{"status":"ok","message-type":"work","message-version":"1.0.0","message":{"indexed":{"date-parts":[[2026,9,7]],"date-time":"2026-09-07T23:55:01Z","timestamp":1788825301772,"version":"build-2803163510"},"reference-count":29,"publisher":"Springer Science and Business Media LLC","issue":"12","license":[{"start":{"date-parts":[[2022,6,27]],"date-time":"2022-06-27T00:00:00Z","timestamp":1656288000000},"content-version":"tdm","delay-in-days":0,"URL":"https:\/\/creativecommons.org\/licenses\/by\/4.0"},{"start":{"date-parts":[[2022,6,27]],"date-time":"2022-06-27T00:00:00Z","timestamp":1656288000000},"content-version":"vor","delay-in-days":0,"URL":"https:\/\/creativecommons.org\/licenses\/by\/4.0"}],"funder":[{"name":"NTNU Norwegian University of Science and Technology"}],"content-domain":{"domain":["link.springer.com"],"crossmark-restriction":false},"short-container-title":["Neuroradiology"],"published-print":{"date-parts":[[2022,12]]},"abstract":"<jats:title>Abstract\n<\/jats:title><jats:sec>\n                <jats:title>Purpose<\/jats:title>\n                <jats:p>To assess the ability of 7\u00a0T MRI to detect hippocampal DWI lesions in the acute phase of TGA compared to 1.5\u00a0T\/3\u00a0T MRI.<\/jats:p>\n              <\/jats:sec><jats:sec>\n                <jats:title>Methods<\/jats:title>\n                <jats:p>Patients with a clinical diagnosis consistent with TGA and a 1.5\/3\u00a0T MRI underwent an additional 7\u00a0T MRI when the 7\u00a0T system was available for clinical use, thus serving as their own controls.<\/jats:p>\n              <\/jats:sec><jats:sec>\n                <jats:title>Results<\/jats:title>\n                <jats:p>Thirteen TGA patients with a median age of 68.5\u00a0years (range 46\u201377\u00a0years) were included and imaged at 1.5\/3\u00a0T (median 17\u00a0h after onset of symptoms, range 3\u201323\u00a0h) and 7\u00a0T (median 23\u00a0h after onset, range 15\u201346\u00a0h). The 7\u00a0T MRIs were performed a median of 15\u00a0h after the 1.5\/3 T MRIs (range 1\u201328\u00a0h). At 1.5\/3\u00a0T, six patients (46%) were found to have at least one hippocampal DWI-lesions supporting the TGA diagnosis, which increased to 11 patients (85%) when examined at 7\u00a0T (<jats:italic>p<\/jats:italic>\u2009=\u20090.03). At 1.5\/3\u00a0T, nine hippocampal DWI lesions were detected, which increased to 19 at 7\u00a0T, giving an increased detection rate of 111% (<jats:italic>p<\/jats:italic>\u2009=\u20090.002). Both neuroradiologists found the hippocampal DWI lesions at 7\u00a0T to have higher conspicuity and be easier to categorize as true findings compared to 1.5\/3\u00a0T.<\/jats:p>\n              <\/jats:sec><jats:sec>\n                <jats:title>Conclusion<\/jats:title>\n                <jats:p>Seven-Tesla MRI showed both a statistically significant increase in the total number of detected hippocampal DWI lesions and the proportion of patients with at least one hippocampal DWI lesion supporting the TGA diagnosis compared to 1.5\/3\u00a0T. Clinical use of 7\u00a0T will increase the number of patients having their TGA diagnosis supported by MRI, which can be especially useful in patients with negative 1.5\/3 T MRI and low clinical certainty.<\/jats:p>\n              <\/jats:sec>","DOI":"10.1007\/s00234-022-02998-7","type":"journal-article","created":{"date-parts":[[2022,6,27]],"date-time":"2022-06-27T03:14:18Z","timestamp":1656299658000},"page":"2217-2226","update-policy":"https:\/\/doi.org\/10.1007\/springer_crossmark_policy","source":"Crossref","is-referenced-by-count":14,"title":["Transient global amnesia: 7 Tesla MRI reveals more hippocampal lesions with diffusion restriction compared to 1.5 and 3 Tesla MRI"],"prefix":"10.1007","volume":"64","author":[{"ORCID":"https:\/\/orcid.org\/0000-0003-2020-8717","authenticated-orcid":false,"given":"Runa Geirmundsdatter","family":"Unsg\u00e5rd","sequence":"first","affiliation":[],"role":[{"vocabulary":"crossref","role":"author"}]},{"ORCID":"https:\/\/orcid.org\/0000-0001-5700-0018","authenticated-orcid":false,"given":"Thanh P.","family":"Doan","sequence":"additional","affiliation":[],"role":[{"vocabulary":"crossref","role":"author"}]},{"given":"Knut Kristian","family":"Nordlid","sequence":"additional","affiliation":[],"role":[{"vocabulary":"crossref","role":"author"}]},{"given":"Kjell Arne","family":"Kvistad","sequence":"additional","affiliation":[],"role":[{"vocabulary":"crossref","role":"author"}]},{"given":"P\u00e5l Erik","family":"Goa","sequence":"additional","affiliation":[],"role":[{"vocabulary":"crossref","role":"author"}]},{"ORCID":"https:\/\/orcid.org\/0000-0001-7929-3739","authenticated-orcid":false,"given":"Erik Magnus","family":"Berntsen","sequence":"additional","affiliation":[],"role":[{"vocabulary":"crossref","role":"author"}]}],"member":"297","published-online":{"date-parts":[[2022,6,27]]},"reference":[{"issue":"2","key":"2998_CR1","doi-asserted-by":"publisher","first-page":"205","DOI":"10.1016\/S1474-4422(09)70344-8","volume":"9","author":"T Bartsch","year":"2010","unstructured":"Bartsch T, Deuschl G (2010) Transient global amnesia: functional anatomy and clinical implications. 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