{"status":"ok","message-type":"work","message-version":"1.0.0","message":{"indexed":{"date-parts":[[2026,8,4]],"date-time":"2026-08-04T14:21:02Z","timestamp":1785853262959,"version":"3.56.0"},"reference-count":50,"publisher":"Elsevier BV","license":[{"start":{"date-parts":[[2020,12,1]],"date-time":"2020-12-01T00:00:00Z","timestamp":1606780800000},"content-version":"tdm","delay-in-days":0,"URL":"https:\/\/www.elsevier.com\/tdm\/userlicense\/1.0\/"},{"start":{"date-parts":[[2020,12,1]],"date-time":"2020-12-01T00:00:00Z","timestamp":1606780800000},"content-version":"tdm","delay-in-days":0,"URL":"https:\/\/www.elsevier.com\/legal\/tdmrep-license"}],"funder":[{"DOI":"10.13039\/100000968","name":"American Heart Association","doi-asserted-by":"publisher","id":[{"id":"10.13039\/100000968","id-type":"DOI","asserted-by":"publisher"}]},{"DOI":"10.13039\/100000050","name":"National Heart, Lung, and Blood Institute","doi-asserted-by":"publisher","id":[{"id":"10.13039\/100000050","id-type":"DOI","asserted-by":"publisher"}]}],"content-domain":{"domain":["clinicalkey.com","clinicalkey.com.au","clinicalkey.es","clinicalkey.fr","clinicalkey.jp","jmmc-online.com","elsevier.com","sciencedirect.com"],"crossmark-restriction":true},"short-container-title":["Journal of Molecular and Cellular Cardiology"],"published-print":{"date-parts":[[2020,12]]},"DOI":"10.1016\/j.yjmcc.2020.09.007","type":"journal-article","created":{"date-parts":[[2020,9,21]],"date-time":"2020-09-21T22:13:06Z","timestamp":1600726386000},"page":"73-81","update-policy":"https:\/\/doi.org\/10.1016\/elsevier_cm_policy","source":"Crossref","is-referenced-by-count":35,"special_numbering":"C","title":["CaMKII exacerbates heart failure progression by activating class I HDACs"],"prefix":"10.1016","volume":"149","author":[{"given":"Manling","family":"Zhang","sequence":"first","affiliation":[],"role":[{"vocabulary":"crossref","role":"author"}]},{"given":"Xue","family":"Yang","sequence":"additional","affiliation":[],"role":[{"vocabulary":"crossref","role":"author"}]},{"given":"Raymond J.","family":"Zimmerman","sequence":"additional","affiliation":[],"role":[{"vocabulary":"crossref","role":"author"}]},{"given":"Qin","family":"Wang","sequence":"additional","affiliation":[],"role":[{"vocabulary":"crossref","role":"author"}]},{"given":"Mark A.","family":"Ross","sequence":"additional","affiliation":[],"role":[{"vocabulary":"crossref","role":"author"}]},{"given":"Jonathan M.","family":"Granger","sequence":"additional","affiliation":[],"role":[{"vocabulary":"crossref","role":"author"}]},{"given":"Elizabeth D.","family":"Luczak","sequence":"additional","affiliation":[],"role":[{"vocabulary":"crossref","role":"author"}]},{"given":"Djahida","family":"Bedja","sequence":"additional","affiliation":[],"role":[{"vocabulary":"crossref","role":"author"}]},{"given":"Hong","family":"Jiang","sequence":"additional","affiliation":[],"role":[{"vocabulary":"crossref","role":"author"}]},{"given":"Ning","family":"Feng","sequence":"additional","affiliation":[],"role":[{"vocabulary":"crossref","role":"author"}]}],"member":"78","reference":[{"issue":"16","key":"10.1016\/j.yjmcc.2020.09.007_bb0005","first-page":"e240","article-title":"2013 ACCF\/AHA guideline for the management of heart failure: a report of the American College of Cardiology Foundation\/American Heart Association Task Force on practice guidelines","volume":"128","author":"M. Writing Committee","year":"2013","journal-title":"Circulation"},{"issue":"2","key":"10.1016\/j.yjmcc.2020.09.007_bb0010","doi-asserted-by":"crossref","first-page":"322","DOI":"10.1016\/j.yjmcc.2009.10.016","article-title":"Beta-adrenergic receptor signaling in the heart: role of CaMKII","volume":"48","author":"Grimm","year":"2010","journal-title":"J. Mol. Cell. Cardiol."