{"status":"ok","message-type":"work","message-version":"1.0.0","message":{"indexed":{"date-parts":[[2023,9,15]],"date-time":"2023-09-15T13:07:18Z","timestamp":1694783238096},"reference-count":12,"publisher":"Wiley","issue":"1","license":[{"start":{"date-parts":[[1987,10,1]],"date-time":"1987-10-01T00:00:00Z","timestamp":560044800000},"content-version":"vor","delay-in-days":0,"URL":"http:\/\/onlinelibrary.wiley.com\/termsAndConditions#vor"}],"content-domain":{"domain":[],"crossmark-restriction":false},"short-container-title":["J of Inher Metab Disea"],"published-print":{"date-parts":[[1989,3]]},"abstract":"<jats:title>Summary<\/jats:title><jats:p>Persistent hyperkalaemia was found in a patient with vitamin B<jats:sub>12<\/jats:sub> unresponsive methylmalonic acidaemia associated with hyperuricaemia. At 3 years and 8 months of age, a serum potassium level of 6.8 mmol L<jats:sup>\u22121<\/jats:sup> was found when blood gas measurement was normal. One year later azotaemia was noted. At the age of 5 years, renal function studies disclosed hyperaldosteronism, decreased creatinine clearance, reduction of the reabsorption of sodium at distal diluting segments and inadequate concentration of urine at the collecting ducts. The reduction of the reabsorption of sodium, which may have resulted in decreased potassium excretion, and the decrease in glomerular filtration rate, together with the superimposed excess intake of potassium, appeared to be responsible for the hyperkalaemia. Dietary potassium restriction was effective in suppressing the hyperkalaemia.<\/jats:p>","DOI":"10.1007\/bf01805536","type":"journal-article","created":{"date-parts":[[2005,6,27]],"date-time":"2005-06-27T23:56:35Z","timestamp":1119916595000},"page":"89-93","source":"Crossref","is-referenced-by-count":8,"title":["Persistent hyperkalaemia in vitamin B<sub>12<\/sub> unresponsive methylmalonic acidaemia"],"prefix":"10.1002","volume":"12","author":[{"given":"J.","family":"Morita","sequence":"first","affiliation":[{"name":"Department of Pediatrics and Child Health Kurume University, School of Medicine  67 Asahi\u2010machi Kurume 830 Japan"}],"role":[{"role":"author","vocabulary":"crossref"}]},{"given":"Y.","family":"Ito","sequence":"additional","affiliation":[{"name":"Department of Pediatrics and Child Health Kurume University, School of Medicine  67 Asahi\u2010machi Kurume 830 Japan"}],"role":[{"role":"author","vocabulary":"crossref"}]},{"given":"M.","family":"Yoshino","sequence":"additional","affiliation":[{"name":"Department of Pediatrics and Child Health Kurume University, School of Medicine  67 Asahi\u2010machi Kurume 830 Japan"}],"role":[{"role":"author","vocabulary":"crossref"}]},{"given":"Y.","family":"Koga","sequence":"additional","affiliation":[{"name":"Department of Pediatrics and Child Health Kurume University, School of Medicine  67 Asahi\u2010machi Kurume 830 Japan"}],"role":[{"role":"author","vocabulary":"crossref"}]},{"given":"S.","family":"Yano","sequence":"additional","affiliation":[{"name":"Department of Pediatrics and Child Health Kurume University, School of Medicine  67 Asahi\u2010machi Kurume 830 Japan"}],"role":[{"role":"author","vocabulary":"crossref"}]},{"given":"I.","family":"Yoshida","sequence":"additional","affiliation":[{"name":"Department of Pediatrics and Child Health Kurume University, School of Medicine  67 Asahi\u2010machi Kurume 830 Japan"}],"role":[{"role":"author","vocabulary":"crossref"}]},{"given":"F.","family":"Yamashita","sequence":"additional","affiliation":[{"name":"Department of Pediatrics and Child Health Kurume University, School of Medicine  67 Asahi\u2010machi Kurume 830 Japan"}],"role":[{"role":"author","vocabulary":"crossref"}]}],"member":"311","published-online":{"date-parts":[[1987,10]]},"reference":[{"key":"e_1_2_1_2_1","doi-asserted-by":"publisher","DOI":"10.1016\/S0025-7125(16)31183-X"},{"key":"e_1_2_1_3_1","first-page":"543","article-title":"Acidemie methyl malonique avec nephropathie hyperuricemique","volume":"31","author":"Broyer M.","year":"1974","journal-title":"Arch. 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