{"status":"ok","message-type":"work","message-version":"1.0.0","message":{"indexed":{"date-parts":[[2026,8,17]],"date-time":"2026-08-17T16:42:44Z","timestamp":1786984964568,"version":"build-2736575974"},"reference-count":56,"publisher":"Elsevier BV","issue":"1","license":[{"start":{"date-parts":[[2019,1,1]],"date-time":"2019-01-01T00:00:00Z","timestamp":1546300800000},"content-version":"tdm","delay-in-days":0,"URL":"https:\/\/www.elsevier.com\/tdm\/userlicense\/1.0\/"},{"start":{"date-parts":[[2019,1,1]],"date-time":"2019-01-01T00:00:00Z","timestamp":1546300800000},"content-version":"tdm","delay-in-days":0,"URL":"https:\/\/www.elsevier.com\/legal\/tdmrep-license"},{"start":{"date-parts":[[2020,1,1]],"date-time":"2020-01-01T00:00:00Z","timestamp":1577836800000},"content-version":"vor","delay-in-days":365,"URL":"http:\/\/www.elsevier.com\/open-access\/userlicense\/1.0\/"}],"funder":[{"DOI":"10.13039\/501100001809","name":"National Natural Science Foundation of China","doi-asserted-by":"publisher","award":["31371157"],"award-info":[{"award-number":["31371157"]}],"id":[{"id":"10.13039\/501100001809","id-type":"DOI","asserted-by":"publisher"}]},{"DOI":"10.13039\/501100001809","name":"National Natural Science Foundation of China","doi-asserted-by":"publisher","award":["81300193"],"award-info":[{"award-number":["81300193"]}],"id":[{"id":"10.13039\/501100001809","id-type":"DOI","asserted-by":"publisher"}]},{"DOI":"10.13039\/501100004608","name":"Natural Science Foundation of Jiangsu Province","doi-asserted-by":"publisher","award":["BK20171482"],"award-info":[{"award-number":["BK20171482"]}],"id":[{"id":"10.13039\/501100004608","id-type":"DOI","asserted-by":"publisher"}]},{"name":"Natural Science Foundation for Young Scholars of Jiangsu Province","award":["BK20130887"],"award-info":[{"award-number":["BK20130887"]}]},{"DOI":"10.13039\/501100012246","name":"Priority Academic Program Development of Jiangsu Higher Education Institutions","doi-asserted-by":"crossref","id":[{"id":"10.13039\/501100012246","id-type":"DOI","asserted-by":"crossref"}]}],"content-domain":{"domain":["elsevier.com","sciencedirect.com"],"crossmark-restriction":true},"short-container-title":["Biochimica et Biophysica Acta (BBA) - Molecular Basis of Disease"],"published-print":{"date-parts":[[2019,1]]},"DOI":"10.1016\/j.bbadis.2018.08.016","type":"journal-article","created":{"date-parts":[[2018,8,16]],"date-time":"2018-08-16T07:22:58Z","timestamp":1534404178000},"page":"218-229","update-policy":"https:\/\/doi.org\/10.1016\/elsevier_cm_policy","source":"Crossref","is-referenced-by-count":21,"title":["Galectin-1 attenuates cardiomyocyte hypertrophy through splice-variant specific modulation of CaV1.2 calcium channel"],"prefix":"10.1016","volume":"1865","author":[{"given":"Jia","family":"Fan","sequence":"first","affiliation":[],"role":[{"vocabulary":"crossref","role":"author"}]},{"given":"Wenyong","family":"Fan","sequence":"additional","affiliation":[],"role":[{"vocabulary":"crossref","role":"author"}]},{"given":"Jianzhen","family":"Lei","sequence":"additional","affiliation":[],"role":[{"vocabulary":"crossref","role":"author"}]},{"given":"Yingying","family":"Zhou","sequence":"additional","affiliation":[],"role":[{"vocabulary":"crossref","role":"author"}]},{"given":"Hongfei","family":"Xu","sequence":"additional","affiliation":[],"role":[{"vocabulary":"crossref","role":"author"}]},{"given":"Isha","family":"Kapoor","sequence":"additional","affiliation":[],"role":[{"vocabulary":"crossref","role":"author"}]},{"given":"Guoqing","family":"Zhu","sequence":"additional","affiliation":[],"role":[{"vocabulary":"crossref","role":"author"}]},{"given":"Juejin","family":"Wang","sequence":"additional","affiliation":[],"role":[{"vocabulary":"crossref","role":"author"}]}],"member":"78","reference":[{"key":"10.1016\/j.bbadis.2018.08.016_bb0005","doi-asserted-by":"crossref","first-page":"199","DOI":"10.1007\/s11906-009-0035-4","article-title":"The role of cardiac autonomic function in hypertension and cardiovascular disease","volume":"11","author":"Palatini","year":"2009","journal-title":"Curr. Hypertens. Rep."},{"key":"10.1016\/j.bbadis.2018.08.016_bb0010","doi-asserted-by":"crossref","first-page":"H1177","DOI":"10.1152\/ajpheart.00007.2015","article-title":"Chasing cardiac physiology and pathology down the CaMKII cascade","volume":"308","author":"Mattiazzi","year":"2015","journal-title":"Am. J. Physiol. Heart Circ. Physiol."},{"key":"10.1016\/j.bbadis.2018.08.016_bb0015","doi-asserted-by":"crossref","first-page":"36","DOI":"10.3389\/fphar.2014.00036","article-title":"Integrated mechanisms of CaMKII-dependent ventricular remodeling","volume":"5","author":"Kreusser","year":"2014","journal-title":"Front. Pharmacol."},{"key":"10.1016\/j.bbadis.2018.08.016_bb0020","doi-asserted-by":"crossref","first-page":"631","DOI":"10.1016\/j.cardiores.2006.11.005","article-title":"Role of Ca2+\/calmodulin-dependent protein kinase (CaMK) in excitation-contraction coupling in the heart","volume":"73","author":"Maier","year":"2007","journal-title":"Cardiovasc. Res."},{"key":"10.1016\/j.bbadis.2018.08.016_bb0025","doi-asserted-by":"crossref","first-page":"460","DOI":"10.1016\/j.yjmcc.2010.11.012","article-title":"Calcium influx through Cav1.2 is a proximal signal for pathological cardiomyocyte hypertrophy","volume":"50","author":"Chen","year":"2011","journal-title":"J. Mol. Cell. Cardiol."},{"key":"10.1016\/j.bbadis.2018.08.016_bb0030","doi-asserted-by":"crossref","first-page":"280","DOI":"10.1172\/JCI58227","article-title":"Decreased cardiac L-type Ca2+ channel activity induces hypertrophy and heart failure in mice","volume":"122","author":"Goonasekera","year":"2012","journal-title":"J. Clin. Invest."},{"key":"10.1016\/j.bbadis.2018.08.016_bb0035","doi-asserted-by":"crossref","first-page":"35247","DOI":"10.1038\/srep35247","article-title":"Aberrant splicing promotes proteasomal degradation of L-type CaV1.2 calcium channels by competitive binding for CaVbeta subunits in cardiac hypertrophy","volume":"6","author":"Hu","year":"2016","journal-title":"Sci. Rep."},{"key":"10.1016\/j.bbadis.2018.08.016_bb0040","doi-asserted-by":"crossref","first-page":"51","DOI":"10.1080\/19336950.2017.1381805","article-title":"Characterization of CaV1.2 exon 33 heterozygous knockout mice and negative correlation between Rbfox1 and CaV1.2 exon 33 expressions in human heart failure","volume":"12","author":"Wang","year":"2018","journal-title":"Channels"},{"key":"10.1016\/j.bbadis.2018.08.016_bb0045","doi-asserted-by":"crossref","first-page":"33","DOI":"10.2165\/00129784-200303010-00004","article-title":"Defining the role of calcium channel antagonists in heart failure due to systolic dysfunction","volume":"3","author":"Mahe","year":"2003","journal-title":"Am. J. Cardiovasc. Drugs"},{"key":"10.1016\/j.bbadis.2018.08.016_bb0050","doi-asserted-by":"crossref","first-page":"1013","DOI":"10.1172\/JCI200214677","article-title":"The L-type calcium channel inhibitor diltiazem prevents cardiomyopathy in a mouse model","volume":"109","author":"Semsarian","year":"2002","journal-title":"J. Clin. Invest."