},{"issue":"3","key":"10.1016\/j.yjmcc.2020.09.007_bb0015","doi-asserted-by":"crossref","first-page":"462","DOI":"10.1016\/j.cell.2008.02.048","article-title":"A dynamic pathway for calcium-independent activation of CaMKII by methionine oxidation","volume":"133","author":"Erickson","year":"2008","journal-title":"Cell"},{"issue":"4","key":"10.1016\/j.yjmcc.2020.09.007_bb0020","doi-asserted-by":"crossref","first-page":"409","DOI":"10.1038\/nm1215","article-title":"Calmodulin kinase II inhibition protects against structural heart disease","volume":"11","author":"Zhang","year":"2005","journal-title":"Nat. Med."},{"issue":"7","key":"10.1016\/j.yjmcc.2020.09.007_bb0025","doi-asserted-by":"crossref","first-page":"2342","DOI":"10.1073\/pnas.0813013106","article-title":"The delta isoform of CaM kinase II is required for pathological cardiac hypertrophy and remodeling after pressure overload","volume":"106","author":"Backs","year":"2009","journal-title":"Proc. Natl. Acad. Sci. U. S. A."},{"issue":"5","key":"10.1016\/j.yjmcc.2020.09.007_bb0030","doi-asserted-by":"crossref","first-page":"1230","DOI":"10.1172\/JCI38022","article-title":"Requirement for Ca2+\/calmodulin-dependent kinase II in the transition from pressure overload-induced cardiac hypertrophy to heart failure in mice","volume":"119","author":"Ling","year":"2009","journal-title":"J. Clin. Invest."},{"issue":"Pt B","key":"10.1016\/j.yjmcc.2020.09.007_bb0035","doi-asserted-by":"crossref","first-page":"160","DOI":"10.1016\/j.yjmcc.2015.10.014","article-title":"Oxidant stress promotes disease by activating CaMKII","volume":"89","author":"Anderson","year":"2015","journal-title":"J. Mol. Cell. Cardiol."},{"key":"10.1016\/j.yjmcc.2020.09.007_bb0040","doi-asserted-by":"crossref","first-page":"102","DOI":"10.1016\/j.yjmcc.2016.12.007","article-title":"CaMKII is a nodal signal for multiple programmed cell death pathways in heart","volume":"103","author":"Feng","year":"2017","journal-title":"J. Mol. Cell. Cardiol."},{"issue":"2","key":"10.1016\/j.yjmcc.2020.09.007_bb0045","doi-asserted-by":"crossref","first-page":"175","DOI":"10.1038\/nm.4017","article-title":"CaMKII is a RIP3 substrate mediating ischemia- and oxidative stress-induced myocardial necroptosis","volume":"22","author":"Zhang","year":"2016","journal-title":"Nat. Med."},{"issue":"6","key":"10.1016\/j.yjmcc.2020.09.007_bb0050","doi-asserted-by":"crossref","first-page":"664","DOI":"10.1161\/CIRCHEARTFAILURE.109.865279","article-title":"Calcium\/calmodulin-dependent protein kinase II contributes to cardiac arrhythmogenesis in heart failure","volume":"2","author":"Sag","year":"2009","journal-title":"Circ. Heart Fail."},{"issue":"22","key":"10.1016\/j.yjmcc.2020.09.007_bb0055","doi-asserted-by":"crossref","first-page":"2530","DOI":"10.1161\/CIRCULATIONAHA.118.034621","article-title":"Inflammation and NLRP3 inflammasome activation initiated in response to pressure overload by Ca(2+)\/calmodulin-dependent protein kinase II delta signaling in cardiomyocytes are essential for adverse cardiac remodeling","volume":"138","author":"Suetomi","year":"2018","journal-title":"Circulation"},{"issue":"12","key":"10.1016\/j.yjmcc.2020.09.007_bb0060","doi-asserted-by":"crossref","first-page":"1610","DOI":"10.1038\/nm.2506","article-title":"Oxidation of CaMKII determines the cardiotoxic effects of aldosterone","volume":"17","author":"He","year":"2011","journal-title":"Nat. Med."},{"issue":"5","key":"10.1016\/j.yjmcc.2020.09.007_bb0065","doi-asserted-by":"crossref","first-page":"e003840","DOI":"10.1161\/CIRCHEARTFAILURE.117.003840","article-title":"Calcium\/calmodulin-dependent protein kinase II activity persists during chronic beta-adrenoceptor blockade in experimental and human heart failure","volume":"10","author":"Dewenter","year":"2017","journal-title":"Circ. Heart Fail."},{"issue":"7","key":"10.1016\/j.yjmcc.2020.09.007_bb0070","doi-asserted-by":"crossref","first-page":"1853","DOI":"10.1172\/JCI27438","article-title":"CaM kinase II selectively signals to histone deacetylase 4 during cardiomyocyte hypertrophy","volume":"116","author":"Backs","year":"2006","journal-title":"J. Clin. Invest."},{"issue":"10","key":"10.1016\/j.yjmcc.2020.09.007_bb0075","doi-asserted-by":"crossref","first-page":"3437","DOI":"10.1128\/MCB.01611-07","article-title":"Histone deacetylase 5 acquires calcium\/calmodulin-dependent kinase II responsiveness by oligomerization with histone deacetylase 4","volume":"28","author":"Backs","year":"2008","journal-title":"Mol. Cell. Biol."},{"issue":"1","key":"10.1016\/j.yjmcc.2020.09.007_bb0080","doi-asserted-by":"crossref","first-page":"91","DOI":"10.1016\/j.jmb.2004.10.033","article-title":"Mechanism of recruitment of class II histone deacetylases by myocyte enhancer factor-2","volume":"345","author":"Han","year":"2005","journal-title":"J. Mol. Biol."},{"issue":"3","key":"10.1016\/j.yjmcc.2020.09.007_bb0085","doi-asserted-by":"crossref","first-page":"324","DOI":"10.1038\/nm1552","article-title":"Hdac2 regulates the cardiac hypertrophic response by modulating Gsk3 beta activity","volume":"13","author":"Trivedi","year":"2007","journal-title":"Nat. Med."},{"issue":"24","key":"10.1016\/j.yjmcc.2020.09.007_bb0090","doi-asserted-by":"crossref","first-page":"9806","DOI":"10.1073\/pnas.1301509110","article-title":"Signal-dependent repression of DUSP5 by class I HDACs controls nuclear ERK activity and cardiomyocyte hypertrophy","volume":"110","author":"Ferguson","year":"2013","journal-title":"Proc. Natl. Acad. Sci. U. S. A."},{"issue":"422","key":"10.1016\/j.yjmcc.2020.09.007_bb0095","doi-asserted-by":"crossref","first-page":"ra34","DOI":"10.1126\/scisignal.aad5736","article-title":"Inhibition of class I histone deacetylases blunts cardiac hypertrophy through TSC2-dependent mTOR repression","volume":"9","author":"Morales","year":"2016","journal-title":"Sci. Signal."},{"issue":"3","key":"10.1016\/j.yjmcc.2020.09.007_bb0100","doi-asserted-by":"crossref","first-page":"403","DOI":"10.1038\/s41591-019-0376-8","article-title":"Targeting epigenetic modifications in cancer therapy: erasing the roadmap to cancer","volume":"25","author":"Mohammad","year":"2019","journal-title":"Nat. Med."},{"issue":"8","key":"10.1016\/j.yjmcc.2020.09.007_bb0105","doi-asserted-by":"crossref","first-page":"912","DOI":"10.1161\/01.RES.0000069686.31472.C5","article-title":"The deltaC isoform of CaMKII is activated in cardiac hypertrophy and induces dilated cardiomyopathy and heart failure","volume":"92","author":"Zhang","year":"2003","journal-title":"Circ. Res."},{"issue":"3","key":"10.1016\/j.yjmcc.2020.09.007_bb0110","doi-asserted-by":"crossref","first-page":"1262","DOI":"10.1172\/JCI65268","article-title":"Diabetes increases mortality after myocardial infarction by oxidizing CaMKII","volume":"123","author":"Luo","year":"2013","journal-title":"J. Clin. Invest."},{"issue":"22","key":"10.1016\/j.yjmcc.2020.09.007_bb0115","doi-asserted-by":"crossref","first-page":"6841","DOI":"10.1158\/1078-0432.CCR-09-0547","article-title":"JNJ-26481585, a novel \u201csecond-generation\u201d oral histone deacetylase inhibitor, shows broad-spectrum preclinical antitumoral activity","volume":"15","author":"Arts","year":"2009","journal-title":"Clin. Cancer Res."