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ZRL\n                    <jats:sub>5<\/jats:sub>\n                    P\n                    <jats:sub>4<\/jats:sub>\n                    is able to emit unique responsive fluorescent signals once it binds with EBNA1 and a Zn\n                    <jats:sup>2+<\/jats:sup>\n                    ion. ZRL\n                    <jats:sub>5<\/jats:sub>\n                    P\n                    <jats:sub>4<\/jats:sub>\n                    can selectively disrupt the EBNA1 oligomerization and cause nasopharyngeal carcinoma (NPC) tumor shrinkage, possibly due to EBV lytic induction. Dicer1 seems essential for this lytic reactivation. As can been seen, EBNA1 is likely to maintain NPC cell survival by suppressing viral reactivation.\n                  <\/jats:p>","DOI":"10.1073\/pnas.1915372116","type":"journal-article","created":{"date-parts":[[2019,12,10]],"date-time":"2019-12-10T17:44:24Z","timestamp":1575999864000},"page":"26614-26624","update-policy":"https:\/\/doi.org\/10.1073\/pnas.cm10313","source":"Crossref","is-referenced-by-count":35,"title":["Reactivation of Epstein\u2013Barr virus by a dual-responsive fluorescent EBNA1-targeting agent with Zn\n                    <sup>2+<\/sup>\n                    -chelating function"],"prefix":"10.1073","volume":"116","author":[{"given":"Lijun","family":"Jiang","sequence":"first","affiliation":[{"name":"Department of Chemistry, Hong Kong Baptist University, Kowloon Tong, Kowloon, Hong Kong;"},{"name":"Department of Chemistry, Imperial College London, London W12 0BZ, United Kingdom;"}],"role":[{"vocabulary":"crossref","role":"author"}]},{"given":"Hong 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