},{"key":"10.1016\/j.bbadis.2018.08.016_bb0055","doi-asserted-by":"crossref","first-page":"879","DOI":"10.1016\/j.cardiores.2004.11.006","article-title":"Benidipine, a long-acting calcium channel blocker, inhibits cardiac remodeling in pressure-overloaded mice","volume":"65","author":"Liao","year":"2005","journal-title":"Cardiovasc. Res."},{"key":"10.1016\/j.bbadis.2018.08.016_bb0060","doi-asserted-by":"crossref","first-page":"44335","DOI":"10.1074\/jbc.M407023200","article-title":"Transcript scanning reveals novel and extensive splice variations in human l-type voltage-gated calcium channel, Cav1.2 alpha1 subunit","volume":"279","author":"Tang","year":"2004","journal-title":"J. Biol. Chem."},{"key":"10.1016\/j.bbadis.2018.08.016_bb0065","doi-asserted-by":"crossref","first-page":"197","DOI":"10.1016\/j.cardiores.2005.06.024","article-title":"Splicing for alternative structures of Cav1.2 Ca2+ channels in cardiac and smooth muscles","volume":"68","author":"Liao","year":"2005","journal-title":"Cardiovasc. Res."},{"key":"10.1016\/j.bbadis.2018.08.016_bb0070","doi-asserted-by":"crossref","first-page":"H680","DOI":"10.1152\/ajpheart.00109.2009","article-title":"Alternative splicing of Cav1.2 channel exons in smooth muscle cells of resistance-size arteries generates currents with unique electrophysiological properties","volume":"297","author":"Cheng","year":"2009","journal-title":"Am. J. Physiol. Heart Circ. Physiol."},{"key":"10.1016\/j.bbadis.2018.08.016_bb0075","doi-asserted-by":"crossref","first-page":"35133","DOI":"10.1074\/jbc.M705478200","article-title":"A smooth muscle Cav1.2 calcium channel splice variant underlies hyperpolarized window current and enhanced state-dependent inhibition by nifedipine","volume":"282","author":"Liao","year":"2007","journal-title":"J. Biol. Chem."},{"key":"10.1016\/j.bbadis.2018.08.016_bb0080","doi-asserted-by":"crossref","first-page":"50329","DOI":"10.1074\/jbc.M409436200","article-title":"Smooth muscle-selective alternatively spliced exon generates functional variation in Cav1.2 calcium channels","volume":"279","author":"Liao","year":"2004","journal-title":"J. Biol. Chem."},{"key":"10.1016\/j.bbadis.2018.08.016_bb0085","doi-asserted-by":"crossref","first-page":"H1820","DOI":"10.1152\/ajpheart.00326.2009","article-title":"CaV1.2 splice variant with exon 9* is critical for regulation of cerebral artery diameter","volume":"297","author":"Nystoriak","year":"2009","journal-title":"Am. J. Physiol. Heart Circ. Physiol."},{"key":"10.1016\/j.bbadis.2018.08.016_bb0090","doi-asserted-by":"crossref","first-page":"1250","DOI":"10.1161\/CIRCRESAHA.111.248849","article-title":"Splice variant specific modulation of CaV1.2 calcium channel by galectin-1 regulates arterial constriction","volume":"109","author":"Wang","year":"2011","journal-title":"Circ. Res."},{"key":"10.1016\/j.bbadis.2018.08.016_bb0095","doi-asserted-by":"crossref","first-page":"1229","DOI":"10.1093\/ajh\/hpv024","article-title":"Expression of calcium channel subunit variants in small mesenteric arteries of WKY and SHR","volume":"28","author":"Cox","year":"2015","journal-title":"Am. J. Hypertens."