{"status":"ok","message-type":"work","message-version":"1.0.0","message":{"indexed":{"date-parts":[[2026,8,17]],"date-time":"2026-08-17T22:09:54Z","timestamp":1787004594103,"version":"build-2736575974"},"reference-count":29,"publisher":"Wiley","issue":"11","license":[{"start":{"date-parts":[[2002,11,8]],"date-time":"2002-11-08T00:00:00Z","timestamp":1036713600000},"content-version":"vor","delay-in-days":7,"URL":"http:\/\/onlinelibrary.wiley.com\/termsAndConditions#vor"}],"content-domain":{"domain":[],"crossmark-restriction":false},"short-container-title":["Transfusion"],"published-print":{"date-parts":[[2002,11]]},"abstract":"<jats:p>\n                    <jats:bold>BACKGROUND<\/jats:bold>\n                    : Antigens of the Cromer blood group system reside on the glycoprotein CD55 (decay\u2010accelerating factor). The Inab phenotype is the null phenotype of this system. So far, only five propositi have been described who exhibit this phenotype, and single\u2010nucleotide substitutions in the CD55 gene have been found in three of them. This report describes the first example of a patient with an acquired and transient form of the Inab phenotype.\n                  <\/jats:p>\n                  <jats:p>\n                    <jats:bold>CASE REPORT<\/jats:bold>\n                    : A 54\u2010year\u2010old black patient was admitted to the hospital because of abdominal pain. Multiple splenic infarctions were visualized in the abdominal computerized tomography scan, and a prophylactic splenectomy was performed. The patient's serum reacted by an IAT with all donor RBCs tested.\n                  <\/jats:p>\n                  <jats:p>\n                    <jats:bold>RESULTS<\/jats:bold>\n                    : Serologic analysis showed that the patient had the rare Inab phenotype and that his serum contained anti\u2010IFC. Flow cytometry demonstrated the absence of CD55 on his RBCs, whereas lymphocytes, monocytes, granulocytes, and platelets expressed CD55, albeit at a weaker level than cells of common phenotypes. cDNA revealed no differences from the published sequences. Flow cytometry performed 12 months after splenectomy showed reappearance of the CD55 antigen; serologic tests performed after 17 months revealed that the anti\u2010IFC had almost disappeared and that the RBCs were again agglutinated by various Cromer antibodies.\n                  <\/jats:p>\n                  <jats:p>\n                    <jats:bold>CONCLUSION<\/jats:bold>\n                    : A patient with an acquired and transient form of the Inab phenotype is described, in whom the CD55 deficiency is limited to the RBCs and is associated with splenic infarctions.\n                  <\/jats:p>","DOI":"10.1046\/j.1537-2995.2002.00214.x","type":"journal-article","created":{"date-parts":[[2003,3,12]],"date-time":"2003-03-12T17:49:23Z","timestamp":1047491363000},"page":"1448-1457","source":"Crossref","is-referenced-by-count":18,"title":["Acquired and transient RBC CD55 deficiency (Inab phenotype) and anti\u2010IFC"],"prefix":"10.1111","volume":"42","author":[{"given":"Thomas","family":"Matthes","sequence":"first","affiliation":[],"role":[{"vocabulary":"crossref","role":"author"}]},{"given":"Emanuelle","family":"Tullen","sequence":"additional","affiliation":[],"role":[{"vocabulary":"crossref","role":"author"}]},{"given":"Joyce","family":"Poole","sequence":"additional","affiliation":[],"role":[{"vocabulary":"crossref","role":"author"}]},{"given":"Jackie","family":"Banks","sequence":"additional","affiliation":[],"role":[{"vocabulary":"crossref","