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BDNF may be released in the mature form, which activates preferentially TrkB receptors, or as proBDNF, which is coupled to the stimulation of the p75<jats:sup>NTR<\/jats:sup>. In the mature form BDNF induces rapid effects on glutamate release, and may induce short\u2010 and long\u2010term effects on the postsynaptic response to the neurotransmitter. BDNF may affect glutamate receptor activity by inducing the phosphorylation of the receptor subunits, which may also affect the interaction with intracellular proteins and, consequently, their recycling and localization to defined postsynaptic sites. Stimulation of the local protein synthesis and transcription activity account for the delayed effects of BDNF on glutamatergic synaptic strength. Several evidences show impaired synaptic plasticity of glutamatergic synapses in diseases where compromised BDNF function has been observed, such as Huntington's disease, depression, anxiety, and the BDNF polymorphism Val66Met, suggesting that upregulating BDNF\u2010activated pathways may be therapeutically relevant. This review focuses on recent advances in the understanding of the regulation of the glutamatergic synapse by BDNF, and its implications in synaptic plasticity.<\/jats:p><jats:p><jats:italic>British Journal of Pharmacology<\/jats:italic> (2008) <jats:bold>153<\/jats:bold>, S310\u2013S324; doi:<jats:ext-link xmlns:xlink=\"http:\/\/www.w3.org\/1999\/xlink\" ext-link-type=\"doi\" xlink:href=\"10.1038\/sj.bjp.0707509\">10.1038\/sj.bjp.0707509<\/jats:ext-link>; published online 3 December 2007<\/jats:p>","DOI":"10.1038\/sj.bjp.0707509","type":"journal-article","created":{"date-parts":[[2007,12,3]],"date-time":"2007-12-03T11:18:18Z","timestamp":1196680698000},"update-policy":"https:\/\/doi.org\/10.1002\/crossmark_policy","source":"Crossref","is-referenced-by-count":202,"title":["Role of the brain\u2010derived neurotrophic factor at glutamatergic synapses"],"prefix":"10.1111","volume":"153","author":[{"given":"A L","family":"Carvalho","sequence":"first","affiliation":[]},{"given":"M 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