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Left ventricular chamber dilation and decreased ejection fraction, observed in D94A hearts, were indicative of systolic dysfunction, a hallmark of DCM. Our study suggests that\n                    <jats:italic>MYL2<\/jats:italic>\n                    may be considered a therapeutic target for dilated cardiomyopathy.\n                  <\/jats:p>","DOI":"10.1073\/pnas.1716925115","type":"journal-article","created":{"date-parts":[[2018,2,20]],"date-time":"2018-02-20T14:35:01Z","timestamp":1519137301000},"update-policy":"https:\/\/doi.org\/10.1073\/pnas.cm10313","source":"Crossref","is-referenced-by-count":37,"title":["Sarcomeric perturbations of myosin motors lead to dilated cardiomyopathy in genetically modified\n                    <i>MYL2<\/i>\n                    mice"],"prefix":"10.1073","volume":"115","author":[{"given":"Chen-Ching","family":"Yuan","sequence":"first","affiliation":[{"name":"Molecular and Cellular Pharmacology, University of Miami Miller School of Medicine, Miami, FL 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