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Kre6p showed extensive co\u2010localization with Och1p\u2010containing\n                    <jats:italic>cis<\/jats:italic>\n                    \u2010Golgi vesicles. The correct localization of Kre6p requires its cytoplasmic tail, Las17p, Sla1p and Vrp1p, suggesting that the cytoplasmic tail of Kre6p acts as a receptor, linking this\n                    <jats:italic>cis<\/jats:italic>\n                    \u2010Golgi protein to Las17p and Sla1p. The actin patch assembly mutants\n                    <jats:italic>las17\u0394<\/jats:italic>\n                    ,\n                    <jats:italic>sla1\u0394<\/jats:italic>\n                    and\n                    <jats:italic>vrp1\u0394<\/jats:italic>\n                    showed elevated levels of cell wall \u03b2\u20101,6\u2010glucan, and mutant cells were capable of only a limited number of cell divisions compared to wild\u2010type. EM image analysis and \u03b2\u20101,6\u2010glucan localization indicated abnormal wall proliferation in the mother cells of these mutants. The pattern of cell wall hypertrophy indicates a failure to restrict cell wall growth to the bud. Copyright \u00a9 2002 John Wiley &amp; Sons, Ltd.\n                  <\/jats:p>","DOI":"10.1002\/yea.904","type":"journal-article","created":{"date-parts":[[2002,10,8]],"date-time":"2002-10-08T22:28:17Z","timestamp":1034116097000},"page":"1097-1112","source":"Crossref","is-referenced-by-count":24,"title":["Actin patch assembly proteins Las17p and Sla1p restrict cell wall growth to daughter cells and interact with\n                    <i>cis<\/i>\n                    \u2010Golgi protein Kre6p"],"prefix":"10.1002","volume":"19","author":[{"given":"Huijuan","family":"Li","sequence":"first","affiliation":[],"role":[{"vocabulary":"crossref","role":"author"}]},{"given":"Nicolas","family":"Pag\u00e9","sequence":"additional","affiliation":[],"role":[{"vocabulary":"crossref","role":"author"}]},{"given":"Howard","family":"Bussey","sequence":"additional","affiliation":[],"role":[{"vocabulary":"crossref","role":"author"}]}],"member":"311","published-online":{"date-parts":[[2002,8,29]]},"reference":[{"key":"e_1_2_6_2_1","doi-asserted-by":"publisher","DOI":"10.1083\/jcb.141.6.1357"},{"key":"e_1_2_6_3_1","doi-asserted-by":"publisher","DOI":"10.1091\/mbc.10.4.1061"},{"key":"e_1_2_6_4_1","doi-asserted-by":"publisher","DOI":"10.1016\/S1097-2765(00)80324-X"},{"key":"e_1_2_6_5_1","doi-asserted-by":"publisher","DOI":"10.1016\/S0092-8674(00)00162-8"},{"key":"e_1_2_6_6_1","doi-asserted-by":"publisher","DOI":"10.1016\/0003-2697(76)90527-3"},{"key":"e_1_2_6_7_1","doi-asserted-by":"publisher","DOI":"10.1002\/yea.755"},{"key":"e_1_2_6_8_1","doi-asserted-by":"crossref","first-page":"837","DOI":"10.1093\/genetics\/133.4.837","article-title":"A 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