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Oral administration of the ethyl acetate extract (extract A<jats:sub>3<\/jats:sub>) inhibited the formation of gastric lesions induced by ethanol in a dose dependent manner. The protective effect of extract A<jats:sub>3<\/jats:sub> against ethanol induced gastric lesions was not abolished by pretreatment with indomethacin (10 mg kg<jats:sup>\u22121<\/jats:sup>). Further, extract A<jats:sub>3<\/jats:sub> inhibited increase in vascular permeability due to ethanol administration. Extent of lipid peroxidation was signi\ufb01cantly reduced in animals treated with extract. Extract A<jats:sub>3<\/jats:sub> also inhibited the formation of gastric ulcers induced by pylorus ligation, when administered both orally and intraperitoneally. Moreover, pretreatment with extract A<jats:sub>3<\/jats:sub> increased mucus production and glycoprotein content, which was evident from the rise in mucin activity and TC: PR ratio. 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