinglais N.","year":"1974","journal-title":"Advances in nephrology"},{"key":"e_1_2_1_11_1","doi-asserted-by":"publisher","DOI":"10.1016\/S0022-3476(70)80042-7"},{"key":"e_1_2_1_12_1","first-page":"475","article-title":"The specificity of split renal membranes in hereditary nephritis","volume":"100","author":"Kohaut E. C.","year":"1976","journal-title":"Arch. Pathol. Lab. Med."},{"key":"e_1_2_1_13_1","doi-asserted-by":"publisher","DOI":"10.1007\/BF00433075"},{"key":"e_1_2_1_14_1","doi-asserted-by":"publisher","DOI":"10.1159\/000180417"},{"key":"e_1_2_1_15_1","doi-asserted-by":"publisher","DOI":"10.1159\/000180666"},{"key":"e_1_2_1_16_1","doi-asserted-by":"publisher","DOI":"10.1016\/0002-9343(74)90749-9"},{"key":"e_1_2_1_17_1","unstructured":"Spear G. S.1974. Pathology of the kidney in Alport's syndrome. InPathology Annual edited by S. C. Sommers New York p.93."},{"key":"e_1_2_1_18_1","first-page":"213","article-title":"Alport's syndrome: Emphasizing electron microscopic studies of the glomerulus","volume":"69","author":"Spear G. S.","year":"1972","journal-title":"Am. J. Pathol."},{"key":"e_1_2_1_19_1","doi-asserted-by":"publisher","DOI":"10.1016\/S0002-9343(70)80113-9"},{"key":"e_1_2_1_20_1","unstructured":"Vernier R. L. Mauer S. M. Fish A. J. andMichael A. F.1971. The mesangial cell in glomerulonephritis. InRecent Advances in Nephrology edited by J. Hamburger J. Crosnier and M. H. Maxwell Chicago p.31."},{"key":"e_1_2_1_21_1","first-page":"120","article-title":"Evaluation of a disposable needle for renal biopsy in children","volume":"2","author":"White R. H. R.","year":"1974","journal-title":"Clin. Nephrol."},{"key":"e_1_2_1_22_1","unstructured":"Yoshikawa N. White R. H. R. andCameron A. H.1980a.Familial haematuria: Clinico\u2010pathological correlations(to be published)."},{"key":"e_1_2_1_23_1","unstructured":"Yoshikawa N. White R. H. R. andCameron A. H.1980b.Non\u2010familial haematura: Clinico\u2010pathological correlations(to be published)."},{"key":"e_1_2_1_24_1","unstructured":"Yoshikawa N. White R. H. R. andCameron A. 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