},{"issue":"8","key":"10.1016\/j.yjmcc.2020.09.007_bb0120","doi-asserted-by":"crossref","first-page":"942","DOI":"10.1161\/CIRCULATIONAHA.112.090977","article-title":"Pathological cardiac hypertrophy alters intracellular targeting of phosphodiesterase type 5 from nitric oxide synthase-3 to natriuretic peptide signaling","volume":"126","author":"Zhang","year":"2012","journal-title":"Circulation"},{"issue":"24","key":"10.1016\/j.yjmcc.2020.09.007_bb0125","doi-asserted-by":"crossref","first-page":"2021","DOI":"10.1016\/j.jacc.2010.08.612","article-title":"Myocardial remodeling is controlled by myocyte-targeted gene regulation of phosphodiesterase type 5","volume":"56","author":"Zhang","year":"2010","journal-title":"J. Am. Coll. Cardiol."},{"issue":"2","key":"10.1016\/j.yjmcc.2020.09.007_bb0130","doi-asserted-by":"crossref","first-page":"581","DOI":"10.1042\/BJ20070779","article-title":"Determination of the class and isoform selectivity of small-molecule histone deacetylase inhibitors","volume":"409","author":"Khan","year":"2008","journal-title":"Biochem. J."},{"issue":"7","key":"10.1016\/j.yjmcc.2020.09.007_bb0135","doi-asserted-by":"crossref","first-page":"2647","DOI":"10.1073\/pnas.1213364110","article-title":"HDAC3-selective inhibitor enhances extinction of cocaine-seeking behavior in a persistent manner","volume":"110","author":"Malvaez","year":"2013","journal-title":"Proc. Natl. Acad. Sci. U. S. A."},{"issue":"3","key":"10.1016\/j.yjmcc.2020.09.007_bb0140","doi-asserted-by":"crossref","first-page":"238","DOI":"10.1038\/nchembio.313","article-title":"Chemical phylogenetics of histone deacetylases","volume":"6","author":"Bradner","year":"2010","journal-title":"Nat. Chem. Biol."},{"issue":"6","key":"10.1016\/j.yjmcc.2020.09.007_bb0145","doi-asserted-by":"crossref","first-page":"690","DOI":"10.1161\/CIRCRESAHA.113.301651","article-title":"Mechanisms of altered Ca(2)(+) handling in heart failure","volume":"113","author":"Luo","year":"2013","journal-title":"Circ. Res."},{"issue":"22","key":"10.1016\/j.yjmcc.2020.09.007_bb0150","doi-asserted-by":"crossref","first-page":"2579","DOI":"10.1161\/CIRCULATIONAHA.106.625467","article-title":"Suppression of class I and II histone deacetylases blunts pressure-overload cardiac hypertrophy","volume":"113","author":"Kong","year":"2006","journal-title":"Circulation"},{"issue":"12","key":"10.1016\/j.yjmcc.2020.09.007_bb0155","doi-asserted-by":"crossref","first-page":"1354","DOI":"10.1161\/CIRCRESAHA.111.248401","article-title":"Location matters: clarifying the concept of nuclear and cytosolic CaMKII subtypes","volume":"109","author":"Mishra","year":"2011","journal-title":"Circ. Res."},{"issue":"48","key":"10.1016\/j.yjmcc.2020.09.007_bb0160","doi-asserted-by":"crossref","first-page":"35078","DOI":"10.1074\/jbc.M707083200","article-title":"CaMKIIdelta isoforms differentially affect calcium handling but similarly regulate HDAC\/MEF2 transcriptional responses","volume":"282","author":"Zhang","year":"2007","journal-title":"J. Biol. Chem."},{"issue":"6","key":"10.1016\/j.yjmcc.2020.09.007_bb0165","doi-asserted-by":"crossref","first-page":"229","DOI":"10.1016\/j.tcm.2012.12.006","article-title":"HDAC-dependent ventricular remodeling","volume":"23","author":"Xie","year":"2013","journal-title":"Trends Cardiovasc. Med."},{"issue":"19","key":"10.1016\/j.yjmcc.2020.09.007_bb0170","doi-asserted-by":"crossref","first-page":"8374","DOI":"10.1128\/MCB.24.19.8374-8385.2004","article-title":"Protein kinases C and D mediate agonist-dependent cardiac hypertrophy through nuclear export of histone deacetylase 5","volume":"24","author":"Vega","year":"2004","journal-title":"Mol. Cell. Biol."