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Deficits are also evident in murine null mutants such as <jats:italic>Zic1<\/jats:italic>, <jats:italic>Fgfr1<\/jats:italic>\/<jats:italic>FgFr2<\/jats:italic>, and <jats:italic>Xpa<\/jats:italic>\/<jats:italic>Ercc8<\/jats:italic>. Behavioral deficits are time\u2010dependent following X\u2010irradiated\u2010 or aspiration\u2010induced lesions of the cerebellum in rats. In addition, motor functions are deficient after lesions in cerebellar\u2010related pathways. As in animal subjects, sensorimotor disturbances have been described in children with cerebellar lesions. 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