},{"key":"10.1016\/j.bbadis.2018.08.016_bb0100","doi-asserted-by":"crossref","first-page":"701","DOI":"10.1007\/s00424-009-0652-4","article-title":"Molecular alteration of CaV1.2 calcium channel in chronic myocardial infarction","volume":"458","author":"Liao","year":"2009","journal-title":"Pflugers Arch."},{"key":"10.1016\/j.bbadis.2018.08.016_bb0105","doi-asserted-by":"crossref","first-page":"17024","DOI":"10.1073\/pnas.0606539103","article-title":"Atherosclerosis-related molecular alteration of the human CaV1.2 calcium channel alpha1C subunit","volume":"103","author":"Tiwari","year":"2006","journal-title":"Proc. Natl. Acad. Sci. U. S. A."},{"key":"10.1016\/j.bbadis.2018.08.016_bb0110","doi-asserted-by":"crossref","first-page":"1183","DOI":"10.1161\/HYPERTENSIONAHA.117.09301","article-title":"Aberrant splicing induced by dysregulated Rbfox2 produces enhanced function of CaV1.2 calcium channel and vascular myogenic tone in hypertension","volume":"70","author":"Zhou","year":"2017","journal-title":"Hypertension"},{"key":"10.1016\/j.bbadis.2018.08.016_bb0115","doi-asserted-by":"crossref","first-page":"597","DOI":"10.1016\/0092-8674(94)90498-7","article-title":"Galectins: a family of animal beta-galactoside-binding lectins","volume":"76","author":"Barondes","year":"1994","journal-title":"Cell"},{"key":"10.1016\/j.bbadis.2018.08.016_bb0120","doi-asserted-by":"crossref","first-page":"31","DOI":"10.1016\/j.vph.2016.02.006","article-title":"The emerging role of galectins in cardiovascular disease","volume":"81","author":"van der Hoeven","year":"2016","journal-title":"Vasc. Pharmacol."},{"key":"10.1016\/j.bbadis.2018.08.016_bb0125","doi-asserted-by":"crossref","first-page":"137R","DOI":"10.1093\/glycob\/cwl025","article-title":"Galectin-1: a small protein with major functions","volume":"16","author":"Camby","year":"2006","journal-title":"Glycobiology"},{"key":"10.1016\/j.bbadis.2018.08.016_bb0130","doi-asserted-by":"crossref","DOI":"10.3390\/ijms17091566","article-title":"The role of galectin-1 in cancer progression, and synthetic multivalent systems for the study of galectin-1","volume":"17","author":"Cousin","year":"2016","journal-title":"Int. J. Mol. Sci."},{"key":"10.1016\/j.bbadis.2018.08.016_bb0135","doi-asserted-by":"crossref","first-page":"493","DOI":"10.1016\/S0008-6363(99)00276-X","article-title":"Galectin 1 is involved in vascular smooth muscle cell proliferation","volume":"45","author":"Moiseeva","year":"2000","journal-title":"Cardiovasc. Res."},{"key":"10.1016\/j.bbadis.2018.08.016_bb0140","doi-asserted-by":"crossref","DOI":"10.1161\/CIRCULATIONAHA.117.031231","article-title":"Regulation of blood pressure by targeting CaV1.2-galectin-1 protein interaction","author":"Hu","year":"2018","journal-title":"Circulation"},{"key":"10.1016\/j.bbadis.2018.08.016_bb0145","doi-asserted-by":"crossref","first-page":"29","DOI":"10.1016\/j.ajpath.2012.09.022","article-title":"Galectin-1 controls cardiac inflammation and ventricular remodeling during acute myocardial infarction","volume":"182","author":"Seropian","year":"2013","journal-title":"Am. J. Pathol."},{"key":"10.1016\/j.bbadis.2018.08.016_bb0150","doi-asserted-by":"crossref","DOI":"10.1371\/journal.pone.0086994","article-title":"Galectin-1 in early acute myocardial infarction","volume":"9","author":"Al-Salam","year":"2014","journal-title":"PLoS One"},{"key":"10.1016\/j.bbadis.2018.08.016_bb0155","doi-asserted-by":"crossref","first-page":"9840","DOI":"10.1074\/jbc.M110.197079","article-title":"Cardiac myocyte-specific ablation of follistatin-like 3 attenuates stress-induced myocardial hypertrophy","volume":"286","author":"Shimano","year":"2011","journal-title":"J. Biol. Chem."