},{"issue":"4","key":"10.1016\/j.yjmcc.2020.09.007_bb0175","doi-asserted-by":"crossref","first-page":"479","DOI":"10.1016\/S0092-8674(02)00861-9","article-title":"Class II histone deacetylases act as signal-responsive repressors of cardiac hypertrophy","volume":"110","author":"Zhang","year":"2002","journal-title":"Cell"},{"issue":"19","key":"10.1016\/j.yjmcc.2020.09.007_bb0180","doi-asserted-by":"crossref","first-page":"8467","DOI":"10.1128\/MCB.24.19.8467-8476.2004","article-title":"Histone deacetylases 5 and 9 govern responsiveness of the heart to a subset of stress signals and play redundant roles in heart development","volume":"24","author":"Chang","year":"2004","journal-title":"Mol. Cell. Biol."},{"issue":"14","key":"10.1016\/j.yjmcc.2020.09.007_bb0185","doi-asserted-by":"crossref","first-page":"1790","DOI":"10.1101\/gad.1563807","article-title":"Histone deacetylases 1 and 2 redundantly regulate cardiac morphogenesis, growth, and contractility","volume":"21","author":"Montgomery","year":"2007","journal-title":"Genes Dev."},{"issue":"11","key":"10.1016\/j.yjmcc.2020.09.007_bb0190","doi-asserted-by":"crossref","first-page":"3588","DOI":"10.1172\/JCI35847","article-title":"Maintenance of cardiac energy metabolism by histone deacetylase 3 in mice","volume":"118","author":"Montgomery","year":"2008","journal-title":"J. Clin. Invest."},{"issue":"39","key":"10.1016\/j.yjmcc.2020.09.007_bb0195","doi-asserted-by":"crossref","first-page":"26484","DOI":"10.1074\/jbc.M803686200","article-title":"Transgenic overexpression of Hdac3 in the heart produces increased postnatal cardiac myocyte proliferation but does not induce hypertrophy","volume":"283","author":"Trivedi","year":"2008","journal-title":"J. Biol. Chem."},{"issue":"1","key":"10.1016\/j.yjmcc.2020.09.007_bb0200","doi-asserted-by":"crossref","first-page":"9291","DOI":"10.1038\/s41598-019-45743-6","article-title":"Myocardial death and dysfunction after ischemia-reperfusion injury require CaMKIIdelta oxidation","volume":"9","author":"Wu","year":"2019","journal-title":"Sci. Rep."},{"issue":"6","key":"10.1016\/j.yjmcc.2020.09.007_bb0205","doi-asserted-by":"crossref","first-page":"935","DOI":"10.1161\/CIRCRESAHA.112.276915","article-title":"Ca2+\/Calmodulin-dependent protein kinase II delta mediates myocardial ischemia\/reperfusion injury through nuclear factor-kappaB","volume":"112","author":"Ling","year":"2013","journal-title":"Circ. Res."},{"issue":"10","key":"10.1016\/j.yjmcc.2020.09.007_bb0210","doi-asserted-by":"crossref","first-page":"1231","DOI":"10.15252\/emmm.201403848","article-title":"CaM Kinase II mediates maladaptive post-infarct remodeling and pro-inflammatory chemoattractant signaling but not acute myocardial ischemia\/reperfusion injury","volume":"6","author":"Weinreuter","year":"2014","journal-title":"EMBO Mol. Med."},{"issue":"10","key":"10.1016\/j.yjmcc.2020.09.007_bb0215","doi-asserted-by":"crossref","first-page":"3549","DOI":"10.1096\/fj.08-108548","article-title":"Histone deacetylase inhibition reduces myocardial ischemia-reperfusion injury in mice","volume":"22","author":"Granger","year":"2008","journal-title":"FASEB J."},{"key":"10.1016\/j.yjmcc.2020.09.007_bb0220","doi-asserted-by":"crossref","first-page":"138","DOI":"10.1016\/j.yjmcc.2014.03.005","article-title":"Selective inhibition of class I but not class IIb histone deacetylases exerts cardiac protection from ischemia reperfusion","volume":"72","author":"Aune","year":"2014","journal-title":"J. Mol. Cell. Cardiol."