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The prize, endowed with 37,500\u202f\u20ac, is the second highest medical award in Germany and serves to promote clinical, therapeutic, and experimental research work in the field of rheumatology. In June 2022, the 50-year anniversary was celebrated. In the symposium preceding the award ceremony, an overview was given on the significance of spondyloarthritis for the work of the awardees in the past 30\u00a0years. This overview has now been put together to inform the interested community of the work performed, including the opinion of the awardees regarding what they consider to be their most important contribution.<\/jats:p>","DOI":"10.1007\/s00393-024-01496-w","type":"journal-article","created":{"date-parts":[[2024,6,12]],"date-time":"2024-06-12T06:01:40Z","timestamp":1718172100000},"page":"563-574","update-policy":"http:\/\/dx.doi.org\/10.1007\/springer_crossmark_policy","source":"Crossref","is-referenced-by-count":0,"title":["Looking back on 51\u00a0years of the Carol Nachman Prize in Rheumatology\u2014significance for the field of spondyloarthritis research","R\u00fcckblick auf 51\u00a0Jahre Carol-Nachman-Preis in der Rheumatologie \u2013 Bedeutung f\u00fcr den Bereich der Spondyloarthritisforschung"],"prefix":"10.1007","volume":"83","author":[{"given":"J\u00fcrgen","family":"Braun","sequence":"first","affiliation":[],"role":[{"role":"author","vocabulary":"crossref"}]},{"given":"Joachim","family":"Sieper","sequence":"additional","affiliation":[],"role":[{"role":"author","vocabulary":"crossref"}]},{"given":"Elisabeth","family":"M\u00e4rker-Hermann","sequence":"additional","affiliation":[],"role":[{"role":"author","vocabulary":"crossref"}]}],"member":"297","published-online":{"date-parts":[[2024,6,12]]},"reference":[{"issue":"5","key":"1496_CR1","doi-asserted-by":"publisher","first-page":"1099","DOI":"10.1016\/0092-8674(90)90512-D","volume":"63","author":"RE Hammer","year":"1990","unstructured":"Hammer\u00a0RE, Maika\u00a0SD, Richardson\u00a0JA, Tang\u00a0JP, Taurog\u00a0JD (1990) Spontaneous inflammatory disease in transgenic rats expressing HLA-B27 and human beta 2m: an animal model of HLA-B27-associated human disorders. 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