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In the current study, we provide evidence that enhancing signaling at the glutamate\n                    <jats:italic>N<\/jats:italic>\n                    -methyl-\n                    <jats:sc>d<\/jats:sc>\n                    -aspartate receptor (NMDAR) can enhance the mechanism underlying many forms of experience-dependent plasticity (i.e., long-term potentiation of synaptic currents) and also enhance experience-dependent learning in healthy adult humans. This suggests exciting possibilities for manipulating plasticity in adults and has implications for treating neurological and neuropsychiatric disorders in which experience-dependent plasticity is impaired.\n                  <\/jats:p>","DOI":"10.1073\/pnas.1509262112","type":"journal-article","created":{"date-parts":[[2015,11,30]],"date-time":"2015-11-30T23:15:18Z","timestamp":1448925318000},"page":"15331-15336","update-policy":"https:\/\/doi.org\/10.1073\/pnas.cm10313","source":"Crossref","is-referenced-by-count":78,"title":["Augmenting NMDA receptor signaling boosts experience-dependent neuroplasticity in the adult human brain"],"prefix":"10.1073","volume":"112","author":[{"given":"Jennifer K.","family":"Forsyth","sequence":"first","affiliation":[{"name":"Department of Psychology, University of California, Los Angeles, CA 90095;"}],"role":[{"vocabulary":"crossref","role":"author"}]},{"given":"Peter","family":"Bachman","sequence":"additional","affiliation":[{"name":"Department of 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