{"status":"ok","message-type":"work","message-version":"1.0.0","message":{"indexed":{"date-parts":[[2026,9,26]],"date-time":"2026-09-26T10:51:13Z","timestamp":1790419873289,"version":"4.1.0"},"reference-count":28,"publisher":"Wiley","issue":"8","license":[{"start":{"date-parts":[[2002,4,24]],"date-time":"2002-04-24T00:00:00Z","timestamp":1019606400000},"content-version":"vor","delay-in-days":631,"URL":"http:\/\/onlinelibrary.wiley.com\/termsAndConditions#vor"}],"content-domain":{"domain":[],"crossmark-restriction":false},"short-container-title":["Transfusion"],"published-print":{"date-parts":[[2000,8]]},"abstract":"<jats:p><jats:bold>BACKGROUND:<\/jats:bold> Acute intravascular hemolysis is rarely associated with platelet transfusion. Out\u2010of\u2010group single\u2010donor platelets may cause hemolysis if the donor has high\u2010titer ABO hemagglutinins.<\/jats:p><jats:p><jats:bold>CASE REPORT:<\/jats:bold> A 44\u2010year\u2010old woman, blood group A, was recently diagnosed with acute myeloid leukemia and was receiving chemotherapy. After the transfusion of apheresis platelets from a group O donor, back pain, hemoglobinuria, and hemoglobinemia developed, and her Hb dropped by 2.3 g per dL, despite the transfusion of 2 units of RBCs.<\/jats:p><jats:p><jats:bold>RESULTS:<\/jats:bold> Investigation revealed acute intravascular hemolysis with a positive DAT due to anti\u2010A<jats:sub>1<\/jats:sub> on her RBCs. The donor's titer of anti\u2010A<jats:sub>1<\/jats:sub> was greater than 16,000.<\/jats:p><jats:p><jats:bold>CONCLUSION:<\/jats:bold> Review of published cases raises the possibility that hemolytic reactions to out\u2010of\u2010group platelets may be more frequent since the use of apheresis platelets has increased.<\/jats:p>","DOI":"10.1046\/j.1537-2995.2000.40080902.x","type":"journal-article","created":{"date-parts":[[2003,3,12]],"date-time":"2003-03-12T22:43:38Z","timestamp":1047509018000},"page":"902-906","source":"Crossref","is-referenced-by-count":90,"title":["Acute intravascular hemolysis secondaryto out\u2010of\u2010group platelet transfusion"],"prefix":"10.1111","volume":"40","author":[{"given":"L.G.","family":"Larsson","sequence":"first","affiliation":[],"role":[{"vocabulary":"crossref","role":"author"}]},{"given":"V.J.","family":"Welsh","sequence":"additional","affiliation":[],"role":[{"vocabulary":"crossref","role":"author"}]},{"given":"D.J.","family":"Ladd","sequence":"additional","affiliation":[],"role":[{"vocabulary":"crossref","role":"author"}]}],"member":"311","published-online":{"date-parts":[[2002,4,24]]},"reference":[{"key":"e_1_2_6_2_2","doi-asserted-by":"publisher","DOI":"10.1046\/j.1537-2995.1990.30790385515.x"},{"key":"e_1_2_6_3_2","doi-asserted-by":"publisher","DOI":"10.7326\/0003-4819-43-6-1334"},{"key":"e_1_2_6_4_2","doi-asserted-by":"crossref","first-page":"425","DOI":"10.4049\/jimmunol.8.6.425","article-title":"Dangerous \u201cuniversal donor\u201d detected by the direct matching of bloods.","volume":"8","author":"Levine P","year":"1923","journal-title":"J Immunol"},{"key":"e_1_2_6_5_2","doi-asserted-by":"publisher","DOI":"10.1084\/jem.13.4.425"},{"key":"e_1_2_6_6_2","volume-title":"Standards for blood banks and transfusion services","author":"Menitove JE","year":"1999"},{"key":"e_1_2_6_7_2","doi-asserted-by":"publisher","DOI":"10.1046\/j.1537-2995.1988.28588337323.x"},{"key":"e_1_2_6_8_2","doi-asserted-by":"publisher","DOI":"10.1046\/j.1537-2995.1977.17177128880.x"},{"key":"e_1_2_6_9_2","doi-asserted-by":"crossref","first-page":"403","DOI":"10.1182\/blood.V11.5.403.403","article-title":"Anti\u2010A agglutinins in pooled plasma as a cause of hemolytic anemia.","volume":"11","author":"Rutzky J","year":"1956","journal-title":"Blood"},{"key":"e_1_2_6_10_2","first-page":"553","article-title":"Dangerous universal donors. 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