{"status":"ok","message-type":"work","message-version":"1.0.0","message":{"indexed":{"date-parts":[[2026,8,28]],"date-time":"2026-08-28T14:46:00Z","timestamp":1787928360791,"version":"build-2784847793"},"reference-count":83,"publisher":"Georg Thieme Verlag KG","issue":"05","content-domain":{"domain":[],"crossmark-restriction":false},"short-container-title":["Horm Metab Res"],"published-print":{"date-parts":[[2024,5]]},"abstract":"<jats:title>Abstract<\/jats:title>\n                  <jats:p>Glucocorticoid-induced myopathy is a non-inflammatory toxic myopathy typified by\n                    proximal muscle weakness, muscle atrophy, fatigue, and easy fatigability. These\n                    vague symptoms coupled with underlying disorders may mask the signs of\n                    glucocorticoid-induced myopathy, leading to an underestimation of the disease\u2019s\n                    impact. This review briefly summarizes the classification, pathogenesis, and\n                    treatment options for glucocorticoid-induced muscle wasting. Additionally, we\n                    discuss current diagnostic measures in clinical research and routine care used\n                    for diagnosing and monitoring glucocorticoid-induced myopathy, which includes\n                    gait speed tests, muscle strength tests, hematologic tests, bioelectrical\n                    impedance analysis (BIA), dual-energy X-ray absorptiometry (DXA), computed\n                    tomography (CT), magnetic resonance imaging (MRI), electromyography,\n                    quantitative muscle ultrasound, histological examination, and genetic analysis.\n                    Continuous monitoring of patients receiving glucocorticoid therapy plays an\n                    important role in enabling early detection of glucocorticoid-induced myopathy,\n                    allowing physicians to modify treatment plans before significant clinical\n                    weakness arises.<\/jats:p>","DOI":"10.1055\/a-2246-2900","type":"journal-article","created":{"date-parts":[[2024,1,15]],"date-time":"2024-01-15T20:11:36Z","timestamp":1705349496000},"page":"341-349","source":"Crossref","is-referenced-by-count":23,"title":["Glucocorticoid-Induced Myopathy: Typology, Pathogenesis, Diagnosis,\n                    and Treatment"],"prefix":"10.1055","volume":"56","author":[{"given":"Mengmeng","family":"Wu","sequence":"additional","affiliation":[{"name":"Department of Nephrology, Xuzhou Medical University Affiliated\n                    Hospital, Xuzhou, China"},{"name":"Graduate School, Xuzhou Medical University, Xuzhou, China"}],"role":[{"vocabulary":"crossref","role":"author"}]},{"given":"Caixia","family":"Liu","sequence":"additional","affiliation":[{"name":"Department of Nephrology, Xuzhou Medical University Affiliated\n                    Hospital, Xuzhou, China"}],"role":[{"vocabulary":"crossref","role":"author"}]},{"given":"Dong","family":"Sun","sequence":"additional","affiliation":[{"name":"Department of Nephrology, Xuzhou Medical University Affiliated\n                    Hospital, Xuzhou, China"},{"name":"Department of Internal Medicine and Diagnostics, Xuzhou Medical\n                    University, Xuzhou, China"}],"role":[{"vocabulary":"crossref","role":"author"}]}],"member":"194","published-online":{"date-parts":[[2024,3,4]]},"reference":[{"key":"ref1","doi-asserted-by":"crossref","first-page":"4294","DOI":"10.1111\/febs.12253","article-title":"Mechanisms regulating skeletal muscle growth and atrophy","volume":"280","author":"S Schiaffino","year":"2013","journal-title":"FEBS J"},{"key":"ref2","doi-asserted-by":"crossref","first-page":"584","DOI":"10.1038\/nm0604-584","article-title":"Balancing muscle hypertrophy and atrophy","volume":"10","author":"E P Hoffman","year":"2004","journal-title":"Nat 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