{"status":"ok","message-type":"work","message-version":"1.0.0","message":{"indexed":{"date-parts":[[2026,9,21]],"date-time":"2026-09-21T11:00:54Z","timestamp":1789988454701,"version":"4.0.1"},"reference-count":28,"publisher":"SAGE Publications","issue":"11-12","license":[{"start":{"date-parts":[[2006,11,1]],"date-time":"2006-11-01T00:00:00Z","timestamp":1162339200000},"content-version":"tdm","delay-in-days":0,"URL":"https:\/\/journals.sagepub.com\/page\/policies\/text-and-data-mining-license"}],"content-domain":{"domain":[],"crossmark-restriction":false},"short-container-title":["Aust N Z J Psychiatry"],"published-print":{"date-parts":[[2006,11]]},"abstract":"<jats:p>Objective: Despite the neuropsychology literature providing reliable evidence of impaired executive functions in obsessive\u2013compulsive disorder (OCD), to date it has not been determined whether these deficits are trait-related (independent of symptomatology) or state-dependent (dependent on symptomatology). The current research examines the executive functions in OCD in a comprehensive manner and, for the first time, assesses the stability of these deficits over the developmental course of the disorder.<\/jats:p>\n                  <jats:p>Method: Using a cross-sectional design, Study 1 examined the executive functions (set shifting, inhibition, planning, verbal fluency and working memory) in 60 subjects (20 actively Symptomatic OCD, 20 Remitted OCD and 20 Panic Disorder). Using a longitudinal design, Study 2 reassessed a subsample of OCD subjects (participants in Study 1) once they reached remitted status.<\/jats:p>\n                  <jats:p>Results: Study 1 found that the OCD groups exhibited deficits in set shifting and inhibition relative to Panic Disorder controls; however, no deficits were observed in planning, verbal fluency or working memory. There were no differences found between the Symptomatic and Remitted OCD groups on any of the executive function measures. Study 2 found that the identified executive function deficits in individuals were stable over time and remained unchanged despite symptom remittance.<\/jats:p>\n                  <jats:p>Conclusion: Current results confirm the presence of specific executive function deficits in OCD, and indicate that these deficits are trait-like in nature.<\/jats:p>","DOI":"10.1080\/j.1440-1614.2006.01928.x","type":"journal-article","created":{"date-parts":[[2009,12,7]],"date-time":"2009-12-07T18:38:00Z","timestamp":1260211080000},"page":"1031-1038","source":"Crossref","is-referenced-by-count":16,"title":["Executive Functions in Obsessive\u2013Compulsive Disorder: State or Trait Deficits?"],"prefix":"10.1177","volume":"40","author":[{"given":"Shelley","family":"Bannon","sequence":"first","affiliation":[{"name":"Brain & Behaviour Research Institute, School of Psychology, University of Wollongong, Wollongong, New South Wales, Australia"}],"role":[{"vocabulary":"crossref","role":"author"}]},{"given":"Craig J.","family":"Gonsalvez","sequence":"additional","affiliation":[{"name":"Brain & Behaviour Research Institute, School of Psychology, University of Wollongong, Wollongong, New South Wales, Australia"}],"role":[{"vocabulary":"crossref","role":"author"}]},{"given":"Rodney J.","family":"Croft","sequence":"additional","affiliation":[{"name":"Brain Sciences Institute, Swinburne University of Technology, Melbourne, Australia"}],"role":[{"vocabulary":"crossref","role":"author"}]},{"given":"Philip M.","family":"Boyce","sequence":"additional","affiliation":[{"name":"Department of Psychological Medicine, University of Sydney, Westmead Hospital, Sydney, Australia"}],"role":[{"vocabulary":"crossref","role":"author"}]}],"member":"179","published-online":{"date-parts":[[2006,11,1]]},"reference":[{"key":"bibr1-j.1440-1614.2006.01928.x","doi-asserted-by":"publisher","DOI":"10.1016\/j.cpr.2005.06.010"},{"key":"bibr2-j.1440-1614.2006.01928.x","doi-asserted-by":"publisher","DOI":"10.1111\/j.1600-0447.2004.00502.x"},{"key":"bibr3-j.1440-1614.2006.01928.x","doi-asserted-by":"publisher","DOI":"10.1016\/S0193-953X(03)00107-2"},{"key":"bibr4-j.1440-1614.2006.01928.x","doi-asserted-by":"publisher","DOI":"10.1016\/j.biopsycho.2003.07.007"},{"key":"bibr5-j.1440-1614.2006.01928.x","doi-asserted-by":"crossref","unstructured":"5. 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