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Defects in centriole number and structure are associated with human diseases including cancer and ciliopathies. Discovery of the centriole dates back to the 19th century. However, recent advances in genetic and biochemical tools, development of high-resolution microscopy, and identification of centriole components have accelerated our understanding of its assembly, function, evolution, and its role in human disease. The centriole is an evolutionarily conserved structure built from highly conserved proteins and is present in all branches of the eukaryotic tree of life. However, centriole number, size, and organization varies among different organisms and even cell types within a single organism, reflecting its cell type\u2013specialized functions. In this review, we provide an overview of our current understanding of centriole biogenesis and how variations around the same theme generate alternatives for centriole formation and function.<\/jats:p>","DOI":"10.1083\/jcb.201704093","type":"journal-article","created":{"date-parts":[[2017,12,28]],"date-time":"2017-12-28T14:50:58Z","timestamp":1514472658000},"page":"823-835","update-policy":"https:\/\/doi.org\/10.1083\/jcb.crossmarkpolicy","source":"Crossref","is-referenced-by-count":93,"title":["Building the right centriole for each cell type"],"prefix":"10.1083","volume":"217","author":[{"ORCID":"https:\/\/orcid.org\/0000-0003-1879-5473","authenticated-orcid":true,"given":"Jadranka","family":"Loncarek","sequence":"first","affiliation":[{"name":"Cell Cycle Regulation Lab, Gulbenkian Institute of Science, Oeiras, Portugal 2"}]},{"ORCID":"https:\/\/orcid.org\/0000-0003-1987-5598","authenticated-orcid":true,"given":"M\u00f3nica","family":"Bettencourt-Dias","sequence":"additional","affiliation":[{"name":"Laboratory of Protein Dynamics and Signaling, National Institutes of Health\/Center for Cancer Research\/National Cancer Institute-Frederick, Frederick, MD 1"}]}],"member":"291","published-online":{"date-parts":[[2017,12,28]]},"reference":[{"key":"2023072309324909000_bib1","doi-asserted-by":"publisher","first-page":"694","DOI":"10.1128\/MCB.00253-09","article-title":"Cdk2 and Cdk4 regulate the centrosome cycle and are critical mediators of centrosome amplification in p53-null cells","volume":"30","author":"Adon","year":"2010","journal-title":"Mol. 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