},{"key":"10.1016\/j.bbadis.2018.08.016_bb0160","doi-asserted-by":"crossref","first-page":"796","DOI":"10.1161\/CIRCULATIONAHA.111.031229","article-title":"Critical role for stromal interaction molecule 1 in cardiac hypertrophy","volume":"124","author":"Hulot","year":"2011","journal-title":"Circulation"},{"key":"10.1016\/j.bbadis.2018.08.016_bb0165","doi-asserted-by":"crossref","first-page":"866","DOI":"10.1161\/HYPERTENSIONAHA.115.07042","article-title":"Cardiac-specific EPI64C blunts pressure overload-induced cardiac hypertrophy","volume":"67","author":"Zhu","year":"2016","journal-title":"Hypertension"},{"key":"10.1016\/j.bbadis.2018.08.016_bb0170","doi-asserted-by":"crossref","first-page":"578","DOI":"10.1161\/01.RES.0000012223.86441.A1","article-title":"Reduction of I(to) causes hypertrophy in neonatal rat ventricular myocytes","volume":"90","author":"Kassiri","year":"2002","journal-title":"Circ. Res."},{"key":"10.1016\/j.bbadis.2018.08.016_bb0175","doi-asserted-by":"crossref","first-page":"261","DOI":"10.1016\/j.yjmcc.2009.04.017","article-title":"Functional protein expression of multiple sodium channel alpha- and beta-subunit isoforms in neonatal cardiomyocytes","volume":"48","author":"Kaufmann","year":"2010","journal-title":"J. Mol. Cell. Cardiol."},{"key":"10.1016\/j.bbadis.2018.08.016_bb0180","doi-asserted-by":"crossref","first-page":"15841","DOI":"10.1074\/jbc.M115.684118","article-title":"Loss of mouse P2Y6 nucleotide receptor is associated with physiological macrocardia and amplified pathological cardiac hypertrophy","volume":"291","author":"Clouet","year":"2016","journal-title":"J. Biol. Chem."},{"key":"10.1016\/j.bbadis.2018.08.016_bb0185","doi-asserted-by":"crossref","first-page":"2431","DOI":"10.1097\/HJH.0000000000000723","article-title":"Modulation of CaV1.2 calcium channel by neuropeptide W regulates vascular myogenic tone via G protein-coupled receptor 7","volume":"33","author":"Ji","year":"2015","journal-title":"J. Hypertens."},{"key":"10.1016\/j.bbadis.2018.08.016_bb0190","doi-asserted-by":"crossref","first-page":"527","DOI":"10.1161\/CIRCRESAHA.113.300895","article-title":"Intracellular dyssynchrony of diastolic cytosolic [Ca2+] decay in ventricular cardiomyocytes in cardiac remodeling and human heart failure","volume":"113","author":"Hohendanner","year":"2013","journal-title":"Circ. Res."},{"key":"10.1016\/j.bbadis.2018.08.016_bb0195","doi-asserted-by":"crossref","first-page":"143","DOI":"10.1016\/j.ymeth.2008.09.025","article-title":"Chemical calcium indicators","volume":"46","author":"Paredes","year":"2008","journal-title":"Methods"},{"key":"10.1016\/j.bbadis.2018.08.016_bb0200","doi-asserted-by":"crossref","first-page":"12","DOI":"10.2174\/1874467208666150507103417","article-title":"Regulation of cardiac calcium channels in the fight-or-flight response","volume":"8","author":"Catterall","year":"2015","journal-title":"Curr. Mol. Pharmacol."},{"key":"10.1016\/j.bbadis.2018.08.016_bb0205","doi-asserted-by":"crossref","first-page":"2342","DOI":"10.1073\/pnas.0813013106","article-title":"The delta isoform of CaM kinase II is required for pathological cardiac hypertrophy and remodeling after pressure overload","volume":"106","author":"Backs","year":"2009","journal-title":"Proc. Natl. Acad. Sci. U. S. A."