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Accordingly, loss of SLC39A14 impairs Mn excretion, leading to Mn accumulation in the brain and most other extrahepatic tissues.\n                    <jats:italic>Slc39a14<\/jats:italic>\n                    -deficient mice, similar to\n                    <jats:italic>SLC39A14<\/jats:italic>\n                    -deficient human patients, display motor deficits, and thus offer a convenient model to study Mn\/SLC39A14-related neurotoxicity.\n                  <\/jats:p>","DOI":"10.1073\/pnas.1720739115","type":"journal-article","created":{"date-parts":[[2018,2,7]],"date-time":"2018-02-07T15:30:14Z","timestamp":1518017414000},"update-policy":"https:\/\/doi.org\/10.1073\/pnas.cm10313","source":"Crossref","is-referenced-by-count":134,"title":["SLC39A14 deficiency alters manganese homeostasis and excretion resulting in brain manganese accumulation and motor deficits in mice"],"prefix":"10.1073","volume":"115","author":[{"given":"Supak","family":"Jenkitkasemwong","sequence":"first","affiliation":[{"name":"Food Science &amp; 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