},{"key":"10.1016\/j.yjmcc.2020.09.007_bb0225","doi-asserted-by":"crossref","first-page":"188","DOI":"10.1016\/j.yjmcc.2019.02.013","article-title":"HDAC inhibition as a therapeutic strategy in myocardial ischemia\/reperfusion injury","volume":"129","author":"Xie","year":"2019","journal-title":"J. Mol. Cell. Cardiol."},{"issue":"7471","key":"10.1016\/j.yjmcc.2020.09.007_bb0230","doi-asserted-by":"crossref","first-page":"372","DOI":"10.1038\/nature12537","article-title":"Diabetic hyperglycaemia activates CaMKII and arrhythmias by O-linked glycosylation","volume":"502","author":"Erickson","year":"2013","journal-title":"Nature"},{"key":"10.1016\/j.yjmcc.2020.09.007_bb0235","doi-asserted-by":"crossref","first-page":"151","DOI":"10.1016\/j.yjmcc.2019.04.003","article-title":"Histone deacetylases in cardiovascular and metabolic diseases","volume":"130","author":"Bagchi","year":"2019","journal-title":"J. Mol. Cell. Cardiol."},{"issue":"16","key":"10.1016\/j.yjmcc.2020.09.007_bb0240","doi-asserted-by":"crossref","first-page":"1748","DOI":"10.1161\/CIRCULATIONAHA.113.003313","article-title":"Oxidized Ca(2+)\/calmodulin-dependent protein kinase II triggers atrial fibrillation","volume":"128","author":"Purohit","year":"2013","journal-title":"Circulation"},{"issue":"6","key":"10.1016\/j.yjmcc.2020.09.007_bb0245","doi-asserted-by":"crossref","first-page":"821","DOI":"10.1161\/CIRCRESAHA.117.312536","article-title":"Stress signaling JNK2 crosstalk with CaMKII underlies enhanced atrial arrhythmogenesis","volume":"122","author":"Yan","year":"2018","journal-title":"Circ. Res."},{"issue":"3","key":"10.1016\/j.yjmcc.2020.09.007_bb0250","doi-asserted-by":"crossref","first-page":"e007071","DOI":"10.1161\/CIRCEP.118.007071","article-title":"HDAC (histone deacetylase) inhibitor valproic acid attenuates atrial remodeling and delays the onset of atrial fibrillation in mice","volume":"12","author":"Scholz","year":"2019","journal-title":"Circ. Arrhythm. Electrophysiol."}],"container-title":["Journal of Molecular and Cellular Cardiology"],"original-title":[],"language":"en","link":[{"URL":"https:\/\/api.elsevier.com\/content\/article\/PII:S002228282030287X?httpAccept=text\/xml","content-type":"text\/xml","content-version":"vor","intended-application":"text-mining"},{"URL":"https:\/\/api.elsevier.com\/content\/article\/PII:S002228282030287X?httpAccept=text\/plain","content-type":"text\/plain","content-version":"vor","intended-application":"text-mining"}],"deposited":{"date-parts":[[2025,9,19]],"date-time":"2025-09-19T20:31:48Z","timestamp":1758313908000},"score":1,"resource":{"primary":{"URL":"https:\/\/linkinghub.elsevier.com\/retrieve\/pii\/S002228282030287X"}},"subtitle":[],"short-title":[],"issued":{"date-parts":[[2020,12]]},"references-count":50,"alternative-id":["S002228282030287X"],"URL":"https:\/\/doi.org\/10.1016\/j.yjmcc.2020.09.007","relation":{},"ISSN":["0022-2828"],"issn-type":[{"value":"0022-2828","type":"print"}],"subject":[],"published":{"date-parts":[[2020,12]]},"assertion":[{"value":"Elsevier","name":"publisher","label":"This article is maintained by"},{"value":"CaMKII exacerbates heart failure progression by activating class I HDACs","name":"articletitle","label":"Article Title"},{"value":"Journal of Molecular and Cellular Cardiology","name":"journaltitle","label":"Journal Title"},{"value":"https:\/\/doi.org\/10.1016\/j.yjmcc.2020.09.007","name":"articlelink","label":"CrossRef DOI link to publisher maintained version"},{"value":"article","name":"content_type","label":"Content Type"},{"value":"\u00a9 2020 Elsevier Ltd. All rights reserved.","name":"copyright","label":"Copyright"}]}}