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Modeling coinfection using macaques, we discovered that one-third of the animals maintained latency despite complete ablation of lung CD4\n                    <jats:sup>+<\/jats:sup>\n                    T cells. We report that protective immune responses mediated by CD8\n                    <jats:sup>+<\/jats:sup>\n                    T cells and B cells correlate with TB control. These findings have important implications in development of both prophylactic and therapeutic measures against TB and acquired immunodeficiency syndrome.\n                  <\/jats:p>","DOI":"10.1073\/pnas.1611987113","type":"journal-article","created":{"date-parts":[[2016,9,6]],"date-time":"2016-09-06T21:55:26Z","timestamp":1473198926000},"update-policy":"https:\/\/doi.org\/10.1073\/pnas.cm10313","source":"Crossref","is-referenced-by-count":137,"title":["CD4\n                    <sup>+<\/sup>\n                    T-cell\u2013independent mechanisms suppress reactivation of latent tuberculosis in a macaque model of HIV coinfection"],"prefix":"10.1073","volume":"113","author":[{"given":"Taylor W.","family":"Foreman","sequence":"first","affiliation":[{"name":"Tulane National Primate Research Center, Covington, LA 70471;"},{"name":"Department of Microbiology and Immunology, Tulane University School of Medicine, New Orleans, LA 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