{"status":"ok","message-type":"work","message-version":"1.0.0","message":{"indexed":{"date-parts":[[2023,12,25]],"date-time":"2023-12-25T00:02:28Z","timestamp":1703462548613},"reference-count":57,"publisher":"Wiley","issue":"9","license":[{"start":{"date-parts":[[2005,8,11]],"date-time":"2005-08-11T00:00:00Z","timestamp":1123718400000},"content-version":"vor","delay-in-days":0,"URL":"http:\/\/onlinelibrary.wiley.com\/termsAndConditions#vor"}],"content-domain":{"domain":[],"crossmark-restriction":false},"short-container-title":["World j. surg."],"published-print":{"date-parts":[[2005,9]]},"abstract":"<jats:title>Abstract<\/jats:title><jats:p>Streptozotocin administration in newborn rats (nSTZ\u2010rats) leads to adults with mild insulin deficiency and normoglycemia, and is accepted as a model of type 2 diabetes. We examined possible differences in the production of inflammatory mediators between healthy and nSTZ\u2010rats after ischemia\u2010reperfusion (I\u2010R). Two\u2010month\u2010old control and nSTZ\u2010rats were randomly separated into control and intestinal I\u2010R groups. After reperfusion, samples were obtained from the portal vein (PV) infrahepatic cava vein (ICV), suprahepatic cava vein (SCV), jejunal wall, and pancreas. Nitric oxide (NO), lipid hydroperoxides (LPO), tumor necrosis factor alpha (TNF\u2010\u03b1), 60 kDa receptor (sTNF\u2010R1), 80 kDa (sTNF\u2010R2), and intercellular adhesion molecule\u20101 (ICAM\u20101), were determined. After I\u2010R, nSTZ\u2010rats showed increased plasma concentrations of LPO, NO, ICAM\u20101 (0.5141 \u00b1 0.083 vs 0.024 \u00b1 0.003, ICV; 0.574 \u00b1 0.075 vs 0.023 \u00b1 0.003, SCV; 0.528 \u00b1 0.067 vs 0.027 \u00b1 0.003\u00a0PV; ng\/ml), TNF\u2010<jats:italic>\u03b1<\/jats:italic> (42.4 \u00b1 5.7 ICV, 248.4 \u00b1 28.2 SCV, and 33.6 \u00b1 4.0\u00a0PV. In n STZ\u2010rats, vs 4.36 \u00b1 0.57, 4.74 \u00b1 0.77, and 3.16 \u00b1 0.32, respectively, in control rats; pg\/ml), and sTNF\u2010R1. Both TNF\u2010\u03b1 and NO plasma levels were higher in SCV than in ICV and PV after I\u2010R. In addition, after I\u2010R, jejunal wall of nSTZ\u2010rats showed an increase of TNF\u2010\u03b1 IL\u20101, and IL\u201010 levels. A pre\u2010existing state of glucose intolerance intensifies the inflammatory response after intestinal I\u2010R.<\/jats:p>","DOI":"10.1007\/s00268-005-7700-9","type":"journal-article","created":{"date-parts":[[2005,8,8]],"date-time":"2005-08-08T13:19:23Z","timestamp":1123507163000},"page":"1143-1150","source":"Crossref","is-referenced-by-count":0,"title":["Glucose Intolerance Modifies the Inflammatory Response After Intestinal Ischemia\u2010Reperfusion"],"prefix":"10.1002","volume":"29","author":[{"given":"Juan C.","family":"Garcia\u2010Perez","sequence":"first","affiliation":[{"name":"School of Medicine and Surgery Hospital San Carlos, Universidad Complutense Madrid Spain"}],"role":[{"role":"author","vocabulary":"crossref"}]},{"given":"Javier","family":"Arias\u2010Diaz","sequence":"additional","affiliation":[{"name":"School of Medicine and Surgery Hospital San Carlos, Universidad Complutense Madrid Spain"}],"role":[{"role":"author","vocabulary":"crossref"}]},{"given":"Elena","family":"Vara","sequence":"additional","affiliation":[{"name":"Department of Biochemistry, Facultad de Medicina Ciudad Universitaria 28040 Madrid Spain"}],"role":[{"role":"author","vocabulary":"crossref"}]},{"given":"Jose L.","family":"Balibrea","sequence":"additional","affiliation":[{"name":"School of Medicine and Surgery Hospital San Carlos, Universidad Complutense Madrid Spain"}],"role":[{"role":"author","vocabulary":"crossref"}]}],"member":"311","published-online":{"date-parts":[[2005,8,11]]},"reference":[{"key":"e_1_2_5_2_2","doi-asserted-by":"publisher","DOI":"10.2337\/diab.46.11.1853"},{"key":"e_1_2_5_3_2","doi-asserted-by":"publisher","DOI":"10.1161\/01.CIR.93.1.161"},{"key":"e_1_2_5_4_2","doi-asserted-by":"publisher","DOI":"10.1002\/bjs.1800791006"},{"key":"e_1_2_5_5_2","doi-asserted-by":"publisher","DOI":"10.1016\/0026-0495(92)90093-P"},{"key":"e_1_2_5_6_2","doi-asserted-by":"publisher","DOI":"10.2337\/diabetes.49.11.1924"},{"key":"e_1_2_5_7_2","doi-asserted-by":"publisher","DOI":"10.1006\/jsre.2002.6370"},{"key":"e_1_2_5_8_2","doi-asserted-by":"publisher","DOI":"10.1007\/BF00400566"},{"key":"e_1_2_5_9_2","doi-asserted-by":"publisher","DOI":"10.1210\/endo-117-5-1735"},{"key":"e_1_2_5_10_2","doi-asserted-by":"publisher","DOI":"10.1016\/0022\u20104804(90)90257\u20103"},{"key":"e_1_2_5_11_2","doi-asserted-by":"publisher","DOI":"10.1093\/oxfordjournals.bmb.a072626"},{"key":"e_1_2_5_12_2","doi-asserted-by":"publisher","DOI":"10.1016\/0531\u20105565(87)90045\u20103"},{"key":"e_1_2_5_13_2","doi-asserted-by":"publisher","DOI":"10.1001\/archsurg.1995.01430120041006"},{"key":"e_1_2_5_14_2","doi-asserted-by":"publisher","DOI":"10.2337\/diab.23.11.889"},{"key":"e_1_2_5_15_2","doi-asserted-by":"publisher","DOI":"10.1007\/BF00400632"},{"key":"e_1_2_5_16_2","first-page":"58","article-title":"Accentuated oxidative stress following reperfusion injury in diabetic rats","volume":"21","author":"Edrees WK","year":"2002","journal-title":"Int Angiol"},{"key":"e_1_2_5_17_2","doi-asserted-by":"publisher","DOI":"10.1007\/s001250051449"},{"key":"e_1_2_5_18_2","doi-asserted-by":"publisher","DOI":"10.2337\/diacare.21.4.487"},{"key":"e_1_2_5_19_2","first-page":"102","article-title":"Explosion respiratoire des polynucl\u00e9aris neutrophiles et profil des cytokines chez le diab\u00e9tique non insulin\u2010d\u00e9pendant","volume":"20","author":"Nguyen P","year":"1995","journal-title":"J. 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