{"status":"ok","message-type":"work","message-version":"1.0.0","message":{"indexed":{"date-parts":[[2026,5,21]],"date-time":"2026-05-21T10:17:27Z","timestamp":1779358647775,"version":"3.51.4"},"reference-count":36,"publisher":"Hogrefe Publishing Group","issue":"9-10","content-domain":{"domain":[],"crossmark-restriction":false},"short-container-title":["Praxis"],"published-print":{"date-parts":[[2018,4]]},"abstract":"<jats:p> Zusammenfassung. Sportliche Aktivit\u00e4ten mit Sprungabl\u00e4ufen stellen f\u00fcr die Kniegelenke eine mechanische Herausforderung dar. H\u00e4ufig projizieren sich chronisch auftretende, atraumatische Beschwerden auf den Patellaunterpol, welcher pathomorphologisch eine patellare Tendinopathie entspricht. Extrinsische und intrinsische Risikofaktoren k\u00f6nnen unterschieden werden. In der klinischen Untersuchung zeigt sich ein meist unauff\u00e4lliges Knie mit einer Druckdolenz am kaudalen Teil der Kniescheibe beim \u00dcbergang zum Ligamentum patellae. In der Bildgebung ist sonografisch ein degenerierter Sehnenanteil der tiefen Patellarsehne zu erkennen, der magnettomografisch mit Hyperintensit\u00e4t des Hoffa-K\u00f6rpers einhergehen kann. Abh\u00e4ngig von der klinischen Entwicklung wird das Jumper\u2019s Knee nach Blazina Grad I\u2013IV eingeteilt. Bis zu 90 % der Patienten k\u00f6nnen konservativ therapiert werden. 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