{"status":"ok","message-type":"work","message-version":"1.0.0","message":{"indexed":{"date-parts":[[2026,5,1]],"date-time":"2026-05-01T18:39:31Z","timestamp":1777660771839,"version":"3.51.4"},"reference-count":17,"publisher":"SAGE Publications","issue":"8","license":[{"start":{"date-parts":[[2013,6,12]],"date-time":"2013-06-12T00:00:00Z","timestamp":1370995200000},"content-version":"vor","delay-in-days":0,"URL":"https:\/\/creativecommons.org\/licenses\/by\/4.0\/"},{"start":{"date-parts":[[2013,6,12]],"date-time":"2013-06-12T00:00:00Z","timestamp":1370995200000},"content-version":"tdm","delay-in-days":0,"URL":"https:\/\/journals.sagepub.com\/page\/policies\/text-and-data-mining-license"}],"content-domain":{"domain":["journals.sagepub.com"],"crossmark-restriction":true},"short-container-title":["Genetic Testing and Molecular Biomarkers"],"published-print":{"date-parts":[[2013,8,1]]},"abstract":"<jats:p>\n                    Ankylosing spondylitis (AS) is a largely genetically determined autoimmune disease.\n                    <jats:italic toggle=\"yes\">JMY<\/jats:italic>\n                    has recently been found to be associated with susceptibility to AS in patients of western European descent. We aimed to examine the influence of\n                    <jats:italic toggle=\"yes\">JMY<\/jats:italic>\n                    polymorphisms on the severity of AS in the Chinese ethnic majority Han population. Blood samples were drawn from 396 Chinese Han AS patients whose duration of disease was about 9-12 years. Four tag single-nucleotide polymorphisms (tagSNPs) in\n                    <jats:italic toggle=\"yes\">JMY<\/jats:italic>\n                    were selected and genotyped. Frequencies of different genotypes and clinical indexes about the severity of AS were analyzed. The rs2607142, rs16876619, and rs4704556 SNPs are related to BASFI. The rs2607142, rs4704556, and rs16876657 SNPs are related to BADAI. The rs4704556 and rs16876657 SNPs are related to mSASSS.\n                    <jats:italic toggle=\"yes\">JMY<\/jats:italic>\n                    is related to the severity of AS in Chinese Han patients.\n                  <\/jats:p>","DOI":"10.1089\/gtmb.2013.0112","type":"journal-article","created":{"date-parts":[[2013,6,12]],"date-time":"2013-06-12T09:21:35Z","timestamp":1371028895000},"page":"637-640","update-policy":"https:\/\/doi.org\/10.1177\/sage-journals-update-policy","source":"Crossref","is-referenced-by-count":1,"title":["<i>JMY<\/i>\n                    Polymorphism Is Related to Severity of Ankylosing Spondylitis in Chinese Han Patients"],"prefix":"10.1177","volume":"17","author":[{"given":"Wei","family":"Chai","sequence":"first","affiliation":[{"name":"Chinese People's Liberation Army General Hospital","place":["China"]}],"role":[{"role":"author","vocabulary":"crossref"}]},{"given":"Zijian","family":"Lian","sequence":"additional","affiliation":[{"name":"Tianjin Hospital","place":["China"]}],"role":[{"role":"author","vocabulary":"crossref"}]},{"given":"Chao","family":"Chen","sequence":"additional","affiliation":[{"name":"Tianjin Hospital","place":["China"]}],"role":[{"role":"author","vocabulary":"crossref"}]},{"given":"Jingyi","family":"Liu","sequence":"additional","affiliation":[{"name":"Medical School of Nankai University","place":["China"]}],"role":[{"role":"author","vocabulary":"crossref"}]},{"given":"Lewis L.","family":"Shi","sequence":"additional","affiliation":[{"name":"University of Chicago Hospital"}],"role":[{"role":"author","vocabulary":"crossref"}]},{"given":"Yan","family":"Wang","sequence":"additional","affiliation":[{"name":"Chinese People's Liberation Army General Hospital","place":["China"]},{"name":"Medical School of Nankai University","place":["China"]}],"role":[{"role":"author","vocabulary":"crossref"}]}],"member":"179","published-online":{"date-parts":[[2013,6,12]]},"reference":[{"key":"e_1_3_3_2_1","first-page":"997","article-title":"The natural course of radiographic progression in ankylosing