{"status":"ok","message-type":"work","message-version":"1.0.0","message":{"indexed":{"date-parts":[[2024,2,14]],"date-time":"2024-02-14T00:20:58Z","timestamp":1707870058440},"reference-count":23,"publisher":"Cambridge University Press (CUP)","issue":"3","license":[{"start":{"date-parts":[[2007,7,24]],"date-time":"2007-07-24T00:00:00Z","timestamp":1185235200000},"content-version":"unspecified","delay-in-days":8119,"URL":"https:\/\/www.cambridge.org\/core\/terms"}],"content-domain":{"domain":[],"crossmark-restriction":false},"short-container-title":["Br J Nutr"],"published-print":{"date-parts":[[1985,5]]},"abstract":"<jats:p>1. Thiamin status has been measured using the erythrocyte transketolase (EC 2.2.1.1) assay in twenty-eight normal children and in twenty-five severely-malnourished children throughout the course of recovery.<\/jats:p><jats:p>2. Subclinical thiamin deficiency was found in 7% of the normal children and 36% of the malnourished children on admission.<\/jats:p><jats:p>3. There was no significant association between thiamin status and oedema, stunting or wasting, history of breast-feeding, pattern of weaning, age or sex.<\/jats:p><jats:p>4. Five malnourished children, who died, all had a normal thiamin status on admission; however, two developed biochemical evidence of thiamin deficiency preterminally.<\/jats:p>","DOI":"10.1079\/bjn19850057","type":"journal-article","created":{"date-parts":[[2005,5,19]],"date-time":"2005-05-19T23:49:52Z","timestamp":1116546592000},"page":"477-483","source":"Crossref","is-referenced-by-count":15,"title":["Thiamin status in normal and malnourished children in Jamaica"],"prefix":"10.1017","volume":"53","author":[{"given":"B.","family":"Hailemariam","sequence":"first","affiliation":[],"role":[{"role":"author","vocabulary":"crossref"}]},{"given":"J. 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