{"status":"ok","message-type":"work","message-version":"1.0.0","message":{"indexed":{"date-parts":[[2026,5,19]],"date-time":"2026-05-19T10:11:38Z","timestamp":1779185498148,"version":"3.51.4"},"reference-count":80,"publisher":"Georg Thieme Verlag KG","issue":"11","content-domain":{"domain":[],"crossmark-restriction":false},"short-container-title":["Rofo"],"published-print":{"date-parts":[[2024,11]]},"abstract":"<jats:p>\n            Background\u2002Cerebral damage after aneurysmal subarachnoid hemorrhage (SAH) results from various, sometimes unrelated causes. After the initial hemorrhage trauma with an increase in intracranial pressure, induced vasoconstriction, but also microcirculatory disturbances, inflammation and pathological electrophysiological processes (cortical spreading depolarization) can occur in the course of the disease, resulting in delayed cerebral ischemia (DCI). In the neuroradiological context, cerebral vasospasm (CVS) remains the focus of diagnostic imaging and endovascular therapy as a frequent component of the genesis of DCI.<\/jats:p><jats:p>\n            Methods\u2002The amount of blood leaked during aneurysm rupture (which can be detected by CT, for example) correlates with the occurrence and severity of CVS.\u200aCT perfusion is then an important component in determining the indication for endovascular spasm therapies (EST). These include intra-arterial drug administration (also as long-term microcatheter treatment) and mechanical procedures (balloon angioplasty, vasodilatation using other instruments such as stent retrievers, stenting).<\/jats:p><jats:p>\n            Conclusion\u2002This review summarizes the current findings on the diagnosis and treatment of CVS after aneurysmal SAH from a neuroradiological perspective, taking into account the complex and up-to-date international literature.<\/jats:p><jats:p>\n            Key Points\n          <\/jats:p><jats:p>\n            Citation Format\n          <\/jats:p>","DOI":"10.1055\/a-2266-3117","type":"journal-article","created":{"date-parts":[[2024,3,14]],"date-time":"2024-03-14T00:00:27Z","timestamp":1710374427000},"page":"1125-1133","source":"Crossref","is-referenced-by-count":6,"title":["Neuroradiological diagnosis and therapy of cerebral vasospasm after subarachnoid hemorrhage"],"prefix":"10.1055","volume":"196","author":[{"ORCID":"https:\/\/orcid.org\/0000-0002-5651-3900","authenticated-orcid":false,"given":"Alexander","family":"Neumann","sequence":"additional","affiliation":[{"name":"Department of Neuroradiology, University Hospital Schleswig-Holstein, Campus L\u00fcbeck, Germany"}],"role":[{"role":"author","vocabulary":"crossref"}]},{"given":"Hannes","family":"Schacht","sequence":"additional","affiliation":[{"name":"Department of Neuroradiology, University Hospital Schleswig-Holstein, Campus L\u00fcbeck, Germany"}],"role":[{"role":"author","vocabulary":"crossref"}]},{"given":"Peter","family":"Schramm","sequence":"additional","affiliation":[{"name":"Department of Neuroradiology, University Hospital Schleswig-Holstein, Campus L\u00fcbeck, Germany"}],"role":[{"role":"author","vocabulary":"crossref"}]}],"member":"194","published-online":{"date-parts":[[2024,3,13]]},"reference":[{"issue":"05","key":"ref1","doi-asserted-by":"crossref","first-page":"1426","DOI":"10.1161\/STROKEAHA.122.040072","article-title":"Early Brain Injury After Subarachnoid Hemorrhage: Incidence and Mechanisms","volume":"54","author":"D C Lauzier","year":"2023","journal-title":"Stroke"},{"issue":"03","key":"ref2","doi-asserted-by":"crossref","first-page":"1015","DOI":"10.3390\/jcm12031015","article-title":"Pathophysiology of Early Brain Injury and Its Association with Delayed Cerebral Ischemia in Aneurysmal Subarachnoid Hemorrhage: A Review of Current Literature","volume":"12","author":"D L Alsbrook","year":"2023","journal-title":"J Clin Med"},{"issue":"15","key":"ref3","doi-asserted-by":"crossref","first-page":"e021845","DOI":"10.1161\/JAHA.121.021845","article-title":"Pathophysiology of Delayed Cerebral Ischemia After Subarachnoid Hemorrhage: A 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