},{"key":"10.1016\/j.bbadis.2018.08.016_bb0210","doi-asserted-by":"crossref","first-page":"2163","DOI":"10.1074\/jbc.270.5.2163","article-title":"Stabilization of calmodulin-dependent protein kinase II through the autoinhibitory domain","volume":"270","author":"Ishida","year":"1995","journal-title":"J. Biol. Chem."},{"key":"10.1016\/j.bbadis.2018.08.016_bb0215","doi-asserted-by":"crossref","first-page":"281","DOI":"10.1016\/j.cell.2014.09.019","article-title":"gammaCaMKII shuttles Ca2+\/CaM to the nucleus to trigger CREB phosphorylation and gene expression","volume":"159","author":"Ma","year":"2014","journal-title":"Cell"},{"key":"10.1016\/j.bbadis.2018.08.016_bb0220","doi-asserted-by":"crossref","first-page":"1359","DOI":"10.1172\/JCI61084","article-title":"HDAC4 controls histone methylation in response to elevated cardiac load","volume":"123","author":"Hohl","year":"2013","journal-title":"J. Clin. Invest."},{"key":"10.1016\/j.bbadis.2018.08.016_bb0225","doi-asserted-by":"crossref","first-page":"1853","DOI":"10.1172\/JCI27438","article-title":"CaM kinase II selectively signals to histone deacetylase 4 during cardiomyocyte hypertrophy","volume":"116","author":"Backs","year":"2006","journal-title":"J. Clin. Invest."},{"key":"10.1016\/j.bbadis.2018.08.016_bb0230","doi-asserted-by":"crossref","first-page":"118","DOI":"10.1016\/j.bbamcr.2007.11.003","article-title":"Differential splicing patterns of L-type calcium channel Cav1.2 subunit in hearts of spontaneously hypertensive rats and Wistar Kyoto rats","volume":"1783","author":"Tang","year":"2008","journal-title":"Biochim. Biophys. Acta"},{"key":"10.1016\/j.bbadis.2018.08.016_bb0235","doi-asserted-by":"crossref","first-page":"67","DOI":"10.1038\/368067a0","article-title":"Calcium channel beta-subunit binds to a conserved motif in the I-II cytoplasmic linker of the alpha 1-subunit","volume":"368","author":"Pragnell","year":"1994","journal-title":"Nature"},{"key":"10.1016\/j.bbadis.2018.08.016_bb0240","doi-asserted-by":"crossref","first-page":"26465","DOI":"10.1074\/jbc.271.43.26465","article-title":"Identification of palmitoylation sites within the L-type calcium channel beta2a subunit and effects on channel function","volume":"271","author":"Chien","year":"1996","journal-title":"J. Biol. Chem."},{"key":"10.1016\/j.bbadis.2018.08.016_bb0245","doi-asserted-by":"crossref","first-page":"4690","DOI":"10.1073\/pnas.95.8.4690","article-title":"Unique regulatory properties of the type 2a Ca2+ channel beta subunit caused by palmitoylation","volume":"95","author":"Qin","year":"1998","journal-title":"Proc. Natl. Acad. Sci. U. S. A."},{"key":"10.1016\/j.bbadis.2018.08.016_bb0250","doi-asserted-by":"crossref","first-page":"21623","DOI":"10.1074\/jbc.M211164200","article-title":"Cardiac L-type calcium channel beta-subunits expressed in human heart have differential effects on single channel characteristics","volume":"278","author":"Hullin","year":"2003","journal-title":"J. Biol. Chem."},{"key":"10.1016\/j.bbadis.2018.08.016_bb0255","doi-asserted-by":"crossref","first-page":"435","DOI":"10.1113\/jphysiol.2002.018515","article-title":"Novel functional properties of Ca2+ channel beta subunits revealed by their expression in adult rat heart cells","volume":"541","author":"Colecraft","year":"2002","journal-title":"J. Physiol."