spondylitis-evidence for major individual variations in a large proportion of patients","volume":"36","author":"Baraliakos X","year":"2009","unstructured":"Baraliakos X, , Listing J, , von der Recke A, , Braun J. 2009. The natural course of radiographic progression in ankylosing spondylitis-evidence for major individual variations in a large proportion of patients. J Rheumatol 2009, 36:997-1002.","journal-title":"J Rheumatol 2009"},{"key":"e_1_3_3_3_1","doi-asserted-by":"publisher","DOI":"10.1016\/S0140-6736(07)60635-7"},{"key":"e_1_3_3_4_1","first-page":"S43","article-title":"Genetics of ankylosing spondylitis","volume":"20","author":"Brown MA","year":"2002","unstructured":"Brown MA, , Wordsworth BP, , Reveille JD. 2002. Genetics of ankylosing spondylitis. Clin Exp Rheumatol, 20:S43-S49.","journal-title":"Clin Exp Rheumatol"},{"key":"e_1_3_3_5_1","first-page":"2281","article-title":"A new approach to defining functional ability in ankylosing spondylitis: the development of Bath ankylosing spondylitis disease functional index (BASFI)","volume":"21","author":"Calin A","year":"1994","unstructured":"Calin A, , Garrett S, , Whitelock H et al. 1994. A new approach to defining functional ability in ankylosing spondylitis: the development of Bath ankylosing spondylitis disease functional index (BASFI) J Rheumatol, 21:2281-2285.","journal-title":"J Rheumatol"},{"key":"e_1_3_3_6_1","doi-asserted-by":"publisher","DOI":"10.1002\/art.1780260210"},{"key":"e_1_3_3_7_1","doi-asserted-by":"publisher","DOI":"10.1038\/sj.embor.7400855"},{"key":"e_1_3_3_8_1","doi-asserted-by":"publisher","DOI":"10.1136\/ard.2004.020503"},{"key":"e_1_3_3_9_1","first-page":"2286","article-title":"A new approach to defining disease status in AS: the Bath ankylosing spondylitis disease activity index (BASDAI)","volume":"21","author":"Garrett S","year":"1994","unstructured":"Garrett S, , Jenkinson TR, , Whitelock HC et al. 1994. A new approach to defining disease status in AS: the Bath ankylosing spondylitis disease activity index (BASDAI) J Rheumatol, 21:2286-2291.","journal-title":"J Rheumatol"},{"key":"e_1_3_3_10_1","doi-asserted-by":"publisher","DOI":"10.1093\/rheumatology\/36.7.766"},{"key":"e_1_3_3_11_1","doi-asserted-by":"publisher","DOI":"10.1038\/gene.2011.23"},{"key":"e_1_3_3_12_1","doi-asserted-by":"publisher","DOI":"10.1016\/S1097-2765(00)80338-X"},{"key":"e_1_3_3_13_1","doi-asserted-by":"crossref","first-page":"1","DOI":"10.1136\/ard.2008.104018","article-title":"The Assessment of SpondyloArthritis international Society (ASAS) handbook: a guide to assess spondyloarthritis","volume":"68","author":"Sieper J","year":"2009","unstructured":"Sieper J, , Rudwaleit M, , Baraliakos X et al. 2009. The Assessment of SpondyloArthritis international Society (ASAS) handbook: a guide to assess spondyloarthritis. Ann Rheum Dis 2009, 68,Suppl II:1-44.","journal-title":"Ann Rheum Dis 2009"},{"key":"e_1_3_3_14_1","first-page":"8","article-title":"Interaction between ERAP1 and HLA-B27 in ankylosing spondylitis implicates peptide handling in the mechanism for HLA-B27 in disease susceptibility","volume":"43","author":"The Australo-Anglo-American Spondyloarthritis Consortium1 (TASC) & the Wellcome Trust Case Control Consortium (WTCCC)","year":"2011","unstructured":"The Australo-Anglo-American Spondyloarthritis Consortium1 (TASC) & the Wellcome Trust Case Control Consortium (WTCCC). 2011. Interaction between ERAP1 and HLA-B27 in ankylosing spondylitis implicates peptide handling in the mechanism for HLA-B27 in disease susceptibility. Nat Genet, 43:8.","journal-title":"Nat Genet"},{"key":"e_1_3_3_15_1","doi-asserted-by":"publisher","DOI":"10.1016\/j.clinbiochem.2004.12.005"},{"issue":"1","key":"e_1_3_3_16_1","first-page":"98","article-title":"Radiographic progression in ankylosing spondylitis","volume":"63","author":"van der Heijde D","year":"2004","unstructured":"van der Heijde D. 2004. Radiographic progression in ankylosing spondylitis. 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