role":"author"}]},{"given":"Monika","family":"Nagy","sequence":"additional","affiliation":[],"role":[{"vocabulary":"crossref","role":"author"}]},{"given":"Marie\u2010Jose","family":"Stelling","sequence":"additional","affiliation":[],"role":[{"vocabulary":"crossref","role":"author"}]},{"given":"Fran\u00e7oise","family":"Boehlen","sequence":"additional","affiliation":[],"role":[{"vocabulary":"crossref","role":"author"}]},{"given":"Martine","family":"Michel","sequence":"additional","affiliation":[],"role":[{"vocabulary":"crossref","role":"author"}]},{"given":"Photis","family":"Beris","sequence":"additional","affiliation":[],"role":[{"vocabulary":"crossref","role":"author"}]},{"given":"Hein","family":"Hustinx","sequence":"additional","affiliation":[],"role":[{"vocabulary":"crossref","role":"author"}]},{"given":"Vanja","family":"Crew","sequence":"additional","affiliation":[],"role":[{"vocabulary":"crossref","role":"author"}]},{"given":"Geoff","family":"Daniels","sequence":"additional","affiliation":[],"role":[{"vocabulary":"crossref","role":"author"}]}],"member":"311","published-online":{"date-parts":[[2002,11,8]]},"reference":[{"key":"e_1_2_7_2_2","doi-asserted-by":"publisher","DOI":"10.1084\/jem.167.6.1993"},{"key":"e_1_2_7_3_2","doi-asserted-by":"publisher","DOI":"10.1146\/annurev.iy.07.040189.000343"},{"key":"e_1_2_7_4_2","doi-asserted-by":"publisher","DOI":"10.1073\/pnas.84.7.2007"},{"key":"e_1_2_7_5_2","doi-asserted-by":"crossref","first-page":"740","DOI":"10.4049\/jimmunol.144.2.740","article-title":"Structure of the gene for human complement protein decay accelerating factor.","volume":"144","author":"Post TW","year":"1990","journal-title":"J Immunol"},{"key":"e_1_2_7_6_2","doi-asserted-by":"crossref","first-page":"1221","DOI":"10.1084\/jem.166.5.1221","article-title":"The mechanism of action of decay\u2010accelerating factor (DAF): DAF inhibits the assembly of C3 convertases by dissociating C2a and Bb.","volume":"166","author":"Fujita T","year":"1987","journal-title":"J Exp Med"},{"key":"e_1_2_7_7_2","doi-asserted-by":"crossref","first-page":"184","DOI":"10.4049\/jimmunol.129.1.184","article-title":"Isolation of a human erythrocyte membrane glycoprotein with decay\u2010accelerating activity for C3 convertases of the complement system.","volume":"129","author":"Nicholson\u2010Weller A","year":"1982","journal-title":"J Immunol"},{"key":"e_1_2_7_8_2","doi-asserted-by":"publisher","DOI":"10.1016\/S0925-4439(99)00068-X"},{"key":"e_1_2_7_9_2","doi-asserted-by":"crossref","first-page":"1057","DOI":"10.1128\/iai.56.5.1057-1060.1988","article-title":"A hemagglutinin of uropathogenic Escherichia coli recognizes the Dr blood group antigen.","volume":"56","author":"Nowicki B","year":"1988","journal-title":"Infect Immun"},{"key":"e_1_2_7_10_2","doi-asserted-by":"crossref","first-page":"6245","DOI":"10.1073\/pnas.91.13.6245","article-title":"Decay\u2010accelerating factor (CD55), a glycosylphosphatidylinositol\u2010 anchored complement regulatory protein, is a receptor for several echoviruses.","volume":"91","author":"Bergelson JM","year":"1994","journal-title":"Proc Natl Acad Sci U S A"},{"key":"e_1_2_7_11_2","doi-asserted-by":"crossref","first-page":"3873","DOI":"10.1128\/jvi.69.6.3873-3877.1995","article-title":"Coxsackieviruses B1, B3, and B5 use decay accelerating factor as a receptor for cell attachment.","volume":"69","author":"Shafren DR","year":"1995","journal-title":"J Virol"},{"key":"e_1_2_7_12_2","doi-asserted-by":"crossref","unstructured":"Daniels G. 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