},{"key":"10.1016\/j.bbadis.2018.08.016_bb0260","doi-asserted-by":"crossref","first-page":"8","DOI":"10.1161\/01.RES.87.1.8","article-title":"Cardiac intracellular calcium release channels: role in heart failure","volume":"87","author":"Marks","year":"2000","journal-title":"Circ. Res."},{"key":"10.1016\/j.bbadis.2018.08.016_bb0265","doi-asserted-by":"crossref","first-page":"904","DOI":"10.1161\/01.RES.0000069685.20258.F1","article-title":"Transgenic CaMKIIdeltaC overexpression uniquely alters cardiac myocyte Ca2+ handling: reduced SR Ca2+ load and activated SR Ca2+ release","volume":"92","author":"Maier","year":"2003","journal-title":"Circ. Res."},{"key":"10.1016\/j.bbadis.2018.08.016_bb0270","doi-asserted-by":"crossref","first-page":"25560","DOI":"10.1074\/jbc.M508661200","article-title":"Calmodulin kinase II is involved in voltage-dependent facilitation of the L-type Cav1.2 calcium channel: identification of the phosphorylation sites","volume":"281","author":"Lee","year":"2006","journal-title":"J. Biol. Chem."},{"key":"10.1016\/j.bbadis.2018.08.016_bb0275","doi-asserted-by":"crossref","first-page":"10285","DOI":"10.1073\/pnas.0914287107","article-title":"Facilitation of murine cardiac L-type CaV1.2 channel is modulated by calmodulin kinase II-dependent phosphorylation of S1512 and S1570","volume":"107","author":"Blaich","year":"2010","journal-title":"Proc. Natl. Acad. Sci. U. S. A."},{"key":"10.1016\/j.bbadis.2018.08.016_bb0280","doi-asserted-by":"crossref","first-page":"15","DOI":"10.1161\/01.RES.0000197782.21444.8f","article-title":"Control of cardiac growth by histone acetylation\/deacetylation","volume":"98","author":"Backs","year":"2006","journal-title":"Circ. Res."}],"container-title":["Biochimica et Biophysica Acta (BBA) - Molecular Basis of Disease"],"original-title":[],"language":"en","link":[{"URL":"https:\/\/api.elsevier.com\/content\/article\/PII:S0925443918303016?httpAccept=text\/xml","content-type":"text\/xml","content-version":"vor","intended-application":"text-mining"},{"URL":"https:\/\/api.elsevier.com\/content\/article\/PII:S0925443918303016?httpAccept=text\/plain","content-type":"text\/plain","content-version":"vor","intended-application":"text-mining"}],"deposited":{"date-parts":[[2025,10,14]],"date-time":"2025-10-14T04:45:40Z","timestamp":1760417140000},"score":1,"resource":{"primary":{"URL":"https:\/\/linkinghub.elsevier.com\/retrieve\/pii\/S0925443918303016"}},"subtitle":[],"short-title":[],"issued":{"date-parts":[[2019,1]]},"references-count":56,"journal-issue":{"issue":"1","published-print":{"date-parts":[[2019,1]]}},"alternative-id":["S0925443918303016"],"URL":"https:\/\/doi.org\/10.1016\/j.bbadis.2018.08.016","relation":{},"ISSN":["0925-4439"],"issn-type":[{"value":"0925-4439","type":"print"}],"subject":[],"published":{"date-parts":[[2019,1]]},"assertion":[{"value":"Elsevier","name":"publisher","label":"This article is maintained by"},{"value":"Galectin-1 attenuates cardiomyocyte hypertrophy through splice-variant specific modulation of CaV1.2 calcium channel","name":"articletitle","label":"Article Title"},{"value":"Biochimica et Biophysica Acta (BBA) - Molecular Basis of Disease","name":"journaltitle","label":"Journal Title"},{"value":"https:\/\/doi.org\/10.1016\/j.bbadis.2018.08.016","name":"articlelink","label":"CrossRef DOI link to publisher maintained version"},{"value":"article","name":"content_type","label":"Content Type"},{"value":"\u00a9 2018 Elsevier B.V.","name":"copyright","label